2004 - German National Academy of Sciences Leopoldina - Deutsche Akademie der Naturforscher Leopoldina – Nationale Akademie der Wissenschaften Internal Medicine and Dermatology
His primary areas of study are Internal medicine, Endocrinology, Immunology, RAGE and Receptor. His research integrates issues of Endothelial stem cell and Type 2 diabetes in his study of Internal medicine. As part of his studies on Endocrinology, Peter P. Nawroth frequently links adjacent subjects like Transcription factor.
His work carried out in the field of Immunology brings together such families of science as Integrin, Ischemia and Cell biology. The study incorporates disciplines such as Cell activation and Diabetic angiopathy in addition to RAGE. His Receptor research is multidisciplinary, relying on both In vitro and Signal transduction.
Peter P. Nawroth focuses on Internal medicine, Endocrinology, Diabetes mellitus, Cell biology and Immunology. His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology and Type 2 diabetes. Many of his studies involve connections with topics such as Methylglyoxal and Endocrinology.
Peter P. Nawroth has included themes like Surgery and Bioinformatics in his Diabetes mellitus study. Peter P. Nawroth studied Cell biology and Endothelial stem cell that intersect with Tumor necrosis factor alpha and Thrombomodulin. His Immunology research integrates issues from RAGE and Tissue factor.
His scientific interests lie mostly in Internal medicine, Diabetes mellitus, Endocrinology, Methylglyoxal and Type 2 diabetes. His study in Internal medicine is interdisciplinary in nature, drawing from both Gastroenterology, Nephropathy and Type 2 Diabetes Mellitus. His studies in Diabetes mellitus integrate themes in fields like Neuropathic pain, Surgery and Bioinformatics.
His Endocrinology research incorporates elements of Reactive oxygen species and Zebrafish. His Methylglyoxal study integrates concerns from other disciplines, such as Glycation, Metabolism and Detoxification. His study in Glycation focuses on RAGE in particular.
His main research concerns Internal medicine, Endocrinology, Methylglyoxal, Diabetes mellitus and Type 2 diabetes. His Internal medicine study which covers Gastroenterology that intersects with Comorbidity. His studies deal with areas such as Signal transduction and Tauroursodeoxycholic acid as well as Endocrinology.
His Methylglyoxal research includes themes of Glycation and Detoxification. His research on Glycation focuses in particular on RAGE. His work deals with themes such as Receptor, Angiotensin II and Renin–angiotensin system, which intersect with Diabetes mellitus.
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RAGE and amyloid-β peptide neurotoxicity in Alzheimer's disease
Shi Du Yan;Xi Chen;Jin Fu;Ming Chen.
Nature (1996)
RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides.
Marion A Hofmann;Steven Drury;Caifeng Fu;Wu Qu.
Cell (1999)
Modulation of endothelial cell hemostatic properties by tumor necrosis factor.
Peter P. Nawroth;David M. Stern.
Journal of Experimental Medicine (1986)
Understanding RAGE, the receptor for advanced glycation end products
Angelika Bierhaus;Per M. Humpert;Michael Morcos;Thoralf Wendt.
Journal of Molecular Medicine (2005)
RAGE mediates amyloid-beta peptide transport across the blood-brain barrier and accumulation in brain.
Rashid Deane;Shi Du Yan;Ram Kumar Submamaryan;Barbara LaRue.
Nature Medicine (2003)
The role of oxidative stress in the onset and progression of diabetes and its complications: a summary of a Congress Series sponsored by UNESCO-MCBN, the American Diabetes Association and the German Diabetes Society.
P Rosen;P P Nawroth;G King;W Moller.
Diabetes-metabolism Research and Reviews (2001)
Diabetes-Associated Sustained Activation of the Transcription Factor Nuclear Factor-κB
Angelika Bierhaus;Stephan Schiekofer;Stephan Schiekofer;Markus Schwaninger;Martin Andrassy;Martin Andrassy.
Diabetes (2001)
Benfotiamine blocks three major pathways of hyperglycemic damage and prevents experimental diabetic retinopathy
Hans Peter Hammes;Xueliang Du;Diane Edelstein;Tetsuya Taguchi.
Nature Medicine (2003)
A mechanism converting psychosocial stress into mononuclear cell activation.
Angelika Bierhaus;Jutta Wolf;Martin Andrassy;Nicolas Rohleder.
Proceedings of the National Academy of Sciences of the United States of America (2003)
The endogenous oestrogen metabolite 2-methoxyoestradiol inhibits angiogenesis and suppresses tumour growth
Theodore Fotsis;Youming Zhang;Michael S. Pepper;Herman Adlercreutz.
Nature (1994)
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