2003 - Fellow of the American Association for the Advancement of Science (AAAS)
Michael A. Caligiuri mainly focuses on Immunology, Natural killer cell, Interleukin 21, Leukemia and Interleukin 12. His Immunology study frequently draws connections to adjacent fields such as Cancer research. His research in Natural killer cell intersects with topics in Lymphopoiesis and Receptor.
He has included themes like Cancer, Myeloid leukemia, Oncology and NPM1 in his Leukemia study. The Myeloid leukemia study which covers BAALC that intersects with Fms-Like Tyrosine Kinase 3. His research integrates issues of Monokine and Cell biology in his study of Interleukin 12.
His primary areas of study are Immunology, Cancer research, Leukemia, Myeloid leukemia and Internal medicine. His study in Immunology is interdisciplinary in nature, drawing from both Natural killer cell and Interleukin 12. Michael A. Caligiuri combines subjects such as Innate immune system and Interleukin 15 with his study of Natural killer cell.
His Cancer research study integrates concerns from other disciplines, such as Haematopoiesis, Stem cell, T cell, Chronic myelogenous leukemia and microRNA. His Leukemia research incorporates themes from Cancer, Fms-Like Tyrosine Kinase 3, Myeloid, BAALC and NPM1. The study incorporates disciplines such as Genetics, Gene, MLL Partial Tandem Duplication, Molecular biology and Bone marrow in addition to Myeloid leukemia.
His main research concerns Cancer research, Immune system, Cell biology, Immunology and Cell. He has researched Cancer research in several fields, including T cell, Leukemia and Cutaneous T-cell lymphoma. His Immune system research is multidisciplinary, relying on both Cytotoxic T cell and Antigen.
As a member of one scientific family, he mostly works in the field of Cell biology, focusing on Granzyme B and, on occasion, Innate immune system. His Immunology study frequently links to related topics such as Natural killer cell. His Myeloid leukemia study incorporates themes from Myeloid, microRNA and Tandem exon duplication.
Cancer research, Antigen, Cell biology, Leukemia and Cell are his primary areas of study. The various areas that Michael A. Caligiuri examines in his Cancer research study include Cell culture, CD8, Cell killing and Interleukin 15. His Antigen research includes themes of Antibody, Immune system and Virology.
The concepts of his Cell biology study are interwoven with issues in Granzyme B, Transcription factor, Innate lymphoid cell and Functional profiling. His work deals with themes such as Myeloid and Myeloid leukemia, which intersect with Leukemia. His Natural killer cell research focuses on Immunology and how it relates to Cell growth.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Molecular classification of cancer: class discovery and class prediction by gene expression monitoring.
T. R. Golub;T. R. Golub;D. K. Slonim;P. Tamayo;C. Huard.
The biology of human natural killer-cell subsets.
Megan A Cooper;Todd A Fehniger;Michael A Caligiuri.
Trends in Immunology (2001)
Innate or Adaptive Immunity? The Example of Natural Killer Cells
Eric Vivier;David H. Raulet;Alessandro Moretta;Michael A. Caligiuri.
Human natural killer cells
Michael A. Caligiuri.
Aberrant CpG-island methylation has non-random and tumour-type-specific patterns.
Joseph F. Costello;Joseph F. Costello;Michael C. Frühwald;Michael C. Frühwald;Dominic J. Smiraglia;Laura J. Rush.
Nature Genetics (2000)
Human natural killer cells: a unique innate immunoregulatory role for the CD56bright subset
Megan A. Cooper;Todd A. Fehniger;Sarah C. Turner;Kenneth S. Chen.
MicroRNAs bind to Toll-like receptors to induce prometastatic inflammatory response
Muller Fabbri;Alessio Paone;Federica Calore;Roberta Galli.
Proceedings of the National Academy of Sciences of the United States of America (2012)
Interleukin 15: biology and relevance to human disease
Todd A. Fehniger;Michael A. Caligiuri.
CD56bright natural killer cells are present in human lymph nodes and are activated by T cell–derived IL-2: a potential new link between adaptive and innate immunity
Todd A Fehniger;Megan A Cooper;Gerard J Nuovo;Marina Cella.
Absence of the Wild-Type Allele Predicts Poor Prognosis in Adult de Novo Acute Myeloid Leukemia with Normal Cytogenetics and the Internal Tandem Duplication of FLT3 A Cancer and Leukemia Group B Study
Susan P. Whitman;Kellie J. Archer;Lan Feng;Claudia Baldus.
Cancer Research (2001)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: