Claude Libert mainly investigates Immunology, Cell biology, Glucocorticoid receptor, Tumor necrosis factor alpha and Inflammation. His Immunology study integrates concerns from other disciplines, such as Matrix metalloproteinase and Bioinformatics. His studies deal with areas such as Nuclear receptor, Transrepression, Transcription factor and Transactivation as well as Glucocorticoid receptor.
His biological study spans a wide range of topics, including Proinflammatory cytokine, RIPK1, Necroptosis, Apoptosis and Necrosis. His work on Systemic inflammation as part of general Inflammation study is frequently connected to Neutrophil collagenase, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. Claude Libert interconnects X chromosome and Sepsis in the investigation of issues within Immune system.
His main research concerns Immunology, Tumor necrosis factor alpha, Inflammation, Internal medicine and Glucocorticoid receptor. His Immunology research is multidisciplinary, relying on both Cancer research and Matrix metalloproteinase. Claude Libert usually deals with Tumor necrosis factor alpha and limits it to topics linked to Pharmacology and In vivo.
His Systemic inflammation study, which is part of a larger body of work in Inflammation, is frequently linked to Intestinal epithelium, bridging the gap between disciplines. His studies deal with areas such as Gastroenterology and Endocrinology as well as Internal medicine. His Glucocorticoid receptor study deals with Nuclear receptor intersecting with Cell biology.
Inflammation, Immunology, Sepsis, Glucocorticoid receptor and Tumor necrosis factor alpha are his primary areas of study. His Inflammation study incorporates themes from Cancer research, Cytokine, Pathology, Receptor and Signal transduction. His research ties Disease and Immunology together.
Claude Libert focuses mostly in the field of Sepsis, narrowing it down to matters related to Intensive care medicine and, in some cases, Clinical trial. Claude Libert combines subjects such as Nuclear receptor, Bioinformatics and Peroxisome proliferator-activated receptor with his study of Glucocorticoid receptor. Within one scientific family, Claude Libert focuses on topics pertaining to Neuroinflammation under Tumor necrosis factor alpha, and may sometimes address concerns connected to Experimental autoimmune encephalomyelitis.
Claude Libert mainly focuses on Inflammation, Immunology, Glucocorticoid receptor, Receptor and Bioinformatics. His research in Inflammation intersects with topics in Tumor necrosis factor alpha, Signal transduction, Pharmacology, In vivo and Choroid plexus. His Tumor necrosis factor alpha study combines topics in areas such as Neuroinflammation, Cancer research and Inflammatory bowel disease.
Immunology connects with themes related to Dexamethasone in his study. His Glucocorticoid receptor research includes themes of Peroxisome proliferator-activated receptor, Phagocytosis and Sepsis. His study in Receptor is interdisciplinary in nature, drawing from both Nuclear receptor and Transcription factor.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Is there new hope for therapeutic matrix metalloproteinase inhibition
Roosmarijn E. Vandenbroucke;Claude Libert.
Nature Reviews Drug Discovery (2014)
Chemokine and cytokine processing by matrix metalloproteinases and its effect on leukocyte migration and inflammation.
Philippe Van Lint;Claude Libert.
Journal of Leukocyte Biology (2007)
The X chromosome in immune functions: when a chromosome makes the difference
Claude Libert;Lien Dejager;Iris Pinheiro.
Nature Reviews Immunology (2010)
Cecal ligation and puncture: the gold standard model for polymicrobial sepsis?
Lien Dejager;Iris Pinheiro;Iris Pinheiro;Eline Dejonckheere;Eline Dejonckheere;Claude Libert;Claude Libert.
Trends in Microbiology (2011)
α1-Acid glycoprotein: an acute phase protein with inflammatory and immunomodulating properties
Tino Hochepied;Franklin G Berger;Heinz Baumann;Claude Libert.
Cytokine & Growth Factor Reviews (2003)
RIP Kinase-Dependent Necrosis Drives Lethal Systemic Inflammatory Response Syndrome
Linde Duprez;Nozomi Takahashi;Nozomi Takahashi;Filip Van Hauwermeiren;Filip Van Hauwermeiren;Benjamin Vandendriessche;Benjamin Vandendriessche.
Essential Role of STAT3 in the Control of the Acute-Phase Response as Revealed by Inducible Gene Activation in the Liver
Tonino Alonzi;Diego Maritano;Barbara Gorgoni;Gabriella Rizzuto.
Molecular and Cellular Biology (2001)
How glucocorticoid receptors modulate the activity of other transcription factors: A scope beyond tethering
Dariusz Ratman;Wim Vanden Berghe;Lien Dejager;Claude Libert.
Molecular and Cellular Endocrinology (2013)
Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease models.
N Takahashi;L Duprez;S Grootjans;A Cauwels.
Cell Death and Disease (2012)
Caspase-14 protects against epidermal UVB photodamage and water loss.
Geertrui Denecker;Esther Hoste;Barbara Gilbert;Tino Hochepied.
Nature Cell Biology (2007)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: