His primary areas of investigation include Senescence, Cell biology, Cell aging, Signal transduction and Embryonic stem cell. The Senescence study combines topics in areas such as Cancer, Cell cycle, Cell cycle checkpoint, Senolytic and Phenotype. His Cell biology research incorporates themes from Chromatin, Cellular differentiation and In vivo.
His studies in Cell aging integrate themes in fields like MAPKAPK2, HEK 293 cells, Regulation of gene expression, PI3K/AKT/mTOR pathway and Kinase. Jesús Gil has researched Signal transduction in several fields, including Cancer research, Protein kinase A, Protein kinase R, EIF-2 kinase and Epigenetics. The study incorporates disciplines such as Glycolysis, Flux and Genetic screen in addition to Embryonic stem cell.
Jesús Gil mostly deals with Cell biology, Senescence, Cancer research, Cell aging and Phenotype. Jesús Gil has included themes like Genetics, Transcription factor and Reprogramming in his Cell biology study. His research in Senescence intersects with topics in Cancer, Cell, Senolytic, Cell cycle checkpoint and Chromatin.
His Cancer research research integrates issues from Carcinogenesis, Immunology, Kinase, Telomere and Prostate cancer. His Cell aging study incorporates themes from Regulation of gene expression and DNA damage. His work deals with themes such as Protein kinase R and Cell growth, which intersect with Signal transduction.
Jesús Gil mainly focuses on Senescence, Cell biology, Cancer, Cancer research and Phenotype. His research integrates issues of Cell, Cell cycle checkpoint, Senolytic, Apoptosis and Chromatin in his study of Senescence. His Cell biology research is multidisciplinary, incorporating perspectives in Ageing, Epigenetics, Immune system and Cellular senescence.
Jesús Gil usually deals with Cancer and limits it to topics linked to Disease and Signal transduction, Cellular Ageing and Fibrosis. His Cancer research research incorporates elements of Prostate, Prostate cancer, Chemotherapy and DNA repair. His Phenotype research incorporates themes from Inflammation, Cancer therapy, Metastasis and Radiation therapy.
His primary areas of study are Senescence, Cell biology, Senolytic, Cellular senescence and DNA damage. His Senescence research is multidisciplinary, relying on both Chromatin, Apoptosis and Regulation of gene expression. His Regulation of gene expression research incorporates elements of Enhancer, Transcription factor and Epigenetics.
His work carried out in the field of Cell biology brings together such families of science as Prodrug and Tissue homeostasis. Jesús Gil has researched Cellular senescence in several fields, including Ageing, Translational research, Bioinformatics and Disease classification. Carcinogenesis, Cancer research and Inflammation is closely connected to Prostate in his research, which is encompassed under the umbrella topic of DNA damage.
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Tumour biology: Senescence in premalignant tumours
Manuel Collado;Jesús Gil;Alejo Efeyan;Carmen Guerra.
Nature (2005)
Chemokine Signaling via the CXCR2 Receptor Reinforces Senescence
Juan C. Acosta;Ana O'Loghlen;Ana Banito;Maria V. Guijarro.
Cell (2008)
Molecular Interplay of the Noncoding RNA ANRIL and Methylated Histone H3 Lysine 27 by Polycomb CBX7 in Transcriptional Silencing of INK4a
Kyoko L. Yap;Side Li;Ana M. Muñoz-Cabello;Selina Raguz.
Molecular Cell (2010)
A complex secretory program orchestrated by the inflammasome controls paracrine senescence
Juan Carlos Acosta;Ana Banito;Torsten Wuestefeld;Athena Georgilis.
Nature Cell Biology (2013)
Senescence surveillance of pre-malignant hepatocytes limits liver cancer development
Tae-Won Kang;Tetyana Yevsa;Norman Woller;Lisa Hoenicke.
Nature (2011)
Impact of Protein Kinase PKR in Cell Biology: from Antiviral to Antiproliferative Action
M. A. García;J. Gil;I. Ventoso;S. Guerra.
Microbiology and Molecular Biology Reviews (2006)
Regulation of the INK4b-ARF-INK4a tumour suppressor locus : all for one or one for all
Jesús Gil;Gordon Peters.
Nature Reviews Molecular Cell Biology (2006)
Cellular Senescence: Defining a Path Forward
Vassilis Gorgoulis;Peter D. Adams;Andrea Alimonti;Dorothy C. Bennett.
Cell (2019)
Glycolytic Enzymes Can Modulate Cellular Life Span
Hiroshi Kondoh;Matilde E. Lleonart;Matilde E. Lleonart;Jesus Gil;Jesus Gil;Jing Wang.
Cancer Research (2005)
Senescence impairs successful reprogramming to pluripotent stem cells
Ana Banito;Sheikh T. Rashid;Juan Carlos Acosta;Si De Li.
Genes & Development (2009)
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