Genetics, Huntington's disease, Internal medicine, Disease and Framingham Heart Study are his primary areas of study. His studies in Genome-wide association study, Locus, Genetic linkage, Allele and Gene are all subfields of Genetics research. His study in Huntington's disease is interdisciplinary in nature, drawing from both Central nervous system disease, Degenerative disease, Age of onset and Trinucleotide repeat expansion.
His Trinucleotide repeat expansion research is multidisciplinary, incorporating elements of Huntingtin, Huntingtin Protein and Linkage disequilibrium. His Internal medicine study incorporates themes from Endocrinology and Genotype. His research in Disease intersects with topics in Psychiatry and Genetic testing.
The scientist’s investigation covers issues in Genetics, Internal medicine, Disease, Huntington's disease and Genetic linkage. His work is connected to Single-nucleotide polymorphism, Locus, Quantitative trait locus, Allele and Gene, as a part of Genetics. He combines subjects such as Endocrinology, Genotype and Cardiology with his study of Internal medicine.
The concepts of his Disease study are interwoven with issues in Test, Psychiatry and Pediatrics. His Huntington's disease research is multidisciplinary, incorporating perspectives in Degenerative disease, Age of onset and Trinucleotide repeat expansion. His Framingham Heart Study research focuses on Demography and how it connects with Risk factor.
Richard H. Myers mainly investigates Genetics, Huntington's disease, Genome-wide association study, Disease and Gene. His work in Single-nucleotide polymorphism, Huntingtin, Locus, Trinucleotide repeat expansion and Regulation of gene expression is related to Genetics. His biological study spans a wide range of topics, including Psychiatry, Neurodegeneration, Asymptomatic, Cognitive decline and Apathy.
His work carried out in the field of Genome-wide association study brings together such families of science as Meta-analysis, Internal medicine, Pathogenesis, LRRK2 and Allele. Richard H. Myers interconnects Endocrinology and Pathology in the investigation of issues within Internal medicine. Richard H. Myers has included themes like microRNA and Prodrome in his Disease study.
Richard H. Myers focuses on Genetics, Genome-wide association study, Huntington's disease, Regulation of gene expression and Gene. Genetics is represented through his Huntingtin, Quantitative trait locus, Locus, Transmembrane transporter activity and Penetrance research. The study incorporates disciplines such as Meta-analysis, Internal medicine, Allele and 1000 Genomes Project in addition to Genome-wide association study.
His work deals with themes such as Cerebellum, Functional ability, Clinical trial, Trinucleotide repeat expansion and microRNA, which intersect with Huntington's disease. His studies link Disease with Trinucleotide repeat expansion. His Regulation of gene expression study also includes
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes
Marcy E. MacDonald;Christine M. Ambrose;Mabel P. Duyao;Richard H. Myers.
Cell (1993)
A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes. The Huntington's Disease Collaborative Research Group.
M Shah;N Datson;L Srinidhi;VP Stanton.
Cell (1993)
Effects of Age, Sex, and Ethnicity on the Association Between Apolipoprotein E Genotype and Alzheimer Disease: A Meta-analysis
Lindsay A. Farrer;L. Adrienne Cupples;Jonathan L. Haines;Bradley T Hyman.
JAMA (1997)
Association analyses of 249,796 individuals reveal 18 new loci associated with body mass index
Elizabeth K. Speliotes;Elizabeth K. Speliotes;Cristen J. Willer;Sonja I. Berndt;Keri L. Monda.
Nature Genetics (2010)
Effects of age, sex, and ethnicity on the association between apolipoprotein E genotype and Alzheimer disease. A meta-analysis. APOE and Alzheimer Disease Meta Analysis Consortium.
L A Farrer;L A Cupples;J L Haines;B Hyman.
JAMA (1997)
Neuropathological classification of Huntington's disease.
Jean-Paul Vonsattel;Richard H. Myers;Thomas J. Stevens;Robert J. Ferrante.
Journal of Neuropathology and Experimental Neurology (1985)
Large-scale meta-analysis of genome-wide association data identifies six new risk loci for Parkinson's disease
Mike A Nalls;Nathan Pankratz;Christina M. Lill;Chuong B. Do.
Nature Genetics (2014)
Effectiveness of Covid-19 Vaccines against the B.1.617.2 (Delta) Variant.
Jamielopez L. Bernal;Nick Andrews;Charlotte Gower;Eileen Gallagher.
The New England Journal of Medicine (2021)
The genetic defect causing familial Alzheimer's disease maps on chromosome 21
Peter H. St George-Hyslop;Rudolph E. Tanzi;Ronald J. Polinsky;Jonathan L. Haines.
Science (1987)
Trinucleotide repeat length instability and age of onset in Huntington's disease
M Duyao;C Ambrose;R Myers;A Novelletto.
Nature Genetics (1993)
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