David M. Stern mainly investigates Cell biology, Receptor, Glycation, RAGE and Internal medicine. David M. Stern has included themes like Endothelial stem cell, Biochemistry, Cell adhesion, Cell surface receptor and Endothelium in his Cell biology study. The study incorporates disciplines such as Inflammation, Proinflammatory cytokine, Immunology and Downregulation and upregulation in addition to Receptor.
His Glycation study integrates concerns from other disciplines, such as Amyloid beta, Intracellular signal transduction, Regulation of gene expression, Neuroscience and Amyloidosis. His RAGE research includes themes of Signal transduction, Kinase and p38 mitogen-activated protein kinases. His Internal medicine research is multidisciplinary, incorporating elements of Diabetes mellitus and Endocrinology.
His primary areas of study are Internal medicine, Cell biology, Endocrinology, Receptor and Immunology. As a part of the same scientific study, he usually deals with the Internal medicine, concentrating on Diabetes mellitus and frequently concerns with Bioinformatics. The concepts of his Cell biology study are interwoven with issues in Endothelial stem cell, Cell, Endothelium and Downregulation and upregulation.
His research in Endocrinology intersects with topics in Proinflammatory cytokine and Pathogenesis. His research on Receptor concerns the broader Biochemistry. His work carried out in the field of Immunology brings together such families of science as Cancer research and Ischemia.
Internal medicine, Endocrinology, Biochemistry, Cell biology and Glycation are his primary areas of study. His research integrates issues of Diabetes mellitus and Cardiology in his study of Internal medicine. His study in Endocrinology is interdisciplinary in nature, drawing from both Angiotensin II, Bone marrow and Phosphorylation.
The various areas that David M. Stern examines in his Cell biology study include Endothelial stem cell, Adult stem cell, Genetically modified mouse and Amyloid precursor protein. His Glycation study necessitates a more in-depth grasp of Receptor. His biological study spans a wide range of topics, including Inflammation, Cancer research and Kinase.
Cell biology, Immunology, Neuroscience, Mitochondrion and RAGE are his primary areas of study. His Cell biology research is multidisciplinary, incorporating perspectives in Cell surface receptor, Protein A and Amyloid precursor protein. His Neuroscience research incorporates themes from Receptor and Cerebral organoid.
Endocrinology and Internal medicine are the main areas of his RAGE studies. His Internal medicine study incorporates themes from Unfolded protein response and Pathology. David M. Stern is interested in Glycation, which is a field of Biochemistry.
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Endothelial Cells in Physiology and in the Pathophysiology of Vascular Disorders
Douglas B. Cines;Eleanor S. Pollak;Clayton A. Buck;Joseph Loscalzo.
RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides.
Marion A Hofmann;Steven Drury;Caifeng Fu;Wu Qu.
Enhanced cellular oxidant stress by the interaction of advanced glycation end products with their receptors/binding proteins.
Shi Du Yan;A. M. Schmidt;G. M. Anderson;Jinghua Zhang.
Journal of Biological Chemistry (1994)
Modulation of endothelial cell hemostatic properties by tumor necrosis factor.
Peter P. Nawroth;David M. Stern.
Journal of Experimental Medicine (1986)
The Receptor for Advanced Glycation End Products (RAGE) Is a Cellular Binding Site for Amphoterin MEDIATION OF NEURITE OUTGROWTH AND CO-EXPRESSION OF RAGE AND AMPHOTERIN IN THE DEVELOPING NERVOUS SYSTEM
Osamu Hori;Jerold Brett;Timothy Slattery;Rong Cao.
Journal of Biological Chemistry (1995)
The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses
Ann Marie Schmidt;Shirley ShiDu Yan;Shi Fang Yan;David M. Stern.
Journal of Clinical Investigation (2001)
Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases
Taguchi A;Blood Dc;del Toro G;Canet A.
RAGE mediates amyloid-beta peptide transport across the blood-brain barrier and accumulation in brain.
Rashid Deane;Shi Du Yan;Ram Kumar Submamaryan;Barbara LaRue.
Nature Medicine (2003)
Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts.
Lisa Park;Kathleen G. Raman;Kenneth J. Lee;Yan Lu.
Nature Medicine (1998)
Activation of NADPH oxidase by AGE links oxidant stress to altered gene expression via RAGE.
Marie-Paule Wautier;Olivier Chappey;Stefano Corda;David M. Stern.
American Journal of Physiology-endocrinology and Metabolism (2001)
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