2009 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary scientific interests are in Molecular biology, Cell biology, Transcription factor, Signal transduction and STAT3. His Molecular biology research is multidisciplinary, relying on both Interferon-Stimulated Gene Factor 3, Cellular differentiation, Alpha interferon, Interferon regulatory factors and Response element. His Cell biology research incorporates themes from Perforin and Interleukin 21.
The Transcription factor study combines topics in areas such as Interferon, Cell nucleus and Epigenetics. He combines subjects such as Virology, stat and Phosphorylation with his study of Signal transduction. His STAT3 research integrates issues from Regulation of gene expression, Cancer research, Cell culture and Downregulation and upregulation.
Cell biology, Molecular biology, Transcription factor, STAT3 and Interferon are his primary areas of study. The concepts of his Cell biology study are interwoven with issues in Regulation of gene expression and Gene expression. His Molecular biology study combines topics in areas such as Interferon-Stimulated Gene Factor 3, Transcription, Gene, STAT1 and Response element.
His research on Transcription factor focuses in particular on Interferon regulatory factors. David E. Levy works mostly in the field of STAT3, limiting it down to topics relating to Cancer research and, in certain cases, Tyrosine phosphorylation, as a part of the same area of interest. Virology and Immunology are the focus of his Interferon studies.
David E. Levy mainly focuses on Cell biology, Cancer research, STAT3, Transcription factor and Particle swarm optimization. The Cell biology study which covers Gene expression that intersects with Molecular biology. His studies deal with areas such as RNA polymerase II and STAT1 as well as Molecular biology.
He interconnects Cancer, Prostate cancer, T cell, Phosphorylation and Tumor initiation in the investigation of issues within Cancer research. He studies STAT protein, a branch of STAT3. Within one scientific family, David E. Levy focuses on topics pertaining to Signal transduction under Transcription factor, and may sometimes address concerns connected to Interferon.
David E. Levy focuses on Cancer research, Cell biology, STAT3, Immunology and STAT protein. His Cancer research research includes elements of Receptor, T cell, Cancer and Phosphorylation. His Cell biology study integrates concerns from other disciplines, such as Chromatin, IL-2 receptor and Transplantation.
His work carried out in the field of STAT3 brings together such families of science as Cancer cell, Cytotoxic T cell, Interleukin 21 and Myeloproliferative Disorders. His Immunology study combines topics from a wide range of disciplines, such as Signal transduction, Cellular differentiation and Transcription factor. His research integrates issues of Response element and RNA polymerase II in his study of Transcription factor.
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STATs: transcriptional control and biological impact
David E. Levy;J. E. Darnell.
Nature Reviews Molecular Cell Biology (2002)
IL-6 programs T(H)-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways.
Liang Zhou;Ivaylo I Ivanov;Rosanne Spolski;Roy Min.
Nature Immunology (2007)
Targeted Disruption of the Mouse Stat1 Gene Results in Compromised Innate Immunity to Viral Disease
Joan E Durbin;Renée Hackenmiller;M.Celeste Simon;David E Levy.
Gr-1+CD115+ Immature Myeloid Suppressor Cells Mediate the Development of Tumor-Induced T Regulatory Cells and T-Cell Anergy in Tumor-Bearing Host
Bo Huang;Ping Ying Pan;Qingsheng Li;Alice I. Sato.
Cancer Research (2006)
Differential viral induction of distinct interferon-α genes by positive feedback through interferon regulatory factor-7
Isabelle Marié;Joan E. Durbin;David E. Levy.
The EMBO Journal (1998)
JAK-STAT Signaling: From Interferons to Cytokines
Christian Schindler;David E. Levy;Thomas Decker.
Journal of Biological Chemistry (2007)
Influenza A Virus Lacking the NS1 Gene Replicates in Interferon-Deficient Systems
Adolfo García-Sastre;Andrej Egorov;Demetrius Matassov;Sabine Brandt.
What does Stat3 do
David E. Levy;Chien-kuo Lee.
Journal of Clinical Investigation (2002)
Mitochondrial STAT3 supports Ras-dependent oncogenic transformation.
Daniel J. Gough;Alicia Corlett;Karni Schlessinger;Joanna Wegrzyn.
Stat3 and Stat4 Direct Development of IL-17-Secreting Th Cells
Anubhav N. Mathur;Hua Chen Chang;Dimitrios G. Zisoulis;Gretta L. Stritesky.
Journal of Immunology (2007)
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