1997 - Fellow of American Physical Society (APS) Citation For his contributions to the understanding and application of nonlinear optical materials and switchable volumetric holograms
Robert L. Sutherland mainly investigates Cancer research, Internal medicine, Cyclin D1, Endocrinology and Cancer. His Cancer research research is multidisciplinary, incorporating perspectives in Carcinogenesis, Cyclin A, Cell cycle, PTEN and Cyclin D. His work carried out in the field of Internal medicine brings together such families of science as Gene expression and Oncology.
His Cyclin E study in the realm of Cyclin D1 connects with subjects such as Cortactin. His Endocrinology research includes elements of Cell culture, Receptor and Cell growth. Robert L. Sutherland combines subjects such as Microarray and Pathology with his study of Cancer.
Robert L. Sutherland mainly focuses on Cancer research, Internal medicine, Endocrinology, Cell cycle and Cancer. His research on Cancer research also deals with topics like
His Endocrinology study combines topics from a wide range of disciplines, such as Cell culture, Epidermal growth factor, Receptor, Cell growth and Cancer cell. His work deals with themes such as Molecular biology and MCF-7, which intersect with Cell cycle. The Cancer study which covers Pathology that intersects with Prostate and Pancreatic cancer.
His primary areas of study are Breast cancer, Cancer research, Cancer, Internal medicine and Pathology. His biological study spans a wide range of topics, including Immunohistochemistry, Hedgehog and Metastasis. His Cancer research study incorporates themes from Cancer cell, Immunology, Signal transduction, Cell cycle and PTEN.
His Cancer research incorporates themes from Epigenetics and Kinase. His Internal medicine research incorporates elements of Endocrinology and Oncology. His research investigates the link between Pathology and topics such as Prostatectomy that cross with problems in Univariate analysis.
His scientific interests lie mostly in Cancer research, Breast cancer, Cancer, Estrogen receptor and Internal medicine. His Cancer research research includes themes of Immunology, Gene signature, Phosphorylation, Regulation of gene expression and Kinase. His research in the fields of Breast disease overlaps with other disciplines such as CXCR4.
His Cancer research is multidisciplinary, relying on both Receptor tyrosine kinase, Signal transduction and Tyrosine phosphorylation. His study in Estrogen receptor is interdisciplinary in nature, drawing from both Wnt signaling pathway, Trastuzumab, Tamoxifen, Stem cell and Hedgehog. Robert L. Sutherland has included themes like Phenotype and Oncology in his Internal medicine study.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Progestin Regulation of Cellular Proliferation
Christine L. Clarke;Robert L. Sutherland.
Endocrine Reviews (1990)
Estrogen-induced activation of Cdk4 and Cdk2 during G1-S phase progression is accompanied by increased cyclin D1 expression and decreased cyclin-dependent kinase inhibitor association with cyclin E-Cdk2.
Owen W. J. Prall;Boris Sarcevic;Elizabeth A. Musgrove;Colin K. W. Watts.
Journal of Biological Chemistry (1997)
Cyclin D1 induction in breast cancer cells shortens G1 and is sufficient for cells arrested in G1 to complete the cell cycle
Elizabeth A. Musgrove;Christine S. L. Lee;Michael F. Buckley;Robert L. Sutherland.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Large-scale delineation of secreted protein biomarkers overexpressed in cancer tissue and serum
John B. Welsh;Lisa M. Sapinoso;Suzanne G. Kern;David A. Brown.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Prolactin controls mammary gland development via direct and indirect mechanisms
Cathrin Brisken;Sarabjeet Kaur;Tony E. Chavarria;Nadine Binart.
Developmental Biology (1999)
Growth factor, steroid, and steroid antagonist regulation of cyclin gene expression associated with changes in T-47D human breast cancer cell cycle progression.
E. A. Musgrove;J. A. Hamilton;C. S. L. Lee;K. J. E. Sweeney.
Molecular and Cellular Biology (1993)
Frequent Loss of Estrogen Receptor-β Expression in Prostate Cancer
Lisa G. Horvath;Susan M. Henshall;C-Soon Lee;Darren R. Head.
Cancer Research (2001)
Cell Proliferation Kinetics of MCF-7 Human Mammary Carcinoma Cells in Culture and Effects of Tamoxifen on Exponentially Growing and Plateau-Phase Cells
Robert L. Sutherland;Rosemary E. Hall;Ian W. Taylor.
Cancer Research (1983)
Androgen receptor inhibits estrogen receptor-alpha activity and is prognostic in breast cancer.
Amelia A. Peters;Grant Buchanan;Carmela Ricciardelli;Tina Bianco-Miotto.
Cancer Research (2009)
Prediction of Local Recurrence, Distant Metastases, and Death After Breast-Conserving Therapy in Early-Stage Invasive Breast Cancer Using a Five-Biomarker Panel
Ewan K. A Millar;Peter H Graham;Sandra A O'Toole;Catriona M McNeil.
Journal of Clinical Oncology (2009)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Glasgow
Royal Prince Alfred Hospital
University of Glasgow
Monash University
University of Southern California
University of New South Wales
University of Adelaide
Nottingham City Hospital
Children's Medical Research Institute
Queen Mary University of London
MIT
Google (United States)
Linnaeus University
Rowan University
University of Ulm
Iowa State University
RIKEN
Ulsan National Institute of Science and Technology
Sorbonne University
Hanyang University
National Institute of Genetics
Newcastle University
Purdue University West Lafayette
Harvard University
Scripps Institution of Oceanography
University of Maryland, College Park