His primary areas of study are Pathology, Programmed cell death, Anesthesia, Cell biology and Apoptosis. The Pathology study combines topics in areas such as Oxidative stress, Endocrinology, Central nervous system disease, Antioxidant and Ischemia. His work deals with themes such as Cerebrospinal fluid and Randomization, which intersect with Anesthesia.
His Cerebrospinal fluid research includes themes of Meningitis and Glasgow Coma Scale. Robert S. B. Clark has researched Cell biology in several fields, including Autophagy, Autophagy database, Apoptosis-inducing factor and Autolysosome. His Apoptosis study combines topics from a wide range of disciplines, such as Cerebral cortex, Neuroscience, Neuroprotection and Immunology.
His primary areas of investigation include Anesthesia, Internal medicine, Pathology, Cerebrospinal fluid and Endocrinology. His Anesthesia research includes elements of Adenosine and Neuroprotection. He interconnects Gastroenterology and Cardiology in the investigation of issues within Internal medicine.
His Pathology research integrates issues from Hippocampus, Blood–brain barrier and Pharmacology. His research in Cerebrospinal fluid intersects with topics in Glasgow Coma Scale, Central nervous system disease, Immunology and Meningitis. His studies link Apoptosis with Endocrinology.
His primary areas of study are Internal medicine, Emergency medicine, Intracranial pressure, Neuroscience and Odds ratio. His Internal medicine study combines topics in areas such as Gastroenterology, Endocrinology, Gut flora and Cardiology. He combines subjects such as Hydrocephalus, Presentation, Cerebral perfusion pressure and Neurointensive care with his study of Intracranial pressure.
His Neuroscience research includes themes of Pharmacotherapy, Cell type, Homeostasis and Ischemia. His Retrospective cohort study research focuses on Cohort and how it connects with Discontinuation and Anesthesia. His research integrates issues of Hippocampus and Cerebral edema in his study of Pathology.
The scientist’s investigation covers issues in Internal medicine, Flux, Neuroscience, Organ dysfunction and Gastroenterology. His Internal medicine research focuses on Ferritin, Prospective cohort study, Hippocampal formation and Inflammation. His Flux research incorporates themes from Programmed cell death, Cell type, Homeostasis and Ischemia.
His study in Neuroscience is interdisciplinary in nature, drawing from both Autophagy, Drug, Mitochondrion and Drug discovery. His work carried out in the field of Organ dysfunction brings together such families of science as Biomarker, Inpatient mortality, Hospitalized patients and C-reactive protein. Robert S. B. Clark has included themes like Gut flora, Gut–brain axis and Head injury in his Gastroenterology study.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)
Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes
Daniel J. Klionsky;Hagai Abeliovich;Patrizia Agostinis;Devendra K. Agrawal.
Autophagy (2008)
Unexpected Increased Mortality After Implementation of a Commercially Sold Computerized Physician Order Entry System
Yong Y. Han;Joseph A. Carcillo;Shekhar T. Venkataraman;Robert S.B. Clark.
Pediatrics (2005)
The far-reaching scope of neuroinflammation after traumatic brain injury
Dennis W. Simon;Mandy J. McGeachy;Hülya Bayır;Robert S. B. Clark.
Nature Reviews Neurology (2017)
Alzheimer's pathology in human temporal cortex surgically excised after severe brain injury.
Milos D. Ikonomovic;Kunihiro Uryu;Eric E. Abrahamson;John R. Ciallella.
Experimental Neurology (2004)
Caspase-3 mediated neuronal death after traumatic brain injury in rats.
R. S. B. Clark;P. M. Kochanek;S. C. Watkins;Minzhi Chen.
Journal of Neurochemistry (2001)
To die or not to die for neurons in ischemia, traumatic brain injury and epilepsy: a review on the stress-activated signaling pathways and apoptotic pathways
Anthony K.F Liou;Robert S Clark;David C Henshall;Xiao-Ming Yin.
Progress in Neurobiology (2003)
Neutrophil Accumulation After Traumatic Brain Injury in Rats: Comparison of Weight Drop and Controlled Cortical Impact Models
Robert S.B. Clark;Joanne K. Schiding;Susan L. Kaczorowski;Donald W. Marion.
Journal of Neurotrauma (1994)
Innate gender-based proclivity in response to cytotoxicity and programmed cell death pathway.
Lina Du;Hülya Bayir;Yichen Lai;Xiaopeng Zhang.
Journal of Biological Chemistry (2004)
Intra-mitochondrial Poly(ADP-ribosylation) Contributes to NAD+ Depletion and Cell Death Induced by Oxidative Stress *
Lina Du;Xiaopeng Zhang;Yong Y. Han;Nancy A. Burke.
Journal of Biological Chemistry (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Pittsburgh
University of Pittsburgh
University of Pittsburgh
University of Pittsburgh
University of Pittsburgh
University of Pittsburgh
University of Florida
University of Pittsburgh
University of Pittsburgh
University of Pittsburgh
Central Queensland University
Aristotle University of Thessaloniki
Friedrich Schiller University Jena
University of Tokyo
University of Zurich
Donald Danforth Plant Science Center
Stanford University
University of Lausanne
Plymouth University
University of Maryland Center For Environmental Sciences
Institute of Science and Technology Austria
University of Maryland, Baltimore
University of Western Ontario
University of Bristol
University of Georgia
University of Colorado Anschutz Medical Campus