D-Index & Metrics Best Publications
Immunology
UK
2023

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 82 Citations 34,140 332 World Ranking 922 National Ranking 81
Medicine D-index 90 Citations 39,303 393 World Ranking 7520 National Ranking 725

Research.com Recognitions

Awards & Achievements

2023 - Research.com Immunology in United Kingdom Leader Award

2017 - Fellow of the Royal Society of Edinburgh

2012 - Member of Academia Europaea

2011 - Fellow of the Royal Society, United Kingdom

Member of the Association of American Physicians

Fellow of The Academy of Medical Sciences, United Kingdom

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Immune system

The scientist’s investigation covers issues in Immunology, Complement system, Lupus erythematosus, Internal medicine and Immune system. The study incorporates disciplines such as Systemic lupus erythematosus and Glomerulonephritis in addition to Immunology. His work in Complement system addresses issues such as Molecular biology, which are connected to fields such as Amyloid, Pentraxins, CD59, Hemoglobinuria and Hemolysis.

His research in Lupus erythematosus tackles topics such as Pathology which are related to areas like Leukotriene B4, Erythema, Prostaglandin E2 and Cardiology. Mark Walport combines subjects such as Gastroenterology and Endocrinology with his study of Internal medicine. His Immune system research is multidisciplinary, relying on both Receptor, Pathogenesis and In vivo.

His most cited work include:

  • Complement. First of two parts. (2353 citations)
  • Macrophage phagocytosis of aging neutrophils in inflammation. Programmed cell death in the neutrophil leads to its recognition by macrophages. (1348 citations)
  • Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies (1287 citations)

What are the main themes of his work throughout his whole career to date?

Mark Walport mainly investigates Immunology, Lupus erythematosus, Immune system, Molecular biology and Internal medicine. His Antibody, Complement system, Autoimmune disease, Autoantibody and Autoimmunity study are his primary interests in Immunology. His Lupus erythematosus research integrates issues from Systemic disease, Immunopathology, Systemic lupus erythematosus, Connective tissue disease and Anti-SSA/Ro autoantibodies.

His Immune system study incorporates themes from Receptor and In vivo. His Molecular biology study integrates concerns from other disciplines, such as Genetics, Gene and Exon. His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology and Endocrinology.

He most often published in these fields:

  • Immunology (53.85%)
  • Lupus erythematosus (17.31%)
  • Immune system (13.46%)

What were the highlights of his more recent work (between 2001-2018)?

  • Immunology (53.85%)
  • Complement system (12.02%)
  • Autoimmunity (6.73%)

In recent papers he was focusing on the following fields of study:

Mark Walport mainly focuses on Immunology, Complement system, Autoimmunity, Immune system and Glomerulonephritis. His study in Autoantibody, Nephritis, Antibody, Lupus erythematosus and Autoimmune disease falls under the purview of Immunology. Mark Walport works mostly in the field of Lupus erythematosus, limiting it down to topics relating to Systemic lupus erythematosus and, in certain cases, In vivo, as a part of the same area of interest.

Mark Walport has included themes like Apoptosis and Complement deficiency in his Autoimmunity study. His biological study deals with issues like Cell biology, which deal with fields such as Complement factor B. His Glomerulonephritis study which covers Bone marrow that intersects with Transplantation.

Between 2001 and 2018, his most popular works were:

  • Grand challenges in global mental health (1255 citations)
  • Arthritis Critically Dependent on Innate Immune System Players (625 citations)
  • Role of Surfactant Proteins A, D, and C1q in the Clearance of Apoptotic Cells In Vivo and In Vitro: Calreticulin and CD91 as a Common Collectin Receptor Complex (441 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Immune system

His primary areas of study are Immunology, Complement system, Classical complement pathway, Autoimmunity and Immune system. The various areas that Mark Walport examines in his Immunology study include Systemic lupus erythematosus, Glomerulonephritis and Haplotype. Mark Walport usually deals with Complement system and limits it to topics linked to Molecular biology and Bone marrow, Phagocytosis, In vitro, Complement factor B and Isotype.

His Classical complement pathway research is multidisciplinary, relying on both Complement and Complement receptor. Mark Walport has researched Autoimmunity in several fields, including Phenotype, Gene, C57BL/6, Lupus erythematosus and In vivo. His research investigates the connection between Immune system and topics such as Apoptosis that intersect with problems in Cell biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Complement. First of two parts.

Mark J. Walport.
The New England Journal of Medicine (2001)

4209 Citations

Grand challenges in global mental health

Pamela Y. Collins;Vikram Patel;Vikram Patel;Sarah S. Joestl;Dana March;Dana March.
(2011)

2146 Citations

Complement. Second of two parts.

Mark J. Walport.
The New England Journal of Medicine (2001)

2092 Citations

Macrophage phagocytosis of aging neutrophils in inflammation. Programmed cell death in the neutrophil leads to its recognition by macrophages.

John S Savill;Andrew H. Wyllie;Janet E Henson;Mark J Walport.
Journal of Clinical Investigation (1989)

1856 Citations

Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies

M Botto;C Dell'Agnola;A E Bygrave;E M Thompson.
Nature Genetics (1998)

1643 Citations

A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo.

Philip R. Taylor;Anna Carugati;Valerie A. Fadok;H. Terence Cook.
Journal of Experimental Medicine (2000)

850 Citations

Arthritis Critically Dependent on Innate Immune System Players

Hong Ji;Koichiro Ohmura;Koichiro Ohmura;Umar Mahmood;David M Lee.
Immunity (2002)

794 Citations

The Role of Complement in the Development of Systemic Lupus Erythematosus

Anthony P Manderson;Marina Botto;Mark J Walport.
Annual Review of Immunology (2004)

694 Citations

Serum amyloid P component controls chromatin degradation and prevents antinuclear autoimmunity.

M.C.M. Bickerstaff;M. Botto;W.L. Hutchinson;J. Herbert.
Nature Medicine (1999)

630 Citations

Family study of the major histocompatibility complex in patients with systemic lupus erythematosus: importance of null alleles of C4A and C4B in determining disease susceptibility.

A H Fielder;M J Walport;J R Batchelor;R I Rynes.
BMJ (1983)

617 Citations

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