H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Microbiology D-index 43 Citations 5,579 81 World Ranking 2925 National Ranking 1180

Overview

What is he best known for?

The fields of study he is best known for:

  • Immune system
  • Cytokine
  • Apoptosis

Cell biology, Immunology, Phosphorylation, Protein kinase B and Francisella are his primary areas of study. His study ties his expertise on Biochemistry together with the subject of Cell biology. Susheela Tridandapani has researched Phosphorylation in several fields, including Receptor, Tyrosine and Signal transduction.

His research integrates issues of Macrophage, Cell cycle and Kinase in his study of Protein kinase B. Within one scientific family, he focuses on topics pertaining to CD52 under Chronic lymphocytic leukemia, and may sometimes address concerns connected to Cancer research. His study looks at the relationship between PI3K/AKT/mTOR pathway and topics such as Inflammation, which overlap with Monocyte.

His most cited work include:

  • Macrophage microvesicles induce macrophage differentiation and miR-223 transfer. (299 citations)
  • Curcumin induces G2/M arrest and apoptosis in cisplatin-resistant human ovarian cancer cells by modulating akt and p38 mAPK (211 citations)
  • Regulated Expression and Inhibitory Function of FcγRIIb in Human Monocytic Cells (170 citations)

What are the main themes of his work throughout his whole career to date?

Susheela Tridandapani mainly focuses on Cell biology, Immunology, Cancer research, Monocyte and Cytokine. His Cell biology research focuses on subjects like Receptor, which are linked to Myeloid. His research in Cancer research intersects with topics in Bruton's tyrosine kinase, Interleukin 21, Lymphokine-activated killer cell and Ibrutinib.

Susheela Tridandapani works mostly in the field of Protein kinase B, limiting it down to topics relating to PI3K/AKT/mTOR pathway and, in certain cases, MAPK/ERK pathway, Kinase and Molecular biology, as a part of the same area of interest. His Phosphorylation research incorporates elements of Tyrosine and Transfection. His Immune system research includes elements of Cancer cell and Microbiology.

He most often published in these fields:

  • Cell biology (35.97%)
  • Immunology (33.81%)
  • Cancer research (26.62%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cancer research (26.62%)
  • Immunology (33.81%)
  • Cytokine (17.99%)

In recent papers he was focusing on the following fields of study:

Susheela Tridandapani spends much of his time researching Cancer research, Immunology, Cytokine, Monocyte and Immunotherapy. Susheela Tridandapani combines subjects such as Bruton's tyrosine kinase, Leukemia, Ibrutinib and Lymphokine-activated killer cell with his study of Cancer research. In his study, Interferon, CD86 and Cancer is inextricably linked to Fc receptor, which falls within the broad field of Cytokine.

The Monocyte study combines topics in areas such as Interferon gamma and Cell biology. Susheela Tridandapani is involved in the study of Cell biology that focuses on Phagocytosis in particular. His work deals with themes such as CCL2, In vitro, Adverse effect, Innate immune system and Antibody, which intersect with Immunotherapy.

Between 2015 and 2021, his most popular works were:

  • Myeloid-derived suppressor cells express Bruton's tyrosine kinase and can be depleted in tumor bearing hosts by ibrutinib treatment (93 citations)
  • Nitric Oxide Production by Myeloid-Derived Suppressor Cells Plays a Role in Impairing Fc Receptor-Mediated Natural Killer Cell Function. (74 citations)
  • Analysis of the Effects of the Bruton's tyrosine kinase (Btk) Inhibitor Ibrutinib on Monocyte Fcγ Receptor (FcγR) Function. (35 citations)

In his most recent research, the most cited papers focused on:

  • Immune system
  • Cytokine
  • Apoptosis

Susheela Tridandapani mainly investigates Cancer research, Immunology, Cytokine, Myeloid-derived Suppressor Cell and Immunotherapy. His Cancer research research is multidisciplinary, relying on both Oncology, Natural killer cell, Interleukin 21, Lymphokine-activated killer cell and Chemokine. The various areas that Susheela Tridandapani examines in his Immunology study include Tyrosine kinase and Bruton's tyrosine kinase.

His research integrates issues of Cancer, Receptor, Fc receptor, Adoptive cell transfer and Monocyte in his study of Cytokine. The various areas that Susheela Tridandapani examines in his Immunotherapy study include Gastroenterology, Adverse effect, Alpha interferon and Vaccination. His Immune system research incorporates elements of Cancer cell and Stromal cell.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Macrophage microvesicles induce macrophage differentiation and miR-223 transfer.

Noura Ismail;Yijie Wang;Duaa Dakhlallah;Leni Moldovan.
Blood (2013)

395 Citations

Curcumin induces G2/M arrest and apoptosis in cisplatin-resistant human ovarian cancer cells by modulating akt and p38 mAPK

Nathan M. Weir;Karuppaiyah Selvendiran;Vijay Kumar Kutala;Liyue Tong.
Cancer Biology & Therapy (2007)

300 Citations

Regulated Expression and Inhibitory Function of FcγRIIb in Human Monocytic Cells

Susheela Tridandapani;Kristina Siefker;Jean-Luc Teillaud;Jo Ellen Carter.
Journal of Biological Chemistry (2002)

257 Citations

Negative signaling in B lymphocytes induces tyrosine phosphorylation of the 145-kDa inositol polyphosphate 5-phosphatase, SHIP.

G W Chacko;S Tridandapani;J E Damen;L Liu.
Journal of Immunology (1996)

198 Citations

Caspase-7 Activation by the Nlrc4/Ipaf Inflammasome Restricts Legionella pneumophila Infection

Anwari Akhter;Mikhail A. Gavrilin;Laura Frantz;Songcerae Washington.
PLOS Pathogens (2009)

195 Citations

miR-155 regulates IFN-γ production in natural killer cells.

Rossana Trotta;Li Chen;David Ciarlariello;Srirama Josyula.
Blood (2012)

186 Citations

MiR-155 induction by F. novicida but not the virulent F. tularensis results in SHIP down-regulation and enhanced pro-inflammatory cytokine response.

Thomas J. Cremer;David H Ravneberg;Corey D. Clay;Melissa G. Piper-Hunter.
PLOS ONE (2009)

184 Citations

Pulmonary Surfactant Protein A Regulates TLR Expression and Activity in Human Macrophages

Lisa N. Henning;Abul K. Azad;Kishore V. L. Parsa;Joy E. Crowther.
Journal of Immunology (2008)

177 Citations

Lipopolysaccharide-Induced Macrophage Inflammatory Response Is Regulated by SHIP

Huiqing Fang;Ruma A. Pengal;Xianhua Cao;Latha P. Ganesan.
Journal of Immunology (2004)

174 Citations

Targeting CD37-positive lymphoid malignancies with a novel engineered small modular immunopharmaceutical

Xiaobin B Zhao;Rosa Lapalombella;Trupti Joshi;Carolyn Cheney.
Blood (2007)

170 Citations

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