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Immunology

D-Index
94
Citations
42494
World Ranking
919
National Ranking
83

Medicine

D-Index
95
Citations
42725
World Ranking
9920
National Ranking
961

Research.com Recognitions

  • 2017 - Fellow of the Royal Society of Edinburgh
  • 2012 - Member of Academia Europaea
  • 2011 - Fellow of the Royal Society, United Kingdom
  • Fellow of The Academy of Medical Sciences, United Kingdom
  • Member of the Association of American Physicians
  • Fellow of The Academy of Medical Sciences, United Kingdom
  • Member of the Association of American Physicians
  • Fellow of The Academy of Medical Sciences, United Kingdom
  • Member of the Association of American Physicians
  • Fellow of The Academy of Medical Sciences, United Kingdom
  • Member of the Association of American Physicians

Overview

Mark Walport is affiliated with Imperial College London in the United Kingdom. Their research spans multiple fields, including Medicine, Environmental Science, and Psychology, with specific subfields in Oncology, Health, Toxicology and Mutagenesis, and Clinical Psychology.

Their work focuses on topics related to healthcare and environmental impacts, with significant contributions in areas such as COVID-19 and healthcare impacts, COVID-19 and mental health, and climate change and health impacts.

Mark Walport has published research in the following venues:

  • Philosophical Transactions of the Royal Society A Mathematical Physical and Engineering Sciences

One of their recent papers is titled "Executive Summary to the Royal Society report 'COVID-19: examining the effectiveness of non-pharmaceutical interventions'," published in 2023 in the Philosophical Transactions of the Royal Society A Mathematical Physical and Engineering Sciences.

There are no frequent co-authors listed for Mark Walport.

Mark Walport has been recognized with several awards and memberships:

  • Fellow of the Royal Society of Edinburgh (2017)
  • Member of Academia Europaea (2012)
  • Fellow of the Royal Society, United Kingdom (2011)
  • Member of the Association of American Physicians
  • Fellow of The Academy of Medical Sciences, United Kingdom

Best Publications

  • Complement. First of two parts.

    Mark J. Walport

  • Grand challenges in global mental health

    Pamela Y. Collins;Vikram Patel;Vikram Patel;Sarah S. Joestl;Dana March;Dana March

  • Complement. Second of two parts.

    Mark J. Walport

  • Macrophage phagocytosis of aging neutrophils in inflammation. Programmed cell death in the neutrophil leads to its recognition by macrophages.

    John S Savill;Andrew H. Wyllie;Janet E Henson;Mark J Walport

  • Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies

    M Botto;C Dell'Agnola;A E Bygrave;E M Thompson

  • A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo.

    Philip R. Taylor;Anna Carugati;Valerie A. Fadok;H. Terence Cook

  • Arthritis Critically Dependent on Innate Immune System Players

    Hong Ji;Koichiro Ohmura;Koichiro Ohmura;Umar Mahmood;David M Lee

  • The Role of Complement in the Development of Systemic Lupus Erythematosus

    Anthony P Manderson;Marina Botto;Mark J Walport

  • Serum amyloid P component controls chromatin degradation and prevents antinuclear autoimmunity.

    M.C.M. Bickerstaff;M. Botto;W.L. Hutchinson;J. Herbert

  • Role of Surfactant Proteins A, D, and C1q in the Clearance of Apoptotic Cells In Vivo and In Vitro: Calreticulin and CD91 as a Common Collectin Receptor Complex

    R. William Vandivier;Carol Anne Ogden;Valerie A. Fadok;Peter R. Hoffmann

  • Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor H

    Matthew C. Pickering;H. Terence Cook;Joanna Warren;Anne E. Bygrave

  • Family study of the major histocompatibility complex in patients with systemic lupus erythematosus: importance of null alleles of C4A and C4B in determining disease susceptibility.

    A H Fielder;M J Walport;J R Batchelor;R I Rynes

  • The Immune System in Health and Disease

    Charles A Janeway;Paul Travers;Mark Walport;Mark J Shlomchik

  • Systemic lupus erythematosus, complement deficiency, and apoptosis.

    M C Pickering;M Botto;P R Taylor;P J Lachmann

  • C1q and systemic lupus erythematosus.

    Mark J. Walport;Kevin A. Davies;Marina Botto

  • The classical pathway is the dominant complement pathway required for innate immunity to Streptococcus pneumoniae infection in mice

    Jeremy S. Brown;Tracy Hussell;Sarah M. Gilliland;David W. Holden

  • Complement deficiency and disease.

    B.Paul Morgan;Mark J Walport

  • Polymyositis, pulmonary fibrosis and autoantibodies to aminoacyl-tRNA synthetase enzymes.

    C. Marguerie;C. C. Bunn;H. L. C. Beynon;R. M. Bernstein

  • C1q, autoimmunity and apoptosis.

    Marina Botto;Mark J Walport

  • Complement facilitates early prion pathogenesis.

    M A Klein;P S Kaeser;P Schwarz;H Weyd

  • Disease-associated loss of erythrocyte complement receptors (CR1, C3b receptors) in patients with systemic lupus erythematosus and other diseases involving autoantibodies and/or complement activation.

    G D Ross;W J Yount;M J Walport;J B Winfield

Frequent Co-Authors

Charles A. Janeway
Charles A. Janeway Yale University
Marina Botto
Marina Botto Imperial College London
Philip R. Taylor
Philip R. Taylor Cardiff University
H. Terence Cook
H. Terence Cook Imperial College London
Timothy J. Vyse
Timothy J. Vyse King's College London
Matthew C. Pickering
Matthew C. Pickering Imperial College London
Graham R. V. Hughes
Graham R. V. Hughes London Bridge Hospital
David A. Isenberg
David A. Isenberg University College London
A. B. Kay
A. B. Kay Imperial College London
B. Paul Morgan
B. Paul Morgan Cardiff University

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