His primary areas of investigation include Immunology, Cell biology, Molecular biology, IL-2 receptor and Cytotoxic T cell. His Immunology research includes elements of Genetically modified mouse and Transgene. His Cell biology research includes themes of Regulatory T cell, Receptor and MHC class II.
Andrew J. Caton has included themes like Hemagglutinin, B cell, Antibody, Immunoglobulin heavy chain and Polymerase chain reaction in his Molecular biology study. His studies in Cytotoxic T cell integrate themes in fields like T cell and CD8. His T-cell receptor course of study focuses on Antigen and Virology.
His scientific interests lie mostly in Immunology, Cell biology, T-cell receptor, Molecular biology and Antigen. As a part of the same scientific study, he usually deals with the Immunology, concentrating on Cytotoxic T cell and frequently concerns with CD8. His studies deal with areas such as Central tolerance and Antigen presentation as well as Cell biology.
The study incorporates disciplines such as Receptor, Thymocyte, Major histocompatibility complex and FOXP3 in addition to T-cell receptor. His work deals with themes such as Transgene, Immunoglobulin heavy chain, Hemagglutinin, Gene and Antibody, which intersect with Molecular biology. The concepts of his Antigen study are interwoven with issues in Adoptive cell transfer and Virology.
His primary areas of study are Immunology, Cell biology, T-cell receptor, Immune system and FOXP3. As part of his studies on Immunology, he often connects relevant subjects like Receptor. His research investigates the connection between Cell biology and topics such as Antigen presentation that intersect with problems in Cellular differentiation.
Andrew J. Caton interconnects Cytotoxic T cell and Thymocyte in the investigation of issues within T-cell receptor. His Immune system research is multidisciplinary, incorporating perspectives in Influenza A virus, Antigen and Virology. His FOXP3 research incorporates themes from Regulatory T cell, IL-2 receptor and Major histocompatibility complex.
Andrew J. Caton focuses on Immunology, T-cell receptor, Immune system, FOXP3 and Virus. His work carried out in the field of Immunology brings together such families of science as Receptor and Virology. His Influenza A virus study in the realm of Virology connects with subjects such as Respiratory tract infections.
His work deals with themes such as Cell and Cell biology, which intersect with T-cell receptor. His work on Effector as part of his general Cell biology study is frequently connected to Repertoire, thereby bridging the divide between different branches of science. His Virus study combines topics in areas such as CD8, Antigen, Adoptive cell transfer and B cell.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Thymic selection of CD4+CD25+ regulatory T cells induced by an agonist self-peptide.
Martha S. Jordan;Alina Boesteanu;Amy J. Reed;Andria L. Petrone.
Nature Immunology (2001)
The antigenic structure of the influenza virus A/PR/8/34 hemagglutinin (H1 subtype).
Andrew J. Caton;George G. Brownlee;Jonathan W. Yewdell;Walter Gerhard.
Cell (1982)
Major Histocompatibility Complex Class II–Positive Cortical Epithelium Mediates the Selection of Cd4+25+ Immunoregulatory T Cells
Steven J. Bensinger;Antonio Bandeira;Martha S. Jordan;Andrew J. Caton.
Journal of Experimental Medicine (2001)
Preferential utilization of specific immunoglobulin heavy chain diversity and joining segments in adult human peripheral blood B lymphocytes.
Masao Yamada;Robert Wasserman;Betty Anne Reichard;Sara Shane.
Journal of Experimental Medicine (1991)
A role for non-MHC genetic polymorphism in susceptibility to spontaneous autoimmunity
Bernadette Scott;Roland Liblau;Sylvia Degermann;Lori Anne Marconi.
Immunity (1994)
Detection of minimal disease in hematopoietic malignancies of the B-cell lineage by using third-complementarity-determining region (CDR-III)-specific probes
Masao Yamada;Sheila Hudson;Olivia Tournay;Susan Bittenbender.
Proceedings of the National Academy of Sciences of the United States of America (1989)
Cutting edge: the natural ligand for glucocorticoid-induced TNF receptor-related protein abrogates regulatory T cell suppression.
Hong Bin Ji;Gongxian Liao;William A. Faubion;Ana C. Abadía-Molina.
Journal of Immunology (2004)
The Impact of T Helper and T Regulatory Cells on the Regulation of Anti-Double-Stranded DNA B Cells
Su-jean Seo;Michele L Fields;Jodi L Buckler;Amy J Reed.
Immunity (2002)
Cutting edge: Self-peptides drive the peripheral expansion of CD4+CD25+ regulatory T cells
Cristina Cozzo;Joseph Larkin;Andrew J. Caton.
Journal of Immunology (2003)
The antigenicity and evolution of influenza H1 haemagglutinin, from 1950-1957 and 1977-1983: two pathways from one gene.
F.L. Raymond;A.J. Caton;N.J. Cox;A.P. Kendal.
Virology (1986)
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