World's Best Scientists 2026 revealed!

D-Index & Metrics

Immunology

D-Index
50
Citations
10419
World Ranking
4224
National Ranking
65

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Enzyme

His primary areas of investigation include Immunology, Trypanosoma brucei, Tumor necrosis factor alpha, Cell biology and Cytokine. His Immunology study combines topics in areas such as Cancer research, In vitro and Gene. His studies in Trypanosoma brucei integrate themes in fields like Spleen and Trypanosoma.

His research in Tumor necrosis factor alpha tackles topics such as Interleukin 10 which are related to areas like CCL2 and Liver cell. His Cell biology research includes themes of Cellular differentiation and Monocyte. His Cytokine study integrates concerns from other disciplines, such as Parasite Infections, Antibody and Macrophage-activating factor.

His most cited work include:

  • Identification of discrete tumor-induced myeloid-derived suppressor cell subpopulations with distinct T cell–suppressive activity (947 citations)
  • Differential expression of FIZZ1 and Ym1 in alternatively versus classically activated macrophages (365 citations)
  • Bone marrow-derived monocytes give rise to self-renewing and fully differentiated Kupffer cells (334 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Immunology, Immune system, Tumor necrosis factor alpha, Cell biology and Trypanosoma brucei. His study looks at the relationship between Immunology and fields such as Disease, as well as how they intersect with chemical problems. His Immune system study combines topics from a wide range of disciplines, such as Macrophage, Kupffer cell and Virology.

His biological study spans a wide range of topics, including Tumor microenvironment, Secretion, Innate immune system and Adenylate kinase. His Trypanosoma brucei research incorporates themes from Trypanosoma, Enzyme activator, Microbiology and Arginase. His T cell research is multidisciplinary, relying on both Cellular differentiation and Myeloid-derived Suppressor Cell.

He most often published in these fields:

  • Immunology (60.87%)
  • Immune system (40.87%)
  • Tumor necrosis factor alpha (31.30%)

What were the highlights of his more recent work (between 2014-2021)?

  • Immune system (40.87%)
  • Immunology (60.87%)
  • Inflammation (24.35%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Immune system, Immunology, Inflammation, Kupffer cell and Cell biology. His study looks at the intersection of Immune system and topics like Trypanosoma brucei with Arginase, Microbiology, Parasite load and Cytokine. His study in the field of Interleukin 10 also crosses realms of Erythrophagocytosis.

His Inflammation research includes elements of Concanavalin A, Secretion, Tumor microenvironment, Tissue homeostasis and Tumor progression. The study incorporates disciplines such as Tumor necrosis factor alpha, Interleukin, Cancer research and Endothelial stem cell in addition to Kupffer cell. His research integrates issues of Inflammasome, Transcription factor and Macrophage in his study of Cell biology.

Between 2014 and 2021, his most popular works were:

  • Bone marrow-derived monocytes give rise to self-renewing and fully differentiated Kupffer cells (334 citations)
  • Stellate Cells, Hepatocytes, and Endothelial Cells Imprint the Kupffer Cell Identity on Monocytes Colonizing the Liver Macrophage Niche (106 citations)
  • The Transcription Factor ZEB2 Is Required to Maintain the Tissue-Specific Identities of Macrophages (77 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Enzyme

Alain Beschin mainly focuses on Cell biology, Kupffer cell, Macrophage, Immunology and Inflammation. His work on Cell adhesion molecule as part of his general Cell biology study is frequently connected to Perisinusoidal space, thereby bridging the divide between different branches of science. His research in Kupffer cell intersects with topics in Lineage, Homeostasis, Colony-stimulating factor, Transcription factor and Immune system.

His Macrophage research is multidisciplinary, incorporating elements of Epithelial–mesenchymal transition, Cellular differentiation, Bone marrow and Progenitor cell. His Immunology study frequently intersects with other fields, such as Myelopoiesis. The various areas that Alain Beschin examines in his Inflammation study include Embryonic stem cell and Liver cell.

Best Publications

  • Identification of discrete tumor-induced myeloid-derived suppressor cell subpopulations with distinct T cell–suppressive activity

    Kiavash Movahedi;Martin Guilliams;Jan Van den Bossche;Rafael Van den Bergh

  • Bone marrow-derived monocytes give rise to self-renewing and fully differentiated Kupffer cells

    Charlotte L Scott;Fang Zheng;Fang Zheng;Patrick De Baetselier;Liesbet Martens

  • Differential expression of FIZZ1 and Ym1 in alternatively versus classically activated macrophages

    Geert Raes;Patrick De Baetselier;Wim Noël;Alain Beschin

  • Stellate Cells, Hepatocytes, and Endothelial Cells Imprint the Kupffer Cell Identity on Monocytes Colonizing the Liver Macrophage Niche

    Johnny Bonnardel;Wouter T’Jonck;Djoere Gaublomme;Robin Browaeys

  • Classical and alternative activation of mononuclear phagocytes: picking the best of both worlds for tumor promotion.

