D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 57 Citations 11,089 125 World Ranking 1960 National Ranking 962

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Enzyme

His primary scientific interests are in Microbiology, Mycobacterium tuberculosis, Phagocytosis, Mannose receptor and Receptor. His Microbiology research is multidisciplinary, relying on both Complement system, Complement receptor, Macrophage and Lipoarabinomannan. He has researched Mycobacterium tuberculosis in several fields, including Immune system, Immunology, Mycobacterium and Alveolar macrophage.

His work carried out in the field of Phagocytosis brings together such families of science as Zymosan, Protein A and Virulence. His studies in Receptor integrate themes in fields like Molecular biology and Antibody opsonization. His Molecular biology study which covers Cell biology that intersects with Surfactant protein A and Innate immune system.

His most cited work include:

  • Macrophage phagocytosis of virulent but not attenuated strains of Mycobacterium tuberculosis is mediated by mannose receptors in addition to complement receptors. (530 citations)
  • Phagocytosis of Mycobacterium tuberculosis is mediated by human monocyte complement receptors and complement component C3. (452 citations)
  • The human macrophage mannose receptor directs Mycobacterium tuberculosis lipoarabinomannan-mediated phagosome biogenesis (405 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Microbiology, Mycobacterium tuberculosis, Immunology, Immune system and Tuberculosis. His work deals with themes such as Francisella tularensis, Macrophage and Lipoarabinomannan, which intersect with Microbiology. His Mycobacterium tuberculosis study integrates concerns from other disciplines, such as Pathogen, Mycobacterium, Virology, Intracellular and Alveolar macrophage.

Larry S. Schlesinger interconnects Granuloma and Disease in the investigation of issues within Immune system. His Tuberculosis study integrates concerns from other disciplines, such as Diabetes mellitus, Type 2 diabetes and Immunity. His Phagocytosis research incorporates elements of Receptor, Mannose receptor and Complement system, Complement receptor.

He most often published in these fields:

  • Microbiology (42.80%)
  • Mycobacterium tuberculosis (36.02%)
  • Immunology (30.08%)

What were the highlights of his more recent work (between 2015-2021)?

  • Mycobacterium tuberculosis (36.02%)
  • Immunology (30.08%)
  • Immune system (22.88%)

In recent papers he was focusing on the following fields of study:

Larry S. Schlesinger spends much of his time researching Mycobacterium tuberculosis, Immunology, Immune system, Tuberculosis and Microbiology. His studies in Mycobacterium tuberculosis integrate themes in fields like Autophagy, Phagosome, Phagocytosis, Surfactant protein A and Virulence. His research in Immunology intersects with topics in Latent tuberculosis, Lung and Macaque.

His Immune system study also includes

  • Sarcoidosis most often made with reference to Granuloma,
  • Cell biology that intertwine with fields like Inflammasome, Caspase 1 and Complement receptor. His study looks at the relationship between Tuberculosis and fields such as Immunity, as well as how they intersect with chemical problems. His Microbiology research includes themes of Macrophage and Intracellular.

Between 2015 and 2021, his most popular works were:

  • Human isotype-dependent inhibitory antibody responses against Mycobacterium tuberculosis. (76 citations)
  • Human Genetic Determinants of Viral Diseases. (62 citations)
  • Human Genetic Determinants of Viral Diseases. (62 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Enzyme

Larry S. Schlesinger mainly investigates Immunology, Mycobacterium tuberculosis, Immune system, Tuberculosis and Microbiology. Larry S. Schlesinger is studying Innate immune system, which is a component of Immunology. His Mycobacterium tuberculosis research integrates issues from Surfactant protein A, Pulmonary Surfactant-Associated Protein A, Pulmonary surfactant, Lipid metabolism and Macrophage.

The various areas that Larry S. Schlesinger examines in his Immune system study include Latent tuberculosis, Cytokine, Disease, Lung and Granuloma. The study incorporates disciplines such as Epidemiology, Family medicine, Alternative medicine and Public health, Population health in addition to Tuberculosis. His Pathogen study in the realm of Microbiology interacts with subjects such as Scavenger receptor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Macrophage phagocytosis of virulent but not attenuated strains of Mycobacterium tuberculosis is mediated by mannose receptors in addition to complement receptors.

L S Schlesinger.
Journal of Immunology (1993)

801 Citations

Phagocytosis of Mycobacterium tuberculosis is mediated by human monocyte complement receptors and complement component C3.

L S Schlesinger;C G Bellinger-Kawahara;N R Payne;M A Horwitz.
Journal of Immunology (1990)

670 Citations

The human macrophage mannose receptor directs Mycobacterium tuberculosis lipoarabinomannan-mediated phagosome biogenesis

Peter B. Kang;Abul K. Azad;Jordi B. Torrelles;Thomas M. Kaufman.
Journal of Experimental Medicine (2005)

581 Citations

Binding of the terminal mannosyl units of lipoarabinomannan from a virulent strain of Mycobacterium tuberculosis to human macrophages.

L S Schlesinger;S R Hull;T M Kaufman.
Journal of Immunology (1994)

442 Citations

Pulmonary surfactant protein A mediates enhanced phagocytosis of Mycobacterium tuberculosis by a direct interaction with human macrophages.

C D Gaynor;F X McCormack;D R Voelker;S E McGowan.
Journal of Immunology (1995)

373 Citations

Innate Immune Gene Polymorphisms in Tuberculosis

Abul K. Azad;Wolfgang Sadee;Larry S. Schlesinger.
Infection and Immunity (2012)

328 Citations

Surfactant Protein D Binds to Mycobacterium tuberculosis Bacilli and Lipoarabinomannan via Carbohydrate-Lectin Interactions Resulting in Reduced Phagocytosis of the Bacteria by Macrophages1

J. Scott Ferguson;Dennis R. Voelker;Francis X. McCormack;Larry S. Schlesinger.
Journal of Immunology (1999)

299 Citations

Virulent Strains of Helicobacter pylori Demonstrate Delayed Phagocytosis and Stimulate Homotypic Phagosome Fusion in Macrophages

Lee-Ann H. Allen;Larry S. Schlesinger;Byoung Kang.
Journal of Experimental Medicine (2000)

273 Citations

Mycobacterium tuberculosis lipomannan blocks TNF biosynthesis by regulating macrophage MAPK-activated protein kinase 2 (MK2) and microRNA miR-125b

Murugesan V. S. Rajaram;Bin Ni;Jessica D. Morris;Michelle N. Brooks.
Proceedings of the National Academy of Sciences of the United States of America (2011)

270 Citations

Modeling the Mycobacterium tuberculosis Granuloma - the Critical Battlefield in Host Immunity and Disease.

Evelyn Guirado;Larry S. Schlesinger.
Frontiers in Immunology (2013)

256 Citations

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