What is he best known for?
The fields of study he is best known for:
- Gene
- Cytokine
- Immune system
His primary scientific interests are in Inflammation, Immunology, Interleukin 6, Cell biology and Receptor.
The concepts of his Inflammation study are interwoven with issues in Macrophage, Innate immune system, Autoimmunity and Infiltration.
Simon Arnett Jones has included themes like Cancer and Blockade in his Immunology study.
His research in Interleukin 6 intersects with topics in Acquired immune system, Immune system, Interleukin 10 and Interleukin.
His studies in Cell biology integrate themes in fields like Glucocorticoid receptor, Glucocorticoid, Cytokine and COS cells.
His Receptor research is multidisciplinary, incorporating perspectives in Molecular biology, Jurkat cells and Interleukin 8.
His most cited work include:
- Chemokine receptor specific for IP10 and mig: structure, function, and expression in activated T-lymphocytes. (1078 citations)
- IL-6 as a keystone cytokine in health and disease (1032 citations)
- IL-6 and Its Soluble Receptor Orchestrate a Temporal Switch in the Pattern of Leukocyte Recruitment Seen during Acute Inflammation (650 citations)
What are the main themes of his work throughout his whole career to date?
Simon Arnett Jones spends much of his time researching Immunology, Interleukin 6, Inflammation, Receptor and Cytokine.
His study in Immunology concentrates on Immune system, Arthritis, Acquired immune system, Chemokine and Immunity.
His work is dedicated to discovering how Interleukin 6, Interleukin are connected with Interleukin 10 and other disciplines.
His research integrates issues of Innate immune system, Macrophage, Monocyte and Peritonitis in his study of Inflammation.
His work deals with themes such as Molecular biology, Endocrinology, Signal transduction and Jurkat cells, which intersect with Receptor.
Simon Arnett Jones has researched Cytokine in several fields, including Cancer research, CCL5, Pathogenesis and Virology.
He most often published in these fields:
- Immunology (53.90%)
- Interleukin 6 (32.47%)
- Inflammation (27.92%)
What were the highlights of his more recent work (between 2014-2020)?
- Immunology (53.90%)
- Interleukin 6 (32.47%)
- Cytokine (20.78%)
In recent papers he was focusing on the following fields of study:
Immunology, Interleukin 6, Cytokine, Immune system and Inflammation are his primary areas of study.
His Interleukin 6 study combines topics from a wide range of disciplines, such as Arthritis, Tocilizumab, Disease, Liver X receptor and Interleukin.
Simon Arnett Jones combines subjects such as Viral disease, Cellular immunity, Signal transduction, Viral pathogenesis and Autoimmunity with his study of Cytokine.
His research investigates the connection between Signal transduction and topics such as Tyrosine that intersect with issues in Cell biology.
The study incorporates disciplines such as Pathogenesis and Effector in addition to Immune system.
His Oncostatin M study in the realm of Inflammation connects with subjects such as Vascular endothelial growth factor.
Between 2014 and 2020, his most popular works were:
- IL-6 as a keystone cytokine in health and disease (1032 citations)
- Recent insights into targeting the IL-6 cytokine family in inflammatory diseases and cancer (201 citations)
- On-going Mechanical Damage from Mastication Drives Homeostatic Th17 Cell Responses at the Oral Barrier. (90 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Cytokine
- Immune system
Simon Arnett Jones mainly investigates Immunology, Inflammation, Immune system, Interleukin 6 and Interleukin.
Simon Arnett Jones works in the field of Immunology, namely Chemokine.
His studies deal with areas such as Receptor, Tube formation and Peritoneum as well as Inflammation.
His Immune system study integrates concerns from other disciplines, such as Cell biology and Effector.
Interleukin 6 is a subfield of Cytokine that he studies.
His work carried out in the field of Interleukin brings together such families of science as Synovitis, Inflammatory arthritis and Arthritis.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
