His primary scientific interests are in Biochemistry, Endocrinology, Internal medicine, Retinol and Retinoid. His Biochemistry study combines topics from a wide range of disciplines, such as Molecular biology, Transthyretin and Hepatic stellate cell. The concepts of his Hepatic stellate cell study are interwoven with issues in Lipid droplet and Hepatocyte.
His research in the fields of Albumin, Creatinine and Risk factor overlaps with other disciplines such as Lean body mass. His work carried out in the field of Retinol brings together such families of science as Acyltransferase, Retinoic acid and Metabolism. In general Retinoid, his work in Retinoid binding protein is often linked to Retinol binding protein 4 linking many areas of study.
Internal medicine, Endocrinology, Biochemistry, Retinoid and Retinol are his primary areas of study. His work in Endocrinology covers topics such as Retinoic acid which are related to areas like Signal transduction. His Biochemistry research is multidisciplinary, incorporating elements of Molecular biology and Cell culture.
His Retinoid research is multidisciplinary, relying on both Liver disease, Cancer research, Homeostasis and Hepatic stellate cell. William S. Blaner has researched Hepatic stellate cell in several fields, including Fibrosis, Lipid droplet, Cell biology and Hepatocyte. His study in Retinol is interdisciplinary in nature, drawing from both Acyltransferase and Radioimmunoassay.
The scientist’s investigation covers issues in Internal medicine, Endocrinology, Retinoid, Hepatic stellate cell and Biochemistry. Internal medicine is frequently linked to Diacylglycerol kinase in his study. The concepts of his Endocrinology study are interwoven with issues in CD36 and Retinoic acid.
The study incorporates disciplines such as Diabetes mellitus, Vitamin, Signal transduction and Vitamin A deficiency in addition to Retinoic acid. His Retinoid research is multidisciplinary, incorporating perspectives in Oxidative stress, Liver disease, Retinyl acetate, Toxicity and Cytochrome P450. His Hepatic stellate cell study combines topics in areas such as Cancer research, Hepatocyte, Molecular biology, Fibrosis and Lipid droplet.
His primary areas of study are Internal medicine, Endocrinology, Retinoic acid, Adipose tissue and Biochemistry. William S. Blaner combines topics linked to Diacylglycerol kinase with his work on Endocrinology. His studies in Retinoic acid integrate themes in fields like Vitamin and Adult stem cell.
His Adipose tissue research incorporates themes from Stem cell and Bioinformatics. His study in the field of Plasma protein binding, Transmembrane protein and Calmodulin is also linked to topics like Membrane transport protein. William S. Blaner combines subjects such as Hepatic stellate cell and Retinol with his study of Retinoid.
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Human hepatic stellate cell lines, LX-1 and LX-2: new tools for analysis of hepatic fibrosis
L Xu;A Y Hui;E Albanis;M J Arthur.
Impaired retinal function and vitamin A availability in mice lacking retinol-binding protein
Loredana Quadro;William S. Blaner;Daniel J. Salchow;Silke Vogel.
The EMBO Journal (1999)
Vitamin A Metabolism: An Update
Diana N. D’Ambrosio;Robin D. Clugston;William S. Blaner.
Glycosylphosphatidylinositol-Anchored High-Density Lipoprotein-Binding Protein 1 Plays a Critical Role in the Lipolytic Processing of Chylomicrons
Anne P. Beigneux;Brandon S.J. Davies;Peter Gin;Michael M. Weinstein.
Cell Metabolism (2007)
Retinoids and retinoid-binding protein expression in rat adipocytes.
C. Tsutsumi;M. Okuno;L. Tannous;R. Piantedosi.
Journal of Biological Chemistry (1992)
Retinol-Binding Protein: The Serum Transport Protein for Vitamin A
William S. Blaner.
Endocrine Reviews (1989)
An immortalized rat liver stellate cell line (HSC-T6): a new cell model for the study of retinoid metabolism in vitro.
Silke Vogel;Roseann Piantedosi;Jorge Frank;Avraham Lalazar.
Journal of Lipid Research (2000)
Hepatic stellate cell lipid droplets: A specialized lipid droplet for retinoid storage
William S. Blaner;Sheila M. O'Byrne;Nuttaporn Wongsiriroj;Johannes Kluwe.
Biochimica et Biophysica Acta (2009)
Hepatic overexpression of hormone-sensitive lipase and adipose triglyceride lipase promotes fatty acid oxidation, stimulates direct release of free fatty acids, and ameliorates steatosis.
Brendan N. Reid;Gene P. Ables;Oleg A. Otlivanchik;Gabriele Schoiswohl.
Journal of Biological Chemistry (2008)
Hand-grip muscle strength, lean body mass, and plasma proteins as markers of nutritional status in patients with chronic renal failure close to start of dialysis therapy
Olof Heimbürger;Abdul Rashid Qureshi;Abdul Rashid Qureshi;William S. Blaner;William S. Blaner;Lars Berglund;Lars Berglund.
American Journal of Kidney Diseases (2000)
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