Alan I. Faden focuses on Traumatic brain injury, Internal medicine, Endocrinology, Neuroscience and Anesthesia. His biological study spans a wide range of topics, including Traumatic injury, Head injury, Pathology, Intracellular and Hippocampus. His study connects Cortex and Endocrinology.
His Neuroscience research incorporates themes from Neuroinflammation, Neurodegeneration and Pharmacology. His study in Anesthesia is interdisciplinary in nature, drawing from both Spinal cord injury, Spinal cord, Blood pressure, -Naloxone and Pathophysiology. His research on -Naloxone also deals with topics like
Alan I. Faden mainly investigates Internal medicine, Endocrinology, Traumatic brain injury, Spinal cord injury and Neuroscience. He has researched Endocrinology in several fields, including Dynorphin and -Naloxone. As a part of the same scientific study, Alan I. Faden usually deals with the Traumatic brain injury, concentrating on Neuroinflammation and frequently concerns with Microglia.
His Spinal cord injury research is multidisciplinary, incorporating elements of Anesthesia and Endogenous opioid. His Anesthesia research includes themes of Pathophysiology and Ischemia. His Neuroscience research includes elements of Agonist and Receptor.
Alan I. Faden spends much of his time researching Traumatic brain injury, Neuroinflammation, Neuroscience, Neuroprotection and Microglia. His Traumatic brain injury study integrates concerns from other disciplines, such as Lesion, Surgery, Anesthesia, Cyclin-dependent kinase and Neurodegeneration. As part of the same scientific family, Alan I. Faden usually focuses on Neuroscience, concentrating on Systemic administration and intersecting with Cyclin A.
His Neuroprotection study is concerned with Pharmacology in general. His Microglia research incorporates elements of Endocrinology, Spinal cord injury and Pathology. His Endocrinology research is multidisciplinary, incorporating perspectives in Proinflammatory cytokine and Premovement neuronal activity.
The scientist’s investigation covers issues in Traumatic brain injury, Neuroscience, Neurodegeneration, Neuroinflammation and Neuroprotection. His work carried out in the field of Traumatic brain injury brings together such families of science as Cognition, Microglia, Cerebral perfusion pressure and Pathology. His research in Microglia tackles topics such as Lesion which are related to areas like NADPH oxidase, Nicotinamide adenine dinucleotide phosphate and Internal medicine.
His Internal medicine study typically links adjacent topics like Endocrinology. The Neuroinflammation study combines topics in areas such as Hippocampal formation and Cyclin-dependent kinase. His study in Neuroprotection is interdisciplinary in nature, drawing from both Surgery, Agonist, Intensive care medicine, Metabotropic glutamate receptor 5 and Programmed cell death.
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The role of excitatory amino acids and NMDA receptors in traumatic brain injury.
Alan I. Faden;Paul Demediuk;S. Scott Panter;Robert Vink.
Science (1989)
Activation of CPP32-Like Caspases Contributes to Neuronal Apoptosis and Neurological Dysfunction after Traumatic Brain Injury
Alexander G. Yakovlev;Susan M. Knoblach;Lei Fan;Gerard B. Fox.
The Journal of Neuroscience (1997)
MASCIS Evaluation of Open Field Locomotor Scores: Effects of Experience and Teamwork on Reliability
D. M. Basso;M. S. Beattie;J. C. Bresnahan;D. K. Anderson.
Journal of Neurotrauma (1996)
Naloxone reversal of endotoxin hypotension suggests role of endorphins in shock
J W Holaday;A I Faden.
Nature (1978)
Neuroprotection for traumatic brain injury: translational challenges and emerging therapeutic strategies.
David J. Loane;Alan I. Faden.
Trends in Pharmacological Sciences (2010)
Endogenous repair after spinal cord contusion injuries in the rat.
M. S. Beattie;J. C. Bresnahan;J. Komon;C. A. Tovar.
Experimental Neurology (1997)
Opiate antagonists: a role in the treatment of hypovolemic shock
A. I. Faden;J. W. Holaday.
Science (1979)
A potential role for excitotoxins in the pathophysiology of spinal cord injury.
Alan I. Faden;Roger P. Simon.
Annals of Neurology (1988)
Caspase pathways, neuronal apoptosis, and CNS injury.
Basil A. Eldadah;Alan I. Faden.
Journal of Neurotrauma (2000)
Cell cycle inhibition provides neuroprotection and reduces glial proliferation and scar formation after traumatic brain injury
Simone Di Giovanni;Vilen Movsesyan;Farid Ahmed;Ibolja Cernak.
Proceedings of the National Academy of Sciences of the United States of America (2005)
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