D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 84 Citations 24,665 380 World Ranking 10054 National Ranking 5303
Biology and Biochemistry D-index 84 Citations 24,524 365 World Ranking 2107 National Ranking 1174

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

The scientist’s investigation covers issues in Calpain, Traumatic brain injury, Biochemistry, Cell biology and Molecular biology. The various areas that he examines in his Calpain study include Caspase 3, Apoptosis, Protease and Proteases. His Traumatic brain injury research incorporates elements of Biomarker, Internal medicine, Cerebrospinal fluid, Pathology and Glasgow Coma Scale.

His Biochemistry study incorporates themes from Neuroprotection and Pharmacology. His Cell biology research is multidisciplinary, incorporating perspectives in Neurodegeneration and Excitatory postsynaptic potential. The Molecular biology study combines topics in areas such as Heterozygote advantage, Protein subunit, In vitro and Chimera.

His most cited work include:

  • Calpain and caspase : can you tell the difference? (851 citations)
  • Traumatic brain injury: integrated approaches to improve prevention, clinical care, and research (556 citations)
  • Non-erythroid alpha-spectrin breakdown by calpain and interleukin 1 beta-converting-enzyme-like protease(s) in apoptotic cells: contributory roles of both protease families in neuronal apoptosis. (418 citations)

What are the main themes of his work throughout his whole career to date?

Kevin K.W. Wang spends much of his time researching Traumatic brain injury, Calpain, Pathology, Internal medicine and Biomarker. His study on Traumatic brain injury also encompasses disciplines like

  • Glasgow Coma Scale that intertwine with fields like Prospective cohort study,
  • Anesthesia together with Neuroprotection. His study in Calpain is interdisciplinary in nature, drawing from both Proteases, Caspase, Molecular biology and Cell biology.

In his study, Spectrin is strongly linked to Caspase 3, which falls under the umbrella field of Caspase. His research in Internal medicine intersects with topics in Endocrinology and Oncology. Kevin K.W. Wang interconnects Stroke, Clinical trial, Bioinformatics and Proteomics in the investigation of issues within Biomarker.

He most often published in these fields:

  • Traumatic brain injury (45.19%)
  • Calpain (29.53%)
  • Pathology (18.79%)

What were the highlights of his more recent work (between 2017-2021)?

  • Traumatic brain injury (45.19%)
  • Biomarker (17.00%)
  • Internal medicine (18.34%)

In recent papers he was focusing on the following fields of study:

Kevin K.W. Wang focuses on Traumatic brain injury, Biomarker, Internal medicine, Tauopathy and Glasgow Coma Scale. His work carried out in the field of Traumatic brain injury brings together such families of science as Emergency department, Glial fibrillary acidic protein, Pathology, Magnetic resonance imaging and Autoimmunity. The concepts of his Biomarker study are interwoven with issues in Concussion, Clinical trial, Bioinformatics and Epilepsy.

His Internal medicine research integrates issues from Gastroenterology, Endocrinology and Oncology. His Proteolysis research includes elements of Alzheimer's disease and Calpain. His Calpain research is multidisciplinary, incorporating elements of Proteases and Caspase.

Between 2017 and 2021, his most popular works were:

  • An update on diagnostic and prognostic biomarkers for traumatic brain injury. (105 citations)
  • Case-mix, care pathways, and outcomes in patients with traumatic brain injury in CENTER-TBI: a European prospective, multicentre, longitudinal, cohort study (69 citations)
  • Protein Biomarkers and Neuroproteomics Characterization of Microvesicles/Exosomes from Human Cerebrospinal Fluid Following Traumatic Brain Injury. (59 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

His primary areas of study are Traumatic brain injury, Glasgow Coma Scale, Internal medicine, Cohort study and Tauopathy. He has researched Traumatic brain injury in several fields, including Clinical trial, Randomized controlled trial, Intensive care medicine, Protein biomarkers and Receiver operating characteristic. He has included themes like Brain trauma, Emergency department, Prospective cohort study, Depression and Comorbidity in his Glasgow Coma Scale study.

His Internal medicine study integrates concerns from other disciplines, such as Computed tomography and Symptom reporting. His Exosome research incorporates themes from Extracellular, Calpain, Staurosporine, Cytotoxic T cell and Synaptophysin. His studies in Microvesicles integrate themes in fields like Molecular biology and Caspase.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Traumatic brain injury: integrated approaches to improve prevention, clinical care, and research

Andrew I R Maas;David K Menon;P David Adelson;Nada Andelic.
Lancet Neurology (2017)

1300 Citations

Calpain and caspase : can you tell the difference?

Kevin K.W Wang.
Trends in Neurosciences (2000)

1150 Citations

Non-erythroid alpha-spectrin breakdown by calpain and interleukin 1 beta-converting-enzyme-like protease(s) in apoptotic cells: contributory roles of both protease families in neuronal apoptosis.

Rathna Nath;Kadee J Raser;Daniel Stafford;Iradj Hajimohammadreza.
Biochemical Journal (1996)

640 Citations

The calpain family and human disease

Yuanhui Huang;Kevin K.W. Wang.
Trends in Molecular Medicine (2001)

597 Citations

Glial fibrillary acidic protein: from intermediate filament assembly and gliosis to neurobiomarker

Zhihui Yang;Kevin K.W. Wang.
Trends in Neurosciences (2015)

466 Citations

CALPAIN INHIBITION : AN OVERVIEW OF ITS THERAPEUTIC POTENTIAL

Kevin K.W. Wang;Yuen Po-Wai.
Trends in Pharmacological Sciences (1994)

399 Citations

Cytochrome c Release and Caspase Activation in Traumatic Axonal Injury

András Büki;David O. Okonkwo;Kevin K. W. Wang;John T. Povlishock.
The Journal of Neuroscience (2000)

386 Citations

Correction: Corrigendum: Temporal MRI characterization, neurobiochemical and neurobehavioral changes in a mouse repetitive concussive head injury model

Zhihui Yang;Ping Wang;Drake Morgan;Adriaan W. Bruijnzeel.
Scientific Reports (2015)

377 Citations

Simultaneous Degradation of αII- and βII-Spectrin by Caspase 3 (CPP32) in Apoptotic Cells

Kevin K.W. Wang;Rand Posmantur;Rathna Nath;Kim McGinnis.
Journal of Biological Chemistry (1998)

376 Citations

Elevated levels of serum glial fibrillary acidic protein breakdown products in mild and moderate traumatic brain injury are associated with intracranial lesions and neurosurgical intervention.

Linda Papa;Lawrence M. Lewis;Jay L. Falk;Zhiqun Zhang.
Annals of Emergency Medicine (2012)

361 Citations

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