    Jo A. Van Ginderachter;Kiavash Movahedi;Gholamreza Hassanzadeh Ghassabeh;Sofie Meerschaut

  • Alternatively activated macrophages during parasite infections.

    Wim Noël;Geert Raes;Gholamreza Hassanzadeh Ghassabeh;Patrick De Baetselier

  • The transcription factor ZEB2 is required to maintain the tissue-specific identities of macrophages

    Charlotte L. Scott;Charlotte L. Scott;Wouter T’Jonck;Liesbet Martens;Helena Todorov

  • Tumor necrosis factor alpha is a key mediator in the regulation of experimental Trypanosoma brucei infections

    Stefan Magez;Magdalena Radwanska;Alain Beschin;Kenji Sekikawa

  • Identification of a common gene signature for type II cytokine-associated myeloid cells elicited in vivo in different pathologic conditions.

    Gholamreza Hassanzadeh Ghassabeh;Patrick De Baetselier;Patrick De Baetselier;Lea Brys;Lea Brys;Wim Noël;Wim Noël

  • Reactive Oxygen Species and 12/15-Lipoxygenase Contribute to the Antiproliferative Capacity of Alternatively Activated Myeloid Cells Elicited during Helminth Infection

    Lea Brys;Alain Beschin;Geert Raes;Gholamreza Hassanzadeh Ghassabeh

  • FIZZ1 and Ym as Tools to Discriminate between Differentially Activated Macrophages

    Geert Raes;Wim Noël;Alain Beschin;Lea Brys

  • Specific Uptake of Tumor Necrosis Factor-α Is Involved in Growth Control of Trypanosoma brucei

    Stefan Magez;Stefan Magez;Maurice Geuskens;Alain Beschin;Herwig del Favero

  • Tip-DC development during parasitic infection is regulated by IL-10 and requires CCL2/CCR2, IFN-γ and MyD88 signaling

    Tom Bosschaerts;Martin Guilliams;Benoît Stijlemans;Yannick Morias

  • Adenylate Cyclases of Trypanosoma brucei Inhibit the Innate Immune Response of the Host

    Didier Salmon;Didier Salmon;Gilles Vanwalleghem;Yannick Morias;Yannick Morias;Julie N.O. Denoeud

  • Myeloid-derived suppressor cells in parasitic infections.

    Jo A. Van Ginderachter;Alain Beschin;Patrick De Baetselier;Geert Raes

  • Relative Contribution of Interferon-γ and Interleukin-10 to Resistance to Murine African Trypanosomosis

    Boniface Namangala;Wim Noël;Patrick De Baetselier;Lea Brys

  • Alternative versus classical macrophage activation during experimental African trypanosomosis

    P.De Baetselier;B Namangala;W Noël;L Brys

  • IL-10 dampens TNF/inducible nitric oxide synthase-producing dendritic cell-mediated pathogenicity during parasitic infection.

    Martin Guilliams;Kiavash Movahedi;Tom Bosschaerts;Thierry VandenDriessche;Thierry VandenDriessche

  • On the existence of cytokines in invertebrates.

    A Beschin;M Bilej;E Torreele;P De Baetselier

  • Alternatively Activated Myeloid Cells Limit Pathogenicity Associated with African Trypanosomiasis through the IL-10 Inducible Gene Selenoprotein P

    Tom Bosschaerts;Martin Guilliams;Wim Noel;Michel Hérin

  • Identification of discrete tumor-induced myeloid-derived suppressor cell subpopulations with distinct T-CELL suppressive activity

    Kiavash Movahedi;Martin Guiliams;Jan Van den Bossche;Rafael Van den Bergh

Frequent Co-Authors

Patrick De Baetselier
Patrick De Baetselier Vrije Universiteit Brussel
Geert Raes
Geert Raes Vrije Universiteit Brussel
Jo A. Van Ginderachter
Jo A. Van Ginderachter Vrije Universiteit Brussel
Martin Guilliams
Martin Guilliams Ghent University
Stefan Magez
Stefan Magez Ghent University
Etienne Pays
Etienne Pays Université Libre de Bruxelles
Rudolf Lucas
Rudolf Lucas Augusta University
Frank Brombacher
Frank Brombacher University of Cape Town
Frank Tacke
Frank Tacke Charité - University Medicine Berlin
Yvan Saeys
Yvan Saeys Ghent University

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Related Online Degrees & Career Pathways

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