What is she best known for?
The fields of study she is best known for:
Her primary areas of investigation include STAT3, Immunology, Interleukin 6, Molecular biology and Cell biology.
Her STAT3 research includes themes of Caspase and Liver regeneration.
Her Interleukin 6 research integrates issues from Receptor, Endocrinology, Genetically modified mouse and Arthritis.
Her biological study spans a wide range of topics, including Inflammation and Transgene.
Her study on Molecular biology also encompasses disciplines like
- Gene expression which intersects with area such as Alpha, Gene targeting, Beta and Gene isoform,
- Regulation of gene expression, which have a strong connection to Ccaat-enhancer-binding proteins and Nuclear protein.
Her Signal transduction study incorporates themes from Cancer research and Mitochondrion.
Her most cited work include:
- Liver Failure and Defective Hepatocyte Regeneration in Interleukin-6-Deficient Mice (1288 citations)
- Role of IL-6 and Its Soluble Receptor in Induction of Chemokines and Leukocyte Recruitment (911 citations)
- Interleukin-6 deficient mice are protected from bone loss caused by estrogen depletion. (672 citations)
What are the main themes of her work throughout her whole career to date?
Her primary areas of study are STAT3, Cancer research, Cell biology, Immunology and Transcription factor.
Her research on STAT3 often connects related areas such as STAT1.
As a member of one scientific family, Valeria Poli mostly works in the field of Cancer research, focusing on Metastasis and, on occasion, Tumor progression.
Interleukin 6, Inflammation and Immune system are the subjects of her Immunology studies.
Her Interleukin 6 research incorporates elements of Interleukin and Lipopolysaccharide, Endocrinology.
Her studies deal with areas such as Molecular biology, Regulation of gene expression, Kinase and Gene isoform as well as Transcription factor.
She most often published in these fields:
- STAT3 (45.95%)
- Cancer research (34.05%)
- Cell biology (25.41%)
What were the highlights of her more recent work (between 2017-2021)?
- STAT3 (45.95%)
- Cancer research (34.05%)
- Cell biology (25.41%)
In recent papers she was focusing on the following fields of study:
Valeria Poli mainly focuses on STAT3, Cancer research, Cell biology, Transcription factor and Breast cancer.
She studies STAT3, namely STAT protein.
The concepts of her STAT protein study are interwoven with issues in Psoriasis, Autoimmunity, Janus kinase and Bioinformatics.
The Cancer research study combines topics in areas such as Cancer, Primary tumor, Metastasis and Brain metastasis.
Her Cell biology research incorporates themes from Transcriptional repressor complex and Cell cycle.
Valeria Poli has included themes like Signal transducing adaptor protein, Targeted therapy, Neuroblastoma, Computational biology and Gene regulatory network in her Breast cancer study.
Between 2017 and 2021, her most popular works were:
- STAT3 labels a subpopulation of reactive astrocytes required for brain metastasis. (110 citations)
- Psoriasis: A STAT3-Centric View (54 citations)
- STAT3 localizes to the ER, acting as a gatekeeper for ER-mitochondrion Ca 2+ fluxes and apoptotic responses (47 citations)
In her most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in STAT3, STAT protein, Cancer research, Cell biology and Transcription factor.
Valeria Poli interconnects Psoriasis, Primary tumor and Janus kinase in the investigation of issues within STAT3.
Her study in STAT protein is interdisciplinary in nature, drawing from both Acquired immune system, Lesion, Brain metastasis, Tumor microenvironment and Central nervous system.
Her research integrates issues of Cancer, Gene expression, T cell, Blockade and Immunity in her study of Cancer research.
The various areas that Valeria Poli examines in her Cell biology study include Oxidative stress and TFEB.
The study incorporates disciplines such as Endoplasmic reticulum, Phosphorylation, Mitochondrion, Energy homeostasis and Gene isoform in addition to Transcription factor.
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