Jonathan L.C. Lee mainly focuses on Memory consolidation, Neuroscience, Stimulus, Basolateral amygdala and Hippocampal formation. His Memory consolidation research incorporates elements of Cognitive psychology, Memoria, Extinction, Addiction and Amygdala. As a part of the same scientific study, Jonathan L.C. Lee usually deals with the Memoria, concentrating on Neuroplasticity and frequently concerns with Avoidance learning.
His Addiction research is multidisciplinary, relying on both Classical conditioning and Drug. His Neuroscience research focuses on Fear conditioning in particular. The concepts of his Hippocampal formation study are interwoven with issues in Hippocampus and Amnesia.
Jonathan L.C. Lee mostly deals with Memory consolidation, Neuroscience, NMDA receptor, Classical conditioning and Extinction. His studies deal with areas such as Cognitive psychology, Addiction, Drug, Drug seeking and Amygdala as well as Memory consolidation. As a part of the same scientific family, Jonathan L.C. Lee mostly works in the field of Neuroscience, focusing on Amnesia and, on occasion, Dopaminergic.
His NMDA receptor research incorporates themes from Glutamate receptor, Psychosis, Antagonist and Dopamine. His research investigates the connection between Classical conditioning and topics such as Conditioned place preference that intersect with issues in Retrograde amnesia. Jonathan L.C. Lee combines subjects such as Conditioning and Anxiety with his study of Extinction.
His scientific interests lie mostly in Memory consolidation, Neuroscience, Nefiracetam, NMDA receptor and Protein degradation. His work investigates the relationship between Memory consolidation and topics such as Dopamine receptor D1 that intersect with problems in Extinction. His Amnesia research extends to Neuroscience, which is thematically connected.
His Amnesia research includes themes of Addiction, Yohimbine and Priming. His work deals with themes such as Cued speech and Nootropic, which intersect with Facilitation. Jonathan L.C. Lee integrates Visual perception and Stimulus in his studies.
Jonathan L.C. Lee mainly investigates Memory consolidation, Neuroscience, Amnesia, NMDA receptor and Extinction. His is doing research in Priming and Amygdala, both of which are found in Neuroscience. His study in Priming is interdisciplinary in nature, drawing from both Antagonist, Yohimbine and Addiction.
His Amygdala study combines topics from a wide range of disciplines, such as AMPA receptor, Hippocampus and Cannabinoid receptor. His Extinction study combines topics in areas such as Dopaminergic, Dopamine receptor D1 and Nefiracetam.
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Independent Cellular Processes for Hippocampal Memory Consolidation and Reconsolidation
Jonathan L.C. Lee;Barry J. Everitt;Kerrie Lorraine Thomas.
Science (2004)
Reconsolidation and Extinction of Conditioned Fear: Inhibition and Potentiation
Jonathan L. C. Lee;Amy L. Milton;Barry J. Everitt.
The Journal of Neuroscience (2006)
Reconsolidation: maintaining memory relevance
Jonathan L.C. Lee.
Trends in Neurosciences (2009)
Disrupting Reconsolidation of Drug Memories Reduces Cocaine-Seeking Behavior
Jonathan L. C. Lee;Patricia Di Ciano;Kerrie Lorraine Thomas;Barry J. Everitt.
Neuron (2005)
Memory reconsolidation mediates the strengthening of memories by additional learning.
Jonathan L C Lee.
Nature Neuroscience (2008)
Cue-Induced Cocaine Seeking and Relapse Are Reduced by Disruption of Drug Memory Reconsolidation
Jonathan L. C. Lee;Amy L. Milton;Barry J. Everitt.
The Journal of Neuroscience (2006)
Intra-Amygdala and Systemic Antagonism of NMDA Receptors Prevents the Reconsolidation of Drug-Associated Memory and Impairs Subsequently Both Novel and Previously Acquired Drug-Seeking Behaviors
Amy L. Milton;Jonathan L. C. Lee;Victoria J. Butler;Richard Gardner.
The Journal of Neuroscience (2008)
An update on memory reconsolidation updating
Jonathan L.C. Lee;Karim Nader;Daniela Schiller.
Trends in Cognitive Sciences (2017)
Memory Reconsolidation Mediates the Updating of Hippocampal Memory Content
Jonathan L C Lee.
Frontiers in Behavioral Neuroscience (2010)
Reconsolidation of appetitive memories for both natural and drug reinforcement is dependent on β-adrenergic receptors
Amy L. Milton;Jonathan L.C. Lee;Barry J. Everitt.
Learning & Memory (2008)
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