What is he best known for?
The fields of study he is best known for:
Charles P. Emerson mainly focuses on Cell biology, Somite, Molecular biology, Sonic hedgehog and Myogenesis.
His Cell biology study integrates concerns from other disciplines, such as Genetics, Biochemistry and SULF1.
His Somite research includes elements of Paraxial mesoderm, Mesoderm, MyoD, MYF5 and Notochord.
His Molecular biology study incorporates themes from Embryonic stem cell, Transcription factor and Stem cell.
In Sonic hedgehog, Charles P. Emerson works on issues like Hedgehog signaling pathway, which are connected to Protein kinase A, Protein kinase C, Mitogen-activated protein kinase and MAPK/ERK pathway.
Charles P. Emerson is interested in Myogenin, which is a field of Myogenesis.
His most cited work include:
- Myogenic regulatory factors and the specification of muscle progenitors in vertebrate embryos. (475 citations)
- Regulation of Wnt Signaling and Embryo Patterning by an Extracellular Sulfatase (388 citations)
- QSulf1 remodels the 6-O sulfation states of cell surface heparan sulfate proteoglycans to promote Wnt signaling (361 citations)
What are the main themes of his work throughout his whole career to date?
Charles P. Emerson focuses on Cell biology, Genetics, Molecular biology, Gene and Myogenesis.
His Cell biology research incorporates elements of Embryonic stem cell and Cellular differentiation.
His Genetics research focuses on Myosin and how it relates to Skeletal muscle, Major histocompatibility complex and Drosophila melanogaster.
His biological study spans a wide range of topics, including Enhancer, Gene expression, Complementary DNA, Messenger RNA and Regulation of gene expression.
His studies in Myogenesis integrate themes in fields like Cancer research and PAX3.
His research in Signal transduction intersects with topics in Endocrinology, SULF1 and Internal medicine.
He most often published in these fields:
- Cell biology (49.59%)
- Genetics (40.65%)
- Molecular biology (27.64%)
What were the highlights of his more recent work (between 2011-2019)?
- Facioscapulohumeral muscular dystrophy (22.76%)
- Genetics (40.65%)
- DUX4 (15.45%)
In recent papers he was focusing on the following fields of study:
Charles P. Emerson mainly investigates Facioscapulohumeral muscular dystrophy, Genetics, DUX4, Cell biology and Muscular dystrophy.
His Facioscapulohumeral muscular dystrophy research incorporates themes from Cancer research, Myopathy, Disease, Skeletal muscle and Epigenetics.
His Cancer research study combines topics from a wide range of disciplines, such as Myogenesis, Somite and GLI2.
His DUX4 research is multidisciplinary, incorporating perspectives in Homeobox and Pathology.
Charles P. Emerson studies Hedgehog signaling pathway, a branch of Cell biology.
His Muscular dystrophy research is multidisciplinary, relying on both Phenotype, Senescence, Telomerase, Telomere and Regulation of gene expression.
Between 2011 and 2019, his most popular works were:
- Facioscapulohumeral muscular dystrophy family studies of DUX4 expression: evidence for disease modifiers and a quantitative model of pathogenesis (122 citations)
- CRISPR/Cas9-mediated genome editing induces exon skipping by alternative splicing or exon deletion (116 citations)
- Telomere position effect regulates DUX4 in human facioscapulohumeral muscular dystrophy. (75 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Genetics, Facioscapulohumeral muscular dystrophy, DUX4, Regulation of gene expression and Telomere.
His Genetics study combines topics in areas such as Biceps and Skeletal muscle.
His Facioscapulohumeral muscular dystrophy research includes themes of Deltoid muscle, Gene expression, Pathogenesis, Bioinformatics and Muscle tissue.
The various areas that Charles P. Emerson examines in his DUX4 study include Immunohistochemistry, Myopathy, Biopsy, Pathology and Anatomy.
His study in Regulation of gene expression is interdisciplinary in nature, drawing from both Phenotype, Senescence, Cell biology, Muscular dystrophy and Telomerase.
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