Her scientific interests lie mostly in Virology, Hepatitis B virus, Immunology, Hepatitis B and cccDNA. Her research is interdisciplinary, bridging the disciplines of Molecular biology and Virology. The study incorporates disciplines such as Viral hepatitis, Adoptive cell transfer and Antigen in addition to Hepatitis B virus.
The Immunology study which covers Cytotoxic T cell that intersects with CD8, T cell and Cell biology. The Hepatitis B study combines topics in areas such as Circular DNA, Position paper, Malaria and Global health, Public health. In her study, Cytidine deamination, Minichromosome, Transcription activator-like effector nuclease and Hepatitis is strongly linked to APOBEC3A, which falls under the umbrella field of cccDNA.
Ulrike Protzer focuses on Virology, Hepatitis B virus, Immunology, Virus and Immune system. Her research in Virology intersects with topics in Hepatocellular carcinoma and Antigen. Her Hepatocellular carcinoma research includes elements of Liver disease and Cirrhosis.
In her research on the topic of Hepatitis B virus, Viral hepatitis is strongly related with Hepatitis B. In Immunology, she works on issues like Cytotoxic T cell, which are connected to Cell biology. Her cccDNA study integrates concerns from other disciplines, such as Transcription and DNA.
Virology, Hepatitis B virus, Virus, Antigen and Immune system are her primary areas of study. Her Virology research is multidisciplinary, relying on both Epitope, Antibody and Oxygenation. Her Hepatitis B virus research is multidisciplinary, incorporating perspectives in Cancer research, Nuclear protein and Genome.
Her Virus research incorporates themes from Hepatitis B, Hepatitis C, Autoimmunity and Cell biology. Her Immune system study improves the overall literature in Immunology. Her study in Immunology is interdisciplinary in nature, drawing from both Cancer and Liver function.
Ulrike Protzer mostly deals with Virus, Severe acute respiratory syndrome coronavirus 2, Virology, Hepatitis B virus and Pandemic. Her research investigates the link between Virus and topics such as Antibody that cross with problems in Public health and Viral hepatitis. Particularly relevant to cccDNA is her body of work in Virology.
Her Hepatitis B virus research is multidisciplinary, incorporating elements of Hepatitis B, Regulation of gene expression, Interferon and Immune system. Her Immune system research incorporates elements of Hepatocyte and Liver infection. While the research belongs to areas of Internal medicine, Ulrike Protzer spends her time largely on the problem of Medical microbiology, intersecting her research to questions surrounding Antigen.
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Specific and nonhepatotoxic degradation of nuclear hepatitis B virus cccDNA.
Julie Lucifora;Yuchen Xia;Florian Reisinger;Ke Zhang.
Living in the liver: hepatic infections
Ulrike Protzer;Mala K. Maini;Percy A. Knolle.
Nature Reviews Immunology (2012)
Metabolic activation of intrahepatic CD8+ T cells and NKT cells causes nonalcoholic steatohepatitis and liver cancer via cross-talk with hepatocytes
Monika Julia Wolf;Arlind Adili;Kira Piotrowitz;Zeinab Abdullah.
Cancer Cell (2014)
Hepatitis B virus X protein is essential to initiate and maintain virus replication after infection
Julie Lucifora;Silke Arzberger;David Durantel;David Durantel;David Durantel;Laura Belloni;Laura Belloni.
Journal of Hepatology (2011)
Investigation of a COVID-19 outbreak in Germany resulting from a single travel-associated primary case: a case series.
Merle M Böhmer;Udo Buchholz;Victor M Corman;Martin Hoch.
Lancet Infectious Diseases (2020)
Not interferon, but interleukin-6 controls early gene expression in hepatitis B virus infection.
Marianna Hösel;Maria Quasdorff;Katja Wiegmann;Dennis Webb.
Multilevel proteomics reveals host perturbations by SARS-CoV-2 and SARS-CoV.
Alexey Stukalov;Virginie Girault;Vincent Grass;Ozge Karayel.
Hepatitis B virus surface antigen impairs myeloid dendritic cell function: a possible immune escape mechanism of hepatitis B virus.
Marjoleine L Op den Brouw;Rekha S Binda;Mark H van Roosmalen;Ulrike Protzer.
Interferon-γ and Tumor Necrosis Factor-α Produced by T Cells Reduce the HBV Persistence Form, cccDNA, Without Cytolysis.
Yuchen Xia;Daniela Stadler;Julie Lucifora;Florian Reisinger.
A global scientific strategy to cure hepatitis B
Peter A Revill;Francis V Chisari;Joan M Block;Maura Dandri.
The Lancet Gastroenterology & Hepatology (2019)
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