H. Terence Cook mainly focuses on Immunology, Internal medicine, Kidney disease, Glomerulonephritis and Pathology. H. Terence Cook studied Immunology and Gene that intersect with Systemic lupus erythematosus. His Internal medicine research is multidisciplinary, relying on both Gastroenterology and Endocrinology.
His Kidney disease study combines topics from a wide range of disciplines, such as Kidney and Intensive care medicine. His Pathology research integrates issues from Myeloperoxidase and Renal pathology. His studies deal with areas such as Alternative complement pathway, Membranoproliferative glomerulonephritis and Cell biology as well as Factor H.
His primary areas of study are Immunology, Glomerulonephritis, Internal medicine, Pathology and Endocrinology. His Immunology study frequently draws connections between adjacent fields such as Glomerulopathy. Within one scientific family, H. Terence Cook focuses on topics pertaining to Nephritis under Glomerulonephritis, and may sometimes address concerns connected to Bone marrow and Nephrotoxicity.
His Internal medicine research focuses on Gastroenterology and how it connects with Methylprednisolone, Proteinuria and Creatinine. His research investigates the connection with Pathology and areas like Renal pathology which intersect with concerns in Lupus nephritis. His Kidney disease research includes themes of Disease and Intensive care medicine.
H. Terence Cook mainly focuses on Immunology, Pathology, Glomerulonephritis, Glomerulopathy and Internal medicine. His work in Immunology addresses subjects such as Glomerular basement membrane, which are connected to disciplines such as Nephritis. His work carried out in the field of Pathology brings together such families of science as Focal segmental glomerulosclerosis, Immunosuppression, Podocyte and Monoclonal.
His Glomerulonephritis research includes elements of Vasculitis, Pathogenesis and Renal pathology. His research integrates issues of Alternative complement pathway, Factor H, Kidney disease, Atypical hemolytic uremic syndrome and Disease in his study of Glomerulopathy. His Internal medicine study often links to related topics such as Endocrinology.
His scientific interests lie mostly in Pathology, Renal pathology, Immunology, Biopsy and Proteinuria. His work deals with themes such as Glomerulosclerosis and Arteriosclerosis, which intersect with Pathology. His Renal pathology research is multidisciplinary, incorporating perspectives in Glomerulonephritis, Kidney disease and Intensive care medicine.
The study incorporates disciplines such as Internal medicine, Kidney and Glomerulopathy in addition to Immunology. His research in Internal medicine intersects with topics in Endocrinology and Factor H. His Biopsy research is multidisciplinary, incorporating elements of Native kidney, Systemic lupus erythematosus, Vasculitis, Anti-neutrophil cytoplasmic antibody and Lupus nephritis.
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Pathologic Classification of Diabetic Nephropathy
Thijs W. Cohen Tervaert;Antien L. Mooyaart;Kerstin Amann;Arthur H. Cohen.
Journal of The American Society of Nephrology (2010)
The Oxford classification of IgA nephropathy: Rationale, clinicopathological correlations, and classification
Daniel C. Cattran;Rosanna Coppo;H. Terence Cook;John Feehally.
Kidney International (2009)
Kidney disease: Improving global outcomes (KDIGO) glomerulonephritis work group. KDIGO clinical practice guideline for glomerulonephritis
Daniel C. Cattran;John Feehally;H. Terence Cook;Zhi Hong Liu.
Kidney International (2012)
A hierarchical role for classical pathway complement proteins in the clearance of apoptotic cells in vivo.
Philip R. Taylor;Anna Carugati;Valerie A. Fadok;H. Terence Cook.
Journal of Experimental Medicine (2000)
Copy number polymorphism in Fcgr3 predisposes to glomerulonephritis in rats and humans
Timothy J. Aitman;Rong Dong;Timothy J. Vyse;Penny J. Norsworthy.
The Oxford classification of IgA nephropathy: pathology definitions, correlations, and reproducibility
Ian S.D. Roberts;H. Terence Cook;Stéphan Troyanov;Charles E. Alpers.
Kidney International (2009)
Nr4a1-dependent Ly6C(low) monocytes monitor endothelial cells and orchestrate their disposal.
Leo M. Carlin;Efstathios G. Stamatiades;Cedric Auffray;Richard N. Hanna.
A role for Dicer in immune regulation.
Bradley S Cobb;Arnulf Hertweck;James P Smith;Eric D. O'Connor.
Journal of Experimental Medicine (2006)
Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor H
Matthew C. Pickering;H. Terence Cook;Joanna Warren;Anne E. Bygrave.
Nature Genetics (2002)
FCGR3B copy number variation is associated with susceptibility to systemic, but not organ-specific, autoimmunity
Manuela Fanciulli;Penny J Norsworthy;Enrico Petretto;Rong Dong.
Nature Genetics (2007)
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