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D-Index & Metrics

Biology and Biochemistry

D-Index
78
Citations
19101
World Ranking
4611
National Ranking
36

Overview

Richard Moriggl is affiliated with the University of Veterinary Medicine Vienna in Austria. Their research spans multiple fields with a focus on Medicine and Biochemistry, Genetics and Molecular Biology. Within these disciplines, their subfields of study include Molecular Biology, Oncology, Immunology, Hematology, and Genetics.

The main topics explored in their work encompass Acute Myeloid Leukemia Research, Immune Cell Function and Interaction, Cytokine Signaling Pathways and Interactions, Histone Deacetylase Inhibitors Research, Protein Degradation and Inhibitors, Chronic Lymphocytic Leukemia Research, and Lymphoma Diagnosis and Treatment.

Moriggl's recent papers include:

  • Advances in covalent kinase inhibitors, 2020, Chemical Society Reviews
  • Proposed refined diagnostic criteria and classification of eosinophil disorders and related syndromes, 2022, Allergy
  • JAK-STAT core cancer pathway: An integrative cancer interactome analysis, 2022, Journal of Cellular and Molecular Medicine
  • CDK6 is an essential direct target of NUP98 fusion proteins in acute myeloid leukemia, 2020, Blood
  • JAK-STAT signaling maintains homeostasis in T cells and macrophages, 2024, Nature Immunology

Their frequent co-authors are:

  • Heidi A. Neubauer
  • Lukas Kenner
  • Patrick T. Gunning
  • Anna Orlova
  • Elvin D. de Araujo

The most common publication venues include:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Cancers
  • Blood
  • Cancer Research
  • Molecular Cancer

Best Publications

  • Nonredundant roles for Stat5a/b in directly regulating Foxp3

    Zhengju Yao;Zhengju Yao;Yuka Kanno;Marc Kerenyi;Geoffrey Stephens

  • Stat5 Is Required for IL-2-Induced Cell Cycle Progression of Peripheral T Cells

    Richard Moriggl;David J Topham;Stephan Teglund;Veronika Sexl

  • Persistent STAT3 activation in colon cancer is associated with enhanced cell proliferation and tumor growth.

    Florian M. Corvinus;Carina Orth;Richard Moriggl;Svetlana A. Tsareva

  • Autocrine PDGFR signaling promotes mammary cancer metastasis

    Martin Jechlinger;Andreas Sommer;Richard Moriggl;Peter Seither

  • A kinase-independent function of CDK6 links the cell cycle to tumor angiogenesis.

    Karoline Kollmann;Gerwin Heller;Christine Schneckenleithner;Wolfgang Warsch

  • Deletion of the carboxyl-terminal transactivation domain of MGF-Stat5 results in sustained DNA binding and a dominant negative phenotype.

    R Moriggl;V Gouilleux-Gruart;R Jähne;S Berchtold

  • Macrophages and neutrophils are the targets for immune suppression by glucocorticoids in contact allergy

    Jan P. Tuckermann;Anna Kleiman;Anna Kleiman;Richard Moriggl;Rainer Spanbroek

  • Stat5 tetramer formation is associated with leukemogenesis

    Richard Moriggl;Veronika Sexl;Lukas Kenner;Christopher Duntsch

  • Stat5 is indispensable for the maintenance of bcr/abl-positive leukaemia.

    Andrea Hoelbl;Christian Schuster;Boris Kovacic;Bingmei Zhu

  • Bone homeostasis in growth hormone receptor–null mice is restored by IGF-I but independent of Stat5

    Natalie A. Sims;Philippe Clément-Lacroix;Francesca Da Ponte;Yasmina Bouali

  • Clarifying the role of Stat5 in lymphoid development and Abelson-induced transformation

    Andrea Hoelbl;Boris Kovacic;Marc A. Kerenyi;Olivia Simma

  • Stat5a/b contribute to interleukin 7-induced B-cell precursor expansion, but abl- and bcr/abl-induced transformation are independent of stat5.

    Veronika Sexl;Roland Piekorz;Richard Moriggl;Juerg Rohrer

  • Epidermal Growth Factor Receptor Signaling Synergizes with Hedgehog/GLI in Oncogenic Transformation via Activation of the MEK/ERK/JUN Pathway

    Harald Schnidar;Markus Eberl;Stefan Klingler;Doris Mangelberger

  • Stat5 Activation Is Uniquely Associated with Cytokine Signaling in Peripheral T Cells

    Richard Moriggl;Veronika Sexl;Roland Piekorz;David Topham

  • Identification of mcl-1 as a BCR/ABL-dependent target in chronic myeloid leukemia (CML): evidence for cooperative antileukemic effects of imatinib and mcl-1 antisense oligonucleotides

    Karl J. Aichberger;Matthias Mayerhofer;Maria-Theresa Krauth;Hans Skvara

  • Antiapoptotic activity of Stat5 required during terminal stages of myeloid differentiation

    Matthias Kieslinger;Irina Woldman;Richard Moriggl;Johannes Hofmann

  • Cancer-associated fibroblast-derived WNT2 increases tumor angiogenesis in colon cancer

    Daniela Unterleuthner;Patrick Neuhold;Katharina Schwarz;Lukas Janker

  • JAK-STAT signaling in cancer: From cytokines to non-coding genome.

    Jan Pencik;Ha Thi Thanh Pham;Johannes Schmoellerl;Tahereh Javaheri

  • High STAT5 levels mediate imatinib resistance and indicate disease progression in chronic myeloid leukemia.

    Wolfgang Warsch;Karoline Kollmann;Eva Eckelhart;Sabine Fajmann

  • Specific DNA binding of Stat5, but not of glucocorticoid receptor, is required for their functional cooperation in the regulation of gene transcription.

    Elisabeth Stoecklin;Manuela Wissler;Richard Moriggl;Bernd Groner

Frequent Co-Authors

Veronika Sexl
Veronika Sexl University of Innsbruck
Peter Valent
Peter Valent Medical University of Vienna
Hartmut Beug
Hartmut Beug Research Institute of Molecular Pathology
Fabrice Gouilleux
Fabrice Gouilleux François Rabelais University
Mathias Müller
Mathias Müller University of Veterinary Medicine Vienna
Christoph Bock
Christoph Bock Austrian Academy of Sciences
James N. Ihle
James N. Ihle St. Jude Children's Research Hospital
Markus Hengstschläger
Markus Hengstschläger Medical University of Vienna
Harald Esterbauer
Harald Esterbauer Medical University of Vienna
Lothar Hennighausen
Lothar Hennighausen National Institutes of Health

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