Cell biology, Immunology, Signal transduction, Molecular biology and X-linked lymphoproliferative disease are his primary areas of study. His study of SH3 domain is a part of Cell biology. His study in Immunology is interdisciplinary in nature, drawing from both Chronic mucocutaneous candidiasis and Mucocutaneous Candidiasis.
Sylvain Latour combines subjects such as Interleukin 12, CD47, Signal-regulatory protein alpha and IL-2 receptor with his study of Signal transduction. His Molecular biology research is multidisciplinary, incorporating elements of Tyrosine, Immunoreceptor tyrosine-based inhibitory motif, Receptor, Phosphorylation and Immunoglobulin E. His research in X-linked lymphoproliferative disease tackles topics such as Lymphoproliferative disorders which are related to areas like Cellular differentiation, Immune system, Pediatrics, Immune dysregulation and Survival rate.
Sylvain Latour focuses on Immunology, Cell biology, Immune system, Immunodeficiency and Molecular biology. His work in the fields of Immunology, such as Natural killer T cell, Virus and Lymphoproliferative disorders, intersects with other areas such as XIAP. His Cell biology research is multidisciplinary, incorporating perspectives in Biochemistry and T-cell receptor.
His studies deal with areas such as NKG2D, X-linked lymphoproliferative disease and Lymphocyte as well as Immune system. His work carried out in the field of Immunodeficiency brings together such families of science as Cancer research, Primary immunodeficiency, Severe combined immunodeficiency and Immune dysregulation. His Molecular biology study combines topics in areas such as Cell culture, Transfection, Receptor, Immunoglobulin E and Gene product.
His primary areas of study are Immune system, Cancer research, Immunology, Epstein–Barr virus and T cell. Specifically, his work in Immune system is concerned with the study of Immunodeficiency. Sylvain Latour focuses mostly in the field of Immunodeficiency, narrowing it down to matters related to Cytokine and, in some cases, Cell biology, Mechanistic target of rapamycin, Phosphorylation and Autoimmune disease.
His study on Cancer research also encompasses disciplines like
Sylvain Latour mostly deals with Epstein–Barr virus, Immune system, Lymphoproliferative disorders, Immunology and T cell. His Epstein–Barr virus study improves the overall literature in Virus. His biological study deals with issues like CD137, which deal with fields such as Primary immunodeficiency, T cell mediated immunity and Calcium flux.
His T cell research incorporates elements of Cancer research, Immunodeficiency, Mechanistic target of rapamycin, Phosphorylation and Lymphocyte. The Cancer research study combines topics in areas such as Mutation and Kinase, MAPK/ERK pathway, Kinase activity. The study incorporates disciplines such as Cytokine, Germline and Cell biology in addition to Immunodeficiency.
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XIAP deficiency in humans causes an X-linked lymphoproliferative syndrome
Stéphanie Rigaud;Marie-Claude Fondanèche;Nathalie Lambert;Nathalie Lambert;Benoit Pasquier.
Efficacy of Gene Therapy for X-Linked Severe Combined Immunodeficiency
Salima Hacein-Bey-Abina;Julia Hauer;Annick Lim;Capucine Picard.
The New England Journal of Medicine (2010)
Stepwise development of MAIT cells in mouse and human.
Emmanuel Martin;Emmanuel Treiner;Emmanuel Treiner;Livine Duban;Lucia Guerri.
PLOS Biology (2009)
The same tyrosine-based inhibition motif, in the intra-cytoplasmic domain of FcγRIIB, regulates negatively BCR-, TCR-, and FcR-dependent cell activation
Marc Daëron;Sylvain Latour;Odile Malbec;Eric Espinosa.
The Syk Protein Tyrosine Kinase Is Essential for Fcγ Receptor Signaling in Macrophages and Neutrophils
Friedemann Kiefer;John Brumell;Nadia Al-Alawi;Sylvain Latour.
Molecular and Cellular Biology (1998)
Regulation of high-affinity IgE receptor-mediated mast cell activation by murine low-affinity IgG receptors.
M Daëron;O Malbec;S Latour;M Arock.
Journal of Clinical Investigation (1995)
Defective NKT cell development in mice and humans lacking the adapter SAP, the X-linked lymphoproliferative syndrome gene product
Benoit Pasquier;Luo Yin;Marie-Claude Fondanèche;Francis Relouzat.
Journal of Experimental Medicine (2005)
Munc18-2 deficiency causes familial hemophagocytic lymphohistiocytosis type 5 and impairs cytotoxic granule exocytosis in patient NK cells.
Marjorie Côte;Mickaël M. Ménager;Agathe Burgess;Nizar Mahlaoui.
Journal of Clinical Investigation (2009)
Impairment of immunity to Candida and Mycobacterium in humans with bi-allelic RORC mutations
Satoshi Okada;Satoshi Okada;Janet G Markle;Elissa K Deenick;Elissa K Deenick;Federico Mele.
Clinical similarities and differences of patients with X-linked lymphoproliferative syndrome type 1 (XLP-1/SAP-deficiency) versus type 2 (XLP-2/XIAP-deficiency)
Jana Pachlopnik Schmid;Jana Pachlopnik Schmid;Danielle Canioni;Despina Moshous;Despina Moshous;Fabien Touzot.
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