What is he best known for?
The fields of study he is best known for:
- Immune system
- Cytokine
- Antibody
His main research concerns Molecular biology, Antigen presentation, Dendritic cell, Antigen and Immune system.
His study in Molecular biology is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Receptor, Melanocortin, CD86 and Mitogen-activated protein kinase.
His research integrates issues of Cell biology and Antigen-presenting cell in his study of Antigen presentation.
His work is connected to Major histocompatibility complex and CD8, as a part of Antigen.
The various areas that Karsten Mahnke examines in his Immune system study include Endocrinology, Internal medicine, Cytokine, CD80 and Receptor expression.
His research ties Immunology and Cytotoxic T cell together.
His most cited work include:
- Dendritic Cells Induce Peripheral T Cell Unresponsiveness under Steady State Conditions in Vivo (1689 citations)
- Efficient Targeting of Protein Antigen to the Dendritic Cell Receptor DEC-205 in the Steady State Leads to Antigen Presentation on Major Histocompatibility Complex Class I Products and Peripheral CD8+ T Cell Tolerance (1213 citations)
- Efficient Targeting of Protein Antigen to the Dendritic Cell Receptor DEC-205 in the Steady State Leads to Antigen Presentation on Major Histocompatibility Complex Class I Products and Peripheral CD8+ T Cell Tolerance (1213 citations)
What are the main themes of his work throughout his whole career to date?
Karsten Mahnke mainly focuses on Immunology, Cell biology, Antigen, Immune system and Dendritic cell.
His Immunology study incorporates themes from Cancer research and Cell growth.
His work deals with themes such as Proinflammatory cytokine, Cytotoxic T cell, Adenosine and T cell, which intersect with Cell biology.
His Cytotoxic T cell research incorporates elements of Ovalbumin and Mononuclear phagocyte system.
His research in Antigen intersects with topics in Molecular biology and Antibody.
His Dendritic cell research integrates issues from Langerhans cell and Antigen presentation.
He most often published in these fields:
- Immunology (59.34%)
- Cell biology (46.15%)
- Antigen (37.36%)
What were the highlights of his more recent work (between 2013-2021)?
- Immunology (59.34%)
- Immune system (30.77%)
- Cell biology (46.15%)
In recent papers he was focusing on the following fields of study:
Karsten Mahnke mainly investigates Immunology, Immune system, Cell biology, Regulatory T cell and Antigen.
His Immunology research includes elements of Cell and CD31.
His biological study spans a wide range of topics, including Receptor and Melanoma.
Karsten Mahnke has researched Cell biology in several fields, including Adenosine, Dendritic cell, Sensitization, Proinflammatory cytokine and Peripheral tolerance.
His research on Antigen focuses in particular on CD8.
In his study, Cytotoxic T cell is inextricably linked to Major histocompatibility complex, which falls within the broad field of Langerhans cell.
Between 2013 and 2021, his most popular works were:
- The role of regulatory T cell (Treg) subsets in gestational diabetes mellitus (31 citations)
- Regulatory T Cell–Derived Adenosine Induces Dendritic Cell Migration through the Epac-Rap1 Pathway (30 citations)
- ATP and Its Metabolite Adenosine as Regulators of Dendritic Cell Activity. (27 citations)
In his most recent research, the most cited papers focused on:
- Immune system
- Cytokine
- Antibody
Karsten Mahnke focuses on Immune system, Cell biology, Immunology, Receptor and T cell.
Karsten Mahnke combines subjects such as Cytotoxic T cell and Melanoma with his study of Immune system.
His study brings together the fields of CD31 and Immunology.
His Receptor study integrates concerns from other disciplines, such as Peripheral tolerance, Antigen and Effector.
His T cell study combines topics from a wide range of disciplines, such as Adenosine, Extracellular, Dendritic cell, Cytokine secretion and Proinflammatory cytokine.
His research investigates the connection between Adenosine and topics such as Signal transduction that intersect with issues in Regulatory T cell.
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