Stephen M. Lanier spends much of his time researching G protein, Heterotrimeric G protein, Cell biology, Signal transduction and Biochemistry. Stephen M. Lanier focuses mostly in the field of Heterotrimeric G protein, narrowing it down to matters related to G protein-coupled receptor and, in some cases, GTPase-activating protein, G alpha subunit and Signal transducing adaptor protein. Stephen M. Lanier interconnects Cell cycle and Cell division in the investigation of issues within Cell biology.
His biological study spans a wide range of topics, including Protein subunit, Activator and Saccharomyces cerevisiae. His research in Activator intersects with topics in Guanosine and Downregulation and upregulation. Biochemistry is represented through his Binding site and Receptor research.
His primary scientific interests are in G protein, Cell biology, Signal transduction, Biochemistry and Heterotrimeric G protein. The concepts of his G protein study are interwoven with issues in Tetratricopeptide, Endocrinology, G protein-coupled receptor and Activator. His G protein-coupled receptor research includes elements of GTPase-activating protein, Signal transducing adaptor protein and G protein-coupled inwardly-rectifying potassium channel.
He combines subjects such as Apoptosis and Cell division with his study of Cell biology. His Signal transduction research focuses on Receptor and how it connects with Phosphorylation and Cell signaling. His Heterotrimeric G protein research incorporates elements of Gs alpha subunit and Gq alpha subunit.
His primary areas of study are Cell biology, G protein, Signal transduction, Activator and Biochemistry. In his study, which falls under the umbrella issue of Cell biology, Cell growth is strongly linked to Receptor. In the subject of general G protein, his work in Heterotrimeric G protein is often linked to Nonsynonymous substitution, thereby combining diverse domains of study.
His Heterotrimeric G protein research includes themes of Cystic kidney, Gs alpha subunit and G alpha subunit. His Signal transduction study integrates concerns from other disciplines, such as Molecular biology and Transcription factor. Within one scientific family, Stephen M. Lanier focuses on topics pertaining to Immune system under Activator, and may sometimes address concerns connected to Null mice and Protein kinase B.
His main research concerns Heterotrimeric G protein, G protein, Cell biology, Signal transduction and Biochemistry. The Heterotrimeric G protein study combines topics in areas such as Cell, Cystic kidney and Intracellular. His primary area of study in Cell biology is in the field of RGS4.
Stephen M. Lanier has researched Signal transduction in several fields, including Cystic kidney disease, Kidney, Polycystic kidney disease and Receptor, Activator. His study on Transmembrane protein and Pertussis toxin is often connected to Cell cortex as part of broader study in Receptor. The study incorporates disciplines such as G protein-coupled receptor, GTPase-activating protein, GTP-Binding Protein alpha Subunits and Adenylyl cyclase, Gs alpha subunit in addition to G12/G13 alpha subunits.
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RECEPTOR-INDEPENDENT ACTIVATORS OF HETEROTRIMERIC G-PROTEIN SIGNALING PATHWAYS
Aya Takesono;Mary J. Cismowski;Catalina Ribas;Michael Bernard.
Journal of Biological Chemistry (1999)
Asymmetrically Distributed C. elegans Homologs of AGS3/PINS Control Spindle Position in the Early Embryo
Monica Gotta;Yan Dong;Yuri K. Peterson;Stephen M. Lanier.
Current Biology (2003)
Genetic screens in yeast to identify mammalian nonreceptor modulators of G-protein signaling.
Mary J. Cismowski;Aya Takesono;Chienling Ma;Jeffrey S. Lizano.
Nature Biotechnology (1999)
Activator of G Protein Signaling 3: A Gatekeeper of Cocaine Sensitization and Drug Seeking
M.Scott Bowers;Krista McFarland;Russell W Lake;Yuri K Peterson.
ACCESSORY PROTEINS FOR G PROTEINS: Partners in Signaling
Motohiko Sato;Joe B. Blumer;Violaine Simon;Stephen M. Lanier.
Annual Review of Pharmacology and Toxicology (2006)
Activation of heterotrimeric G-protein signaling by a ras-related protein. Implications for signal integration.
Mary J. Cismowski;Chienling Ma;Catalina Ribas;Xiaobing Xie.
Journal of Biological Chemistry (2000)
Interaction of Arrestins with Intracellular Domains of Muscarinic and α2-Adrenergic Receptors
Guangyu Wu;Jason G. Krupnick;Jeffrey L. Benovic;Stephen M. Lanier.
Journal of Biological Chemistry (1997)
The elusive family of imidazoline binding sites
Angelo Parini;Charilaos Gargalidis Moudanos;Nathalie Pizzinat;Stephen M. Lanier.
Trends in Pharmacological Sciences (1996)
Selective Interaction of AGS3 with G-proteins and the Influence of AGS3 on the Activation State of G-proteins
Michael L. Bernard;Yuri K. Peterson;Peter Chung;Jane Jourdan.
Journal of Biological Chemistry (2001)
Expression analysis and subcellular distribution of the two G-protein regulators AGS3 and LGN indicate distinct functionality. Localization of LGN to the midbody during cytokinesis.
Joe B. Blumer;L. Judson Chandler;Stephen M. Lanier.
Journal of Biological Chemistry (2002)
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