His primary areas of investigation include Neuroscience, Parkinson's disease, Dopamine, Dyskinesia and MPTP. The Neuroscience study combines topics in areas such as Parkinsonism and Subthalamic nucleus. Erwan Bezard has researched Parkinson's disease in several fields, including Neurodegeneration and Neuroprotection.
His biological study spans a wide range of topics, including Degeneration and Receptor. His studies in Dyskinesia integrate themes in fields like Levodopa and Dopamine receptor, Dopamine receptor D3. His work deals with themes such as Neurotoxin and Dopamine Plasma Membrane Transport Proteins, which intersect with MPTP.
His primary areas of study are Neuroscience, Parkinson's disease, Dopamine, Dyskinesia and MPTP. His Neuroscience study combines topics in areas such as Disease and Parkinsonism. His Parkinson's disease research includes themes of Agonist, Neurodegeneration, Neuroprotection, Putamen and Degenerative disease.
As part of his research on Dopamine, studies on Endocrinology and Internal medicine are part of the effort. His research in Dyskinesia intersects with topics in Levodopa, Pharmacology and Dopamine receptor D3. His MPTP research integrates issues from Dopamine transporter, Neurotoxin, Tyrosine hydroxylase and Macaque.
His scientific interests lie mostly in Neuroscience, Parkinson's disease, Dopamine, Dopaminergic and Dyskinesia. His Neuroscience research incorporates themes from Receptor and Disease, Neurodegeneration. His work carried out in the field of Parkinson's disease brings together such families of science as Autophagy and Agonist.
Erwan Bezard works on Dopamine which deals in particular with Striatum. His Dopaminergic research is multidisciplinary, incorporating elements of Protein aggregation, Intracellular, Dystonia, Basal ganglia and Mediator. His Dyskinesia study incorporates themes from Cholinergic, Circadian rhythm, Physical medicine and rehabilitation and Pharmacology.
Erwan Bezard focuses on Neuroscience, Dopaminergic, Parkinson's disease, Neurodegeneration and Dopamine. His Neuroscience study integrates concerns from other disciplines, such as Pathological, Disease, Parkinsonism and Signal transduction. His studies deal with areas such as Serotonergic, Neurotransmitter and Mass spectrometry as well as Dopaminergic.
Many of his research projects under Parkinson's disease are closely connected to Glycine binding with Glycine binding, tying the diverse disciplines of science together. His biological study focuses on Striatum. His Striatum study which covers Dyskinesia that intersects with Pharmacology, Guanine nucleotide exchange factor and PI3K/AKT/mTOR pathway.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Initial clinical manifestations of Parkinson's disease: features and pathophysiological mechanisms
Maria C Rodriguez-Oroz;Maria C Rodriguez-Oroz;Marjan Jahanshahi;Paul Krack;Paul Krack;Irene Litvan.
Lancet Neurology (2009)
Relationship between the Appearance of Symptoms and the Level of Nigrostriatal Degeneration in a Progressive 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine-Lesioned Macaque Model of Parkinson's Disease
Erwan Bezard;Erwan Bezard;Sandra Dovero;Caroline Prunier;Paula Ravenscroft.
The Journal of Neuroscience (2001)
Pathophysiology of levodopa-induced dyskinesia: Potential for new therapies
Erwan Bezard;Jonathan M. Brotchie;Christian E. Gross.
Nature Reviews Neuroscience (2001)
Lewy body extracts from Parkinson disease brains trigger α‐synuclein pathology and neurodegeneration in mice and monkeys
Ariadna Recasens;Benjamin Dehay;Jordi Bové;Iria Carballo-Carbajal.
Annals of Neurology (2014)
Past, present, and future of Parkinson's disease: A special essay on the 200th Anniversary of the Shaking Palsy
J.A. Obeso;M. Stamelou;M. Stamelou;C.G. Goetz;W. Poewe.
Movement Disorders (2017)
Priorities in Parkinson's disease research
Wassilios G Meissner;Wassilios G Meissner;Mark Frasier;Thomas Gasser;Christopher G Goetz.
Nature Reviews Drug Discovery (2011)
A brain–spine interface alleviating gait deficits after spinal cord injury in primates
Marco Capogrosso;Tomislav Milekovic;David Borton;David Borton;Fabien Wagner.
Nature (2016)
Attenuation of levodopa-induced dyskinesia by normalizing dopamine D3 receptor function.
Erwan Bézard;Sandrine Ferry;Ulrich Mach;Holger Stark.
Nature Medicine (2003)
Increased D1 dopamine receptor signaling in levodopa-induced dyskinesia.
Incarnation Aubert;Céline Guigoni;Kerstin Håkansson;Qin Li.
Annals of Neurology (2005)
Slowing of neurodegeneration in Parkinson's disease and Huntington's disease: future therapeutic perspectives.
Anthony H V Schapira;C Warren Olanow;J Timothy Greenamyre;Erwan Bezard;Erwan Bezard.
The Lancet (2014)
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