D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 80 Citations 72,002 185 World Ranking 986 National Ranking 31

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

Tsuneyasu Kaisho mainly investigates Cell biology, Signal transduction, Toll-like receptor, Immunology and Receptor. His study in Cell biology is interdisciplinary in nature, drawing from both Acquired immune system, TLR3, Cytokine and Myeloid Differentiation Factor 88. His Signal transduction research integrates issues from Toll-like receptor signaling pathway, Gene expression and UNC93B1.

His research integrates issues of TLR9 and Dendritic cell in his study of Toll-like receptor. His research investigates the connection between Receptor and topics such as Molecular biology that intersect with issues in Cytoplasm and B cell. His study in the field of TLR2 also crosses realms of DEAD Box Protein 58.

His most cited work include:

  • A Toll-like receptor recognizes bacterial DNA. (5617 citations)
  • Toll-like receptors. (5355 citations)
  • Toll-like receptors: critical proteins linking innate and acquired immunity. (4049 citations)

What are the main themes of his work throughout his whole career to date?

Tsuneyasu Kaisho focuses on Cell biology, Immunology, Molecular biology, Immune system and Receptor. His biological study spans a wide range of topics, including Dendritic cell, Innate immune system, Toll-like receptor and Cytokine. Tsuneyasu Kaisho has included themes like Interferon and Plasmacytoid dendritic cell in his Toll-like receptor study.

His studies deal with areas such as Transcription factor, Gene and B cell as well as Molecular biology. Tsuneyasu Kaisho interconnects Virus and Effector in the investigation of issues within Immune system. His Receptor research incorporates elements of Toll and Nucleic acid.

He most often published in these fields:

  • Cell biology (53.33%)
  • Immunology (41.33%)
  • Molecular biology (21.33%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (53.33%)
  • Immunology (41.33%)
  • T cell (14.67%)

In recent papers he was focusing on the following fields of study:

Tsuneyasu Kaisho mostly deals with Cell biology, Immunology, T cell, Inflammation and Interferon. Specifically, his work in Cell biology is concerned with the study of Protein kinase A. His Immunology study incorporates themes from Missense mutation, CD80 and Islet.

His T cell research is multidisciplinary, incorporating elements of Cancer research and Priming, CD8, Antigen. His research in Inflammation intersects with topics in Psoriasis, Toll-like receptor, Diabetes mellitus, Imiquimod and Severity of illness. His work deals with themes such as Influenza A virus, Antibiotics, Viral replication and Interferon regulatory factors, which intersect with Interferon.

Between 2017 and 2021, his most popular works were:

  • A defined commensal consortium elicits CD8 T cells and anti-cancer immunity (288 citations)
  • Topical application of aminoglycoside antibiotics enhances host resistance to viral infections in a microbiota-independent manner. (43 citations)
  • Heme ameliorates dextran sodium sulfate-induced colitis through providing intestinal macrophages with noninflammatory profiles. (19 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Cytokine

His primary areas of study are Cell biology, Immunology, Immune system, Inflammation and In vivo. Tsuneyasu Kaisho integrates Cell biology and RANKL in his research. His work in T cell and Effector is related to Immunology.

His Immune system research incorporates themes from Virus, Virus Physiological Phenomena and Macrophage. Tsuneyasu Kaisho combines subjects such as Myeloid, Receptor, Colitis and Heme with his study of Inflammation. Tsuneyasu Kaisho focuses mostly in the field of In vivo, narrowing it down to matters related to Downregulation and upregulation and, in some cases, Autoimmunity, CD80, Antibiotics, Virology and Influenza A virus.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Toll-like receptors.

Kiyoshi Takeda;Tsuneyasu Kaisho;Shizuo Akira.
Annual Review of Immunology (2003)

12145 Citations

Toll-like receptors.

Kiyoshi Takeda;Tsuneyasu Kaisho;Shizuo Akira.
Annual Review of Immunology (2003)

8659 Citations

A Toll-like receptor recognizes bacterial DNA.

Hiroaki Hemmi;Osamu Takeuchi;Taro Kawai;Tsuneyasu Kaisho.
Nature (2000)

7984 Citations

Erratum A Toll-like receptor recognizes bacterial DNA

Hiroaki Hemmi;Osamu Takeuchi;Taro Kawai;Tsuneyasu Kaisho.
Nature (2001)

6782 Citations

Toll-like receptors: critical proteins linking innate and acquired immunity.

Shizuo Akira;Kiyoshi Takeda;Tsuneyasu Kaisho.
Nature Immunology (2001)

6076 Citations

Innate antiviral responses by means of TLR7-mediated recognition of single-stranded RNA.

Sandra S. Diebold;Tsuneyasu Kaisho;Hiroaki Hemmi;Shizuo Akira.
Science (2004)

4189 Citations

Role of adaptor TRIF in the MyD88-independent toll-like receptor signaling pathway.

Masahiro Yamamoto;Shintaro Sato;Hiroaki Hemmi;Katsuaki Hoshino.
Science (2003)

3811 Citations

Small anti-viral compounds activate immune cells via the TLR7 MyD88-dependent signaling pathway.

Hiroaki Hemmi;Tsuneyasu Kaisho;Osamu Takeuchi;Shintaro Sato.
Nature Immunology (2002)

3102 Citations

Enhanced Th1 activity and development of chronic enterocolitis in mice devoid of Stat3 in macrophages and neutrophils.

Kiyoshi Takeda;Björn E Clausen;Tsuneyasu Kaisho;Tohru Tsujimura.
Immunity (1999)

1468 Citations

TRAM is specifically involved in the Toll-like receptor 4-mediated MyD88-independent signaling pathway

Masahiro Yamamoto;Shintaro Sato;Hiroaki Hemmi;Satoshi Uematsu.
Nature Immunology (2003)

1370 Citations

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