What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Immunology, Haematopoiesis, Cell biology, Molecular biology and Virology.
His Immunology research is multidisciplinary, incorporating elements of Cell and Transplantation.
The Haematopoiesis study combines topics in areas such as Myeloid, Macrophage, Cellular differentiation and Monocyte.
His Cell biology study incorporates themes from Chemokine, Fibronectin and CXCL9.
His Molecular biology research incorporates elements of T cell, Gene product, Gene expression and Binding site.
The study incorporates disciplines such as In vitro and Scid mice in addition to Virology.
His most cited work include:
- Targeted disruption of the PU.1 gene results in multiple hematopoietic abnormalities. (974 citations)
- Transduction of Human CD34+ Cells That Mediate Long-Term Engraftment of NOD/SCID Mice by HIV Vectors (627 citations)
- Infection by porcine endogenous retrovirus after islet xenotransplantation in SCID mice. (356 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Virology, Immunology, Molecular biology, Cell biology and Haematopoiesis.
His study in Virology is interdisciplinary in nature, drawing from both Protease, HIV Protease Inhibitor and Mutant.
Bruce E. Torbett interconnects CD34, Cell and Transplantation in the investigation of issues within Immunology.
The concepts of his Molecular biology study are interwoven with issues in Cell culture, T cell, Intrabody, Antibody and Gene delivery.
Bruce E. Torbett combines subjects such as Macrophage and Viral vector with his study of Cell biology.
Bruce E. Torbett has researched Haematopoiesis in several fields, including Myeloid, Genetic enhancement, Interleukin 3 and Progenitor cell.
He most often published in these fields:
- Virology (25.29%)
- Immunology (25.86%)
- Molecular biology (26.44%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (21.84%)
- Virology (25.29%)
- Haematopoiesis (20.11%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Cell biology, Virology, Haematopoiesis, Viral vector and Progenitor cell.
His Cell biology research includes elements of Blood–brain barrier and Gene delivery.
His work carried out in the field of Virology brings together such families of science as Gene and Zinc finger.
His multidisciplinary approach integrates Haematopoiesis and Induced pluripotent stem cell in his work.
His Progenitor cell research includes themes of Immunology, Immune system, Total body surface area, Pathology and CD34.
His Immunology research incorporates themes from Neutrophil differentiation and Cell.
Between 2013 and 2021, his most popular works were:
- Mystery solved: VSV-G-LVs do not allow efficient gene transfer into unstimulated T cells, B cells, and HSCs because they lack the LDL receptor (96 citations)
- CCR5 Disruption in Induced Pluripotent Stem Cells Using CRISPR/Cas9 Provides Selective Resistance of Immune Cells to CCR5-tropic HIV-1 Virus (77 citations)
- Rapamycin relieves lentiviral vector transduction resistance in human and mouse hematopoietic stem cells (67 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Virology, Cell biology, Viral vector, Haematopoiesis and Transduction.
His Virology research is multidisciplinary, relying on both RNA, Transfer RNA, Long terminal repeat and Nucleic acid.
His Cell biology study combines topics in areas such as Sendai virus and Genetic enhancement, Gene delivery.
His research in Genetic enhancement intersects with topics in TOR Serine-Threonine Kinases, Progenitor cell, Stem cell, Immunology and Transplantation.
Haematopoiesis and Induced pluripotent stem cell are two areas of study in which Bruce E. Torbett engages in interdisciplinary research.
His Transduction study integrates concerns from other disciplines, such as Vesicular stomatitis virus, Wiskott–Aldrich syndrome and Lentivirus.
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