Her primary areas of investigation include Internal medicine, Endocrinology, Diabetic nephropathy, Glycation and Diabetes mellitus. Her work in Endocrinology addresses issues such as Reactive oxygen species, which are connected to fields such as Superoxide. Her Diabetic nephropathy research incorporates elements of Ramipril, Nephropathy and Protein kinase C.
Her study looks at the relationship between Glycation and fields such as Fibronectin, as well as how they intersect with chemical problems. Her Diabetes mellitus research includes elements of Nitrotyrosine, Reverse cholesterol transport, Kidney disease and Apolipoprotein B. Josephine M. Forbes works mostly in the field of Oxidative stress, limiting it down to topics relating to Mitochondrion and, in certain cases, Signal transduction, as a part of the same area of interest.
Josephine M. Forbes mainly investigates Internal medicine, Endocrinology, Glycation, Diabetes mellitus and Diabetic nephropathy. Her Internal medicine research is multidisciplinary, incorporating perspectives in Nephropathy and Type 2 diabetes. Kidney, Oxidative stress, Ramipril, Albuminuria and Kidney disease are among the areas of Endocrinology where she concentrates her study.
Her work carried out in the field of Glycation brings together such families of science as Type 1 diabetes and Immunology. Her studies examine the connections between Diabetes mellitus and genetics, as well as such issues in Disease, with regards to Diabetic kidney. Her Diabetic nephropathy research is multidisciplinary, relying on both Renin–angiotensin system, Protein kinase C, CTGF and Streptozotocin.
Her primary areas of study are Internal medicine, Diabetes mellitus, Endocrinology, Glycation and Disease. Josephine M. Forbes combines subjects such as Gastroenterology and Type 2 diabetes with her study of Internal medicine. The study incorporates disciplines such as Kidney, Pharmacology, Kidney disease and Clinical trial in addition to Diabetes mellitus.
Her work blends Endocrinology and Hip fracture studies together. Her Glycation study combines topics in areas such as Proinflammatory cytokine, Type 1 diabetes and Gene isoform. Her Disease research is multidisciplinary, incorporating elements of Cancer research, Diabetic kidney, Bioinformatics, RAGE and Insulin induced hypoglycemia.
Josephine M. Forbes mainly focuses on Disease, Internal medicine, Mitochondrion, Diabetes mellitus and Endocrinology. Her Disease study integrates concerns from other disciplines, such as Glycated hemoglobin-A1c, Bioinformatics, Hematology and RAGE, Glycation. Her research on Internal medicine frequently connects to adjacent areas such as Streptozotocin.
Her work deals with themes such as Glomerulosclerosis, Diabetic nephropathy and Pharmacology, which intersect with Mitochondrion. Her Diabetes mellitus research incorporates themes from Inflammation, Interquartile range, Kidney and Kidney disease. Her Endocrinology study frequently links to related topics such as Mitochondrial permeability transition pore.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mechanisms of Diabetic Complications
Josephine M. Forbes;Mark E. Cooper.
Physiological Reviews (2013)
Oxidative Stress as a Major Culprit in Kidney Disease in Diabetes
Josephine M Forbes;Melinda T Coughlan;Mark Emmanuel Cooper.
Diabetes (2008)
A Breaker of Advanced Glycation End Products Attenuates Diabetes-Induced Myocardial Structural Changes
Riccardo Candido;Josephine M. Forbes;Merlin C. Thomas;Vicki Thallas.
Circulation Research (2003)
Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)
Matthew D Oldfield;Leon A Bach;Josephine M Forbes;David J Nikolic-Paterson.
Journal of Clinical Investigation (2001)
Characterization of Renal Angiotensin-Converting Enzyme 2 in Diabetic Nephropathy
Chris Tikellis;Colin Ivor Johnston;Colin Ivor Johnston;Josephine Maree Forbes;Josephine Maree Forbes;Wendy C Burns;Wendy C Burns;Wendy C Burns.
Hypertension (2003)
Receptor for Advanced Glycation End Products (RAGE) Deficiency Attenuates the Development of Atherosclerosis in Diabetes
Aino Soro-Paavonen;Anna M.D. Watson;Jiaze Li;Karri Paavonen.
Diabetes (2008)
AGE, RAGE, and ROS in Diabetic Nephropathy
Adeline L.Y. Tan;Josephine M. Forbes;Mark E. Cooper.
Seminars in Nephrology (2007)
Methylglyoxal modification of Na v 1.8 facilitates nociceptive neuron firing and causes hyperalgesia in diabetic neuropathy
Angelika Bierhaus;Thomas Fleming;Stoyan Stoyanov;Andreas Leffler;Andreas Leffler.
Nature Medicine (2012)
RAGE-Induced Cytosolic ROS Promote Mitochondrial Superoxide Generation in Diabetes
Melinda T. Coughlan;David R. Thorburn;Sally A. Penfold;Sally A. Penfold;Adrienne Laskowski.
Journal of The American Society of Nephrology (2009)
Role of Advanced Glycation End Products in Diabetic Nephropathy
Josephine M. Forbes;Mark E. Cooper;Matthew D. Oldfield;Merlin C. Thomas.
Journal of The American Society of Nephrology (2003)
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