D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 94 Citations 42,914 206 World Ranking 901 National Ranking 559

Research.com Recognitions

Awards & Achievements

2009 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Genetics

His primary scientific interests are in Molecular biology, Cell biology, Transcription factor, Signal transduction and STAT3. His Molecular biology research is multidisciplinary, relying on both Interferon-Stimulated Gene Factor 3, Cellular differentiation, Alpha interferon, Interferon regulatory factors and Response element. His Cell biology research incorporates themes from Perforin and Interleukin 21.

The Transcription factor study combines topics in areas such as Interferon, Cell nucleus and Epigenetics. He combines subjects such as Virology, stat and Phosphorylation with his study of Signal transduction. His STAT3 research integrates issues from Regulation of gene expression, Cancer research, Cell culture and Downregulation and upregulation.

His most cited work include:

  • STATs: transcriptional control and biological impact (2441 citations)
  • IL-6 programs T(H)-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways. (1701 citations)
  • Targeted Disruption of the Mouse Stat1 Gene Results in Compromised Innate Immunity to Viral Disease (1378 citations)

What are the main themes of his work throughout his whole career to date?

Cell biology, Molecular biology, Transcription factor, STAT3 and Interferon are his primary areas of study. The concepts of his Cell biology study are interwoven with issues in Regulation of gene expression and Gene expression. His Molecular biology study combines topics in areas such as Interferon-Stimulated Gene Factor 3, Transcription, Gene, STAT1 and Response element.

His research on Transcription factor focuses in particular on Interferon regulatory factors. David E. Levy works mostly in the field of STAT3, limiting it down to topics relating to Cancer research and, in certain cases, Tyrosine phosphorylation, as a part of the same area of interest. Virology and Immunology are the focus of his Interferon studies.

He most often published in these fields:

  • Cell biology (23.84%)
  • Molecular biology (22.29%)
  • Transcription factor (15.79%)

What were the highlights of his more recent work (between 2010-2021)?

  • Cell biology (23.84%)
  • Cancer research (12.38%)
  • STAT3 (16.10%)

In recent papers he was focusing on the following fields of study:

David E. Levy mainly focuses on Cell biology, Cancer research, STAT3, Transcription factor and Particle swarm optimization. The Cell biology study which covers Gene expression that intersects with Molecular biology. His studies deal with areas such as RNA polymerase II and STAT1 as well as Molecular biology.

He interconnects Cancer, Prostate cancer, T cell, Phosphorylation and Tumor initiation in the investigation of issues within Cancer research. He studies STAT protein, a branch of STAT3. Within one scientific family, David E. Levy focuses on topics pertaining to Signal transduction under Transcription factor, and may sometimes address concerns connected to Interferon.

Between 2010 and 2021, his most popular works were:

  • Chemokine Gene Silencing in Decidual Stromal Cells Limits T Cell Access to the Maternal-Fetal Interface (277 citations)
  • Constitutive Type I Interferon Modulates Homeostatic Balance through Tonic Signaling (274 citations)
  • STAT3 Plays a Critical Role in KRAS-Induced Pancreatic Tumorigenesis (273 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Genetics

David E. Levy focuses on Cancer research, Cell biology, STAT3, Immunology and STAT protein. His Cancer research research includes elements of Receptor, T cell, Cancer and Phosphorylation. His Cell biology study integrates concerns from other disciplines, such as Chromatin, IL-2 receptor and Transplantation.

His work carried out in the field of STAT3 brings together such families of science as Cancer cell, Cytotoxic T cell, Interleukin 21 and Myeloproliferative Disorders. His Immunology study combines topics from a wide range of disciplines, such as Signal transduction, Cellular differentiation and Transcription factor. His research integrates issues of Response element and RNA polymerase II in his study of Transcription factor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

STATs: transcriptional control and biological impact

David E. Levy;J. E. Darnell.
Nature Reviews Molecular Cell Biology (2002)

3386 Citations

IL-6 programs T(H)-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways.

Liang Zhou;Ivaylo I Ivanov;Rosanne Spolski;Roy Min.
Nature Immunology (2007)

2192 Citations

Targeted Disruption of the Mouse Stat1 Gene Results in Compromised Innate Immunity to Viral Disease

Joan E Durbin;Renée Hackenmiller;M.Celeste Simon;David E Levy.
Cell (1996)

1806 Citations

Gr-1+CD115+ Immature Myeloid Suppressor Cells Mediate the Development of Tumor-Induced T Regulatory Cells and T-Cell Anergy in Tumor-Bearing Host

Bo Huang;Ping Ying Pan;Qingsheng Li;Alice I. Sato.
Cancer Research (2006)

1417 Citations

Differential viral induction of distinct interferon-α genes by positive feedback through interferon regulatory factor-7

Isabelle Marié;Joan E. Durbin;David E. Levy.
The EMBO Journal (1998)

1240 Citations

JAK-STAT Signaling: From Interferons to Cytokines

Christian Schindler;David E. Levy;Thomas Decker.
Journal of Biological Chemistry (2007)

1236 Citations

Influenza A Virus Lacking the NS1 Gene Replicates in Interferon-Deficient Systems

Adolfo García-Sastre;Andrej Egorov;Demetrius Matassov;Sabine Brandt.
Virology (1998)

1181 Citations

What does Stat3 do

David E. Levy;Chien-kuo Lee.
Journal of Clinical Investigation (2002)

1111 Citations

Mitochondrial STAT3 supports Ras-dependent oncogenic transformation.

Daniel J. Gough;Alicia Corlett;Karni Schlessinger;Joanna Wegrzyn.
Science (2009)

654 Citations

Stat3 and Stat4 Direct Development of IL-17-Secreting Th Cells

Anubhav N. Mathur;Hua Chen Chang;Dimitrios G. Zisoulis;Gretta L. Stritesky.
Journal of Immunology (2007)

633 Citations

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