His main research concerns Molecular biology, STAT protein, Transcription factor, STAT3 and STAT5. His Molecular biology research includes themes of Promoter, Transcription, Gene expression, Gene and Antibody. His STAT protein research integrates issues from STAT4 and Transactivation.
His work deals with themes such as Hormone response element, Glucocorticoid receptor and Cell biology, which intersect with Transcription factor. His STAT3 study integrates concerns from other disciplines, such as Carcinogenesis and Cancer research. The various areas that Bernd Groner examines in his Cancer research study include Signal transduction and Epithelial cell differentiation.
Bernd Groner mainly investigates Molecular biology, Cancer research, Cell biology, STAT protein and Transcription factor. His biological study spans a wide range of topics, including Transcription, Gene, Transactivation, Receptor and Promoter. He has researched Cancer research in several fields, including Cancer cell, Mammary gland, Signal transduction, Fusion protein and Cytotoxic T cell.
His research integrates issues of Internal medicine, Stromal cell, Cellular differentiation and Endocrinology in his study of Cell biology. His study in STAT protein is interdisciplinary in nature, drawing from both STAT4, stat and STAT5. His Transcription factor study combines topics in areas such as Regulation of gene expression, General transcription factor and Tyrosine phosphorylation.
His primary areas of study are Cell biology, Cancer research, STAT3, STAT protein and Cancer cell. His work carried out in the field of Cell biology brings together such families of science as Cytokine, stat, Suppressor of cytokine signaling 1 and SOX2, KLF4. His Cancer research research incorporates themes from Cell signaling, Signal transduction, Cancer stem cell, Stem cell and Breast cancer.
The study incorporates disciplines such as Gene knockdown, Tumor microenvironment, Immunology, Immune system and Cytotoxic T cell in addition to STAT3. Bernd Groner combines subjects such as JAK-STAT signaling pathway, STAT5 and Transactivation with his study of STAT protein. His studies in STAT5 integrate themes in fields like Metastatic breast cancer, Pathology, Molecular biology, Reporter gene and Tumor initiation.
His primary scientific interests are in Cell biology, STAT3, Cell migration, STAT protein and Cancer stem cell. As a part of the same scientific study, Bernd Groner usually deals with the Cell biology, concentrating on Transplantation and frequently concerns with Endocrinology. The STAT3 study combines topics in areas such as Tumor microenvironment, Cancer research, Infiltration and Gene knockdown.
His work investigates the relationship between STAT protein and topics such as Breast cancer that intersect with problems in microRNA. His research in Cancer stem cell intersects with topics in CD44, Mammary tumor and Adult stem cell. His Signal transduction research is multidisciplinary, relying on both Carcinogenesis and Downregulation and upregulation.
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Three-dimensional structure of the Stat3β homodimer bound to DNA
Stefan Becker;Bernd Groner;Christoph W. Müller.
Functional interactions between Stat5 and the glucocorticoid receptor
Elisabeth Stöcklin;Manuela Wissler;Fabrice Gouilleux;Bernd Groner.
Cloning and expression of Stat5 and an additional homologue (Stat5b) involved in prolactin signal transduction in mouse mammary tissue
Xiuwen Liu;Gertraud W. Robinson;Fabrice Gouilleux;Bernd Groner.
Proceedings of the National Academy of Sciences of the United States of America (1995)
STAT-related transcription factors are constitutively activated in peripheral blood cells from acute leukemia patients
Valerie Gouilleux-Gruart;Fabrice Gouilleux;Corinne Desaint;Jean-Francois Claisse.
Prolactin and glucocorticoid hormones synergistically induce expression of transfected rat beta-casein gene promoter constructs in a mammary epithelial cell line.
Wolfgang Doppler;Bernd Groner;Roland K. Ball.
Proceedings of the National Academy of Sciences of the United States of America (1989)
p300/CREB-Binding Protein Enhances the Prolactin-Mediated Transcriptional Induction through Direct Interaction with the Transactivation Domain of Stat5, but Does Not Participate in the Stat5-Mediated Suppression of the Glucocorticoid Response
Edith Pfitzner;Ruth Jähne;Manuela Wissler;Elisabeth Stoecklin.
Molecular Endocrinology (1998)
Deletion of the carboxyl-terminal transactivation domain of MGF-Stat5 results in sustained DNA binding and a dominant negative phenotype.
R Moriggl;V Gouilleux-Gruart;R Jähne;S Berchtold.
Molecular and Cellular Biology (1996)
Cytotoxic T lymphocytes with a grafted recognition specificity for ERBB2-expressing tumor cells.
Dirk Moritz;Winfried Wels;Jurgen Mattern;Bernd Groner.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Ha-ras oncogene expression directed by a milk protein gene promoter: tissue specificity, hormonal regulation, and tumor induction in transgenic mice.
Anne-Catherine Andres;Cora-Ann Schonenberger;Bernd Groner;Lothar Hennighausen.
Proceedings of the National Academy of Sciences of the United States of America (1987)
Selective Inhibition of Tumor Cell Growth by a Recombinant Single-Chain Antibody-Toxin Specific for the erbB-2 Receptor
Winfried Wels;Ina-Maria Harwerth;Marcel Mueller;Bernd Groner.
Cancer Research (1992)
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