Fellow of The Academy of Medical Sciences, United Kingdom
Her primary areas of investigation include Immunology, Cytotoxic T cell, Hepatitis B virus, Interleukin 21 and Antigen. Her work on Immune system, Interferon gamma and Leishmaniasis as part of general Immunology research is frequently linked to Liposome, thereby connecting diverse disciplines of science. Her Cytotoxic T cell study integrates concerns from other disciplines, such as Interleukin 2, T cell, T-cell receptor and CD8.
She focuses mostly in the field of Hepatitis B virus, narrowing it down to topics relating to Hepatitis B and, in certain cases, Regulatory B cells. As a member of one scientific family, she mostly works in the field of Antigen, focusing on Viral load and, on occasion, CTLA-4, T lymphocyte, Avidity and Immune tolerance. Mala K. Maini studies Virus, a branch of Virology.
The scientist’s investigation covers issues in Immunology, Virology, Cytotoxic T cell, Immune system and CD8. Immunology is represented through her Hepatitis B virus, Virus, T cell, Hepatitis B and Chronic hepatitis research. Her studies deal with areas such as Interferon and Viral load as well as Hepatitis B virus.
Her Virology study incorporates themes from Antigen presentation and Antigen. Her research links Epitope with Cytotoxic T cell. Her CD8 research is multidisciplinary, incorporating elements of Molecular biology and Human leukocyte antigen.
Her scientific interests lie mostly in Immunology, T cell, Antigen, CD8 and Virology. Her research on Immunology often connects related topics like Prospective cohort study. Her studies in T cell integrate themes in fields like Cell, In vitro, Hepatitis b surface antigen and Cell biology.
Her work deals with themes such as Cytotoxic T cell, Molecular biology and Immunotherapy, which intersect with CD8. Mala K. Maini interconnects Pathology, Cytotoxicity, Transplantation and Hepatitis B virus in the investigation of issues within Cytotoxic T cell. Her study in the field of Virus and Anti hbs antibody is also linked to topics like Prime boost vaccination.
Her primary scientific interests are in CD8, Hepatitis B virus, Hepatitis B, Immunity and T cell. Her work on HBsAg as part of general Hepatitis B virus study is frequently linked to Liver biopsy, therefore connecting diverse disciplines of science. Mala K. Maini works mostly in the field of Hepatitis B, limiting it down to topics relating to Virus and, in certain cases, Hepatic fibrosis, Bioinformatics and Disease.
Her Immunity research is within the category of Immunology. Her research brings together the fields of Epigenetics and Immunology. Her T cell research integrates issues from Cytokine, Downregulation and upregulation, Antigen and Cytotoxic T cell, Interleukin 15.
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The role of virus-specific CD8(+) cells in liver damage and viral control during persistent hepatitis B virus infection.
Mala K. Maini;Carolina Boni;Chun Kyon Lee;Juan R. Larrubia.
Journal of Experimental Medicine (2000)
Incubation phase of acute hepatitis B in man: dynamic of cellular immune mechanisms
George J.M. Webster;Stephanie Reignat;Mala K. Maini;Simon A. Whalley.
Living in the liver: hepatic infections
Ulrike Protzer;Mala K. Maini;Percy A. Knolle.
Nature Reviews Immunology (2012)
Cytokines induced during chronic hepatitis B virus infection promote a pathway for NK cell–mediated liver damage
Claire Dunn;R. Maurizia Brunetto;Gary Reynolds;Theodoros Christophides.
Journal of Experimental Medicine (2007)
Direct ex vivo analysis of hepatitis B virus-specific CD8+ T cells associated with the control of infection
Mala K. Maini;Carolina Boni;Graham S. Ogg;Abigail S. King.
Modulation of the CD8+-T-Cell Response by CD4+ CD25+ Regulatory T Cells in Patients with Hepatitis B Virus Infection
Ornella Franzese;Patrick T. F. Kennedy;Adam J. Gehring;Jim Gotto.
Journal of Virology (2005)
IL-10–Producing Regulatory B Cells in the Pathogenesis of Chronic Hepatitis B Virus Infection
Abhishek Das;Gidon Ellis;Celeste Pallant;A. Ross Lopes.
Journal of Immunology (2012)
Up-regulation of a death receptor renders antiviral T cells susceptible to NK cell–mediated deletion
Dimitra Peppa;Upkar S. Gill;Gary Reynolds;Nicholas J.W. Easom.
Journal of Experimental Medicine (2013)
Role of the coinhibitory receptor cytotoxic T lymphocyte antigen-4 on apoptosis-Prone CD8 T cells in persistent hepatitis B virus infection.
Anna Schurich;Pooja Khanna;A. Ross Lopes;Ki Jun Han.
Escaping high viral load exhaustion: CD8 cells with altered tetramer binding in chronic hepatitis B virus infection
Stephanie Reignat;George J.M. Webster;David Brown;Graham S. Ogg.
Journal of Experimental Medicine (2002)
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