Richard Jove focuses on Cancer research, STAT3, STAT protein, Signal transduction and Proto-oncogene tyrosine-protein kinase Src. His Cancer research research incorporates themes from Protein kinase B, Carcinogenesis, Tyrosine kinase and Kinase, Kinase activity. STAT3 is the subject of his research, which falls under Cell biology.
His STAT protein research is multidisciplinary, incorporating elements of Inhibitor of apoptosis, Transcription factor, SOCS3, Immunology and Tyrosine phosphorylation. The Signal transduction study combines topics in areas such as Microarray analysis techniques, Protein structure, Breast cancer and Peptidomimetic, Peptide. Ectopic expression is closely connected to Platelet-derived growth factor receptor in his research, which is encompassed under the umbrella topic of Proto-oncogene tyrosine-protein kinase Src.
The scientist’s investigation covers issues in Cancer research, STAT3, Cell biology, Proto-oncogene tyrosine-protein kinase Src and Signal transduction. His Cancer research research integrates issues from Cell growth, Carcinogenesis, Tyrosine kinase, Apoptosis and Kinase. The various areas that he examines in his STAT3 study include Transcription factor, STAT5 and Tyrosine phosphorylation.
In his study, Fibronectin is inextricably linked to Cell adhesion, which falls within the broad field of Cell biology. His Proto-oncogene tyrosine-protein kinase Src research incorporates elements of Molecular biology, Epidermal growth factor and Kinase activity. Richard Jove focuses mostly in the field of STAT protein, narrowing it down to topics relating to STAT6 and, in certain cases, STAT1.
Richard Jove mainly investigates Cancer research, STAT3, Cell biology, Signal transduction and Kinase. His Cancer research study combines topics from a wide range of disciplines, such as Cancer, Janus kinase and Androgen receptor. He studies STAT3, focusing on STAT protein in particular.
His work on Proto-oncogene tyrosine-protein kinase Src, Cancer stem cell and Effector as part of his general Cell biology study is frequently connected to Karyopherin, thereby bridging the divide between different branches of science. His Signal transduction research is multidisciplinary, incorporating perspectives in Viability assay, Apoptosis, Berbamine and Protein kinase A. His studies in Kinase integrate themes in fields like Cell culture, Protein kinase B and Nucleophilic substitution.
Richard Jove spends much of his time researching Cancer research, Signal transduction, Cell biology, STAT3 and Angiogenesis. Many of his studies on Cancer research involve topics that are commonly interrelated, such as Cell culture. He works in the field of Signal transduction, namely Kinase activity.
His biological study focuses on STAT protein. Within one scientific family, Richard Jove focuses on topics pertaining to Regulation of gene expression under STAT protein, and may sometimes address concerns connected to Janus kinase. His studies deal with areas such as Tyrosine kinase, Biochemistry and Protein kinase A as well as Angiogenesis.
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The STATs of cancer — new molecular targets come of age
Hua Yu;Richard Jove.
Nature Reviews Cancer (2004)
STATs in oncogenesis.
Tammy Bowman;Roy Garcia;James Turkson;Richard Jove.
Constitutive Activation of Stat3 Signaling Confers Resistance to Apoptosis in Human U266 Myeloma Cells
Robyn Catlett-Falcone;Terry H Landowski;Marc M Oshiro;James Turkson.
Revisiting STAT3 signalling in cancer: new and unexpected biological functions
Hua Yu;Heehyoung Lee;Andreas Herrmann;Ralf Buettner.
Nature Reviews Cancer (2014)
Constitutive Stat3 activity up-regulates VEGF expression and tumor angiogenesis
Guilian Niu;Kenneth L Wright;Mei Huang;Lanxi Song.
Activated STAT Signaling in Human Tumors Provides Novel Molecular Targets for Therapeutic Intervention
Ralf Buettner;Linda B Mora;Richard Jove.
Clinical Cancer Research (2002)
Regulation of the innate and adaptive immune responses by Stat-3 signaling in tumor cells.
Tianhong Wang;Guilian Niu;Marcin Kortylewski;Lyudmila Burdelya.
Nature Medicine (2004)
Stat3 Activation by Src Induces Specific Gene Regulation and Is Required for Cell Transformation
James Turkson;Tammy Bowman;Roy Garcia;Eric Caldenhoven.
Molecular and Cellular Biology (1998)
Constitutive activation of Stat3 by the Src and JAK tyrosine kinases participates in growth regulation of human breast carcinoma cells.
Roy Garcia;Tammy L Bowman;Guilian Niu;Hua Yu.
Inhibition of STAT3 signaling leads to apoptosis of leukemic large granular lymphocytes and decreased Mcl-1 expression
P. K. Epling-Burnette;Jin Hong Liu;Robyn Catlett-Falcone;James Turkson.
Journal of Clinical Investigation (2001)
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