Member of the Association of American Physicians
Glutamate receptor and NMDA receptor are the main topics of his Receptor study. His studies link Receptor with Glutamate receptor. His study in Alzheimer's disease extends to Disease with its themes. His research on Neuroscience frequently links to adjacent areas such as Neuroprotection. J. Timothy Greenamyre performs integrative study on Mitochondrion and Gene. J. Timothy Greenamyre incorporates Gene and Disease in his research. His Toxicity research extends to Internal medicine, which is thematically connected. His Toxicity study frequently draws parallels with other fields, such as Internal medicine. His multidisciplinary approach integrates Parkinson's disease and Alpha-synuclein in his work.
His work blends Neuroscience and Pharmacology studies together. He merges Pharmacology with Neuroscience in his research. Disease is closely attributed to Neurodegeneration in his study. His Neurodegeneration study frequently draws connections to other fields, such as Pathology. Many of his studies on Pathology apply to Parkinson's disease as well. By researching both Parkinson's disease and Dopaminergic, he produces research that crosses academic boundaries. J. Timothy Greenamyre combines Dopaminergic and Substantia nigra in his research. His Substantia nigra study often links to related topics such as Disease. J. Timothy Greenamyre merges Biochemistry with Enzyme in his research.
The research on Mitochondrial DNA and Messenger RNA is part of his Gene project. J. Timothy Greenamyre regularly links together related areas like Genetics in his Messenger RNA studies. He merges many fields, such as Genetics and Mitochondrial DNA, in his writings. His Disease study frequently draws connections between related disciplines such as LRRK2. As part of his studies on LRRK2, he frequently links adjacent subjects like Pathology. J. Timothy Greenamyre performs integrative Pathology and Pathogenesis research in his work. J. Timothy Greenamyre performs multidisciplinary studies into Pathogenesis and Disease in his work. J. Timothy Greenamyre conducts interdisciplinary study in the fields of Neuroscience and Cell biology through his works. His Cell biology study frequently draws connections between related disciplines such as Rotenone.
Neuroscience is closely attributed to Astrocyte in his work. His studies link Neuroscience with Astrocyte. As part of his studies on Pathology, J. Timothy Greenamyre frequently links adjacent subjects like Amyloid (mycology). His research on Amyloid (mycology) often connects related areas such as Pathology. In his research, J. Timothy Greenamyre undertakes multidisciplinary study on Disease and Etiology. J. Timothy Greenamyre carries out multidisciplinary research, doing studies in Etiology and Pathogenesis. Much of his study explores Pathogenesis relationship to Immunology. J. Timothy Greenamyre integrates many fields in his works, including Immunology and Genetics. In his study, J. Timothy Greenamyre carries out multidisciplinary Genetics and Bioinformatics research.
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Early mitochondrial calcium defects in Huntington's disease are a direct effect of polyglutamines
Alexander V. Panov;Claire-Anne Gutekunst;Blair R. Leavitt;Michael R. Hayden.
Nature Neuroscience (2002)
Mechanism of toxicity in rotenone models of Parkinson's disease.
Todd B. Sherer;Ranjita Betarbet;Claudia M. Testa;Byoung Boo Seo.
The Journal of Neuroscience (2003)
Subcutaneous Rotenone Exposure Causes Highly Selective Dopaminergic Degeneration and α-Synuclein Aggregation
Todd B Sherer;Jin Ho Kim;Jin Ho Kim;Ranjita Betarbet;J Timothy Greenamyre.
Experimental Neurology (2003)
Animal models of Parkinson's disease
Ranjita Betarbet;Todd B. Sherer;J. Timothy Greenamyre.
A highly reproducible rotenone model of Parkinson's disease
Jason R. Cannon;Victor M. Tapias;Hye Mee Na;Anthony S. Honick.
Neurobiology of Disease (2009)
An In Vitro Model of Parkinson's Disease: Linking Mitochondrial Impairment to Altered α-Synuclein Metabolism and Oxidative Damage
Todd B. Sherer;Ranjita Betarbet;Amy K. Stout;Serena Lund.
The Journal of Neuroscience (2002)
Excitatory amino acids and Alzheimer's disease.
J.Timothy Greenamyre;Anne B. Young.
Neurobiology of Aging (1989)
Parkinson's--Divergent Causes, Convergent Mechanisms
J. Timothy Greenamyre;Teresa G. Hastings.
Increased apoptosis of Huntington disease lymphoblasts associated with repeat length-dependent mitochondrial depolarization
Akira Sawa;Gordon W. Wiegand;Jillian Cooper;Russell L. Margolis.
Nature Medicine (1999)
N-Terminal Mutant Huntingtin Associates with Mitochondria and Impairs Mitochondrial Trafficking
Adam L. Orr;Shihua Li;Chuan-En Wang;He Li.
The Journal of Neuroscience (2008)
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