Sandy R. Shultz mainly investigates Traumatic brain injury, Neuroinflammation, Anesthesia, Neuroscience and Concussion. The Traumatic brain injury study combines topics in areas such as Head injury, Magnetic resonance imaging and Brain damage. His Neuroinflammation research is multidisciplinary, relying on both Neurology, Astrogliosis, Polytrauma and Autism.
In his study, Depression, Spinal cord injury, Cell adhesion molecule, Antibody and Spinal cord is inextricably linked to Oxidative stress, which falls within the broad field of Anesthesia. His Neuroscience study combines topics in areas such as Neuropathology, Endocrinology and Neurodegeneration. His Concussion study is concerned with Injury prevention in general.
The scientist’s investigation covers issues in Traumatic brain injury, Concussion, Neuroinflammation, Neuroscience and Epilepsy. His research in Traumatic brain injury is mostly focused on Closed head injury. Many of his research projects under Concussion are closely connected to Athletes, Biomarker, Magnetic resonance imaging and Diffusion MRI with Athletes, Biomarker, Magnetic resonance imaging and Diffusion MRI, tying the diverse disciplines of science together.
The study incorporates disciplines such as Neuropathology, Neurology, Brain damage and Microglia in addition to Neuroinflammation. His studies in Epilepsy integrate themes in fields like Neurocognitive and Disease. His biological study spans a wide range of topics, including Head injury and Clinical trial.
Sandy R. Shultz mainly investigates Traumatic brain injury, Concussion, Biomarker, Neuroscience and Athletes. His Traumatic brain injury research incorporates elements of Anesthesia, Hippocampus, Neuroinflammation, Polytrauma and Heterotopic ossification. His Neuroinflammation research integrates issues from Inflammasome, Neurology and Suprachiasmatic nucleus.
When carried out as part of a general Concussion research project, his work on Chronic traumatic encephalopathy is frequently linked to work in White matter, Physiology and Intensive care medicine, therefore connecting diverse disciplines of study. His work in the fields of Blood biomarkers overlaps with other areas such as Biomarker discovery. His studies deal with areas such as Inflammation and Clinical psychology as well as Athletes.
His primary scientific interests are in Traumatic brain injury, Polytrauma, Heterotopic ossification, Neuroscience and Concussion. The various areas that Sandy R. Shultz examines in his Traumatic brain injury study include Anesthesia, Intensive care medicine, Neuroinflammation, Hippocampus and Amygdala. His Neuroinflammation study integrates concerns from other disciplines, such as Rehabilitation, Fluid percussion, Epileptogenesis, Genetic ablation and Dementia.
His research integrates issues of Acute kidney injury, Neuropathology, Lung injury and Cause of death in his study of Polytrauma. Concussion is closely attributed to Alcohol Use Disorders Identification Test in his work. His Biomarker study combines topics in areas such as Inflammasome, Caspase 1 and Neurology.
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Intracerebroventricular Injection of Propionic Acid, an Enteric Bacterial Metabolic End-Product, Impairs Social Behavior in the Rat: Implications for an Animal Model of Autism
Sandy R. Shultz;Derrick F. MacFabe;Klaus Peter Ossenkopp;Shannon Scratch.
Neuropharmacology (2008)
Repeated mild lateral fluid percussion brain injury in the rat causes cumulative long-term behavioral impairments, neuroinflammation, and cortical loss in an animal model of repeated concussion.
Sandy Richard Shultz;Feng Bao;Vanessa Omana;Charlotte Chiu.
Journal of Neurotrauma (2012)
Inflammation in epileptogenesis after traumatic brain injury.
Kyria M. Webster;Mujun Sun;Peter Crack;Terence J. O’Brien.
Journal of Neuroinflammation (2017)
Intracerebroventricular injections of the enteric bacterial metabolic product propionic acid impair cognition and sensorimotor ability in the Long-Evans rat: further development of a rodent model of autism.
Sandy R. Shultz;Derrick F. MacFabe;Samantha Martin;Jordana Jackson.
Behavioural Brain Research (2009)
Sub-concussive brain injury in the Long-Evans rat induces acute neuroinflammation in the absence of behavioral impairments
Sandy R. Shultz;Derrick F. MacFabe;Kelly A. Foley;Roy Taylor.
Behavioural Brain Research (2012)
Can structural or functional changes following traumatic brain injury in the rat predict epileptic outcome
Sandy R. Shultz;Lisa Cardamone;Ying R. Liu;R. Edward Hogan.
Epilepsia (2013)
Sodium selenate reduces hyperphosphorylated tau and improves outcomes after traumatic brain injury
Sandy R Shultz;David K Wright;Ping Zheng;Ryan Stuchbery.
Brain (2015)
The potential for animal models to provide insight into mild traumatic brain injury: Translational challenges and strategies.
Sandy R. Shultz;Stuart J. McDonald;Cole Vonder Haar;Alicia Meconi.
Neuroscience & Biobehavioral Reviews (2017)
Progesterone treatment reduces neuroinflammation, oxidative stress and brain damage and improves long-term outcomes in a rat model of repeated mild traumatic brain injury
Kyria M. Webster;David K. Wright;David K. Wright;Mujun Sun;Bridgette D. Semple.
Journal of Neuroinflammation (2015)
A single mild fluid percussion injury induces short-term behavioral and neuropathological changes in the Long-Evans rat: support for an animal model of concussion
Sandy R. Shultz;Derrick F. MacFabe;Kelly A. Foley;Roy Taylor.
Behavioural Brain Research (2011)
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