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Neuroscience

D-Index
82
Citations
19857
World Ranking
1529
National Ranking
757

Overview

C. Edward Dixon is affiliated with the University of Pittsburgh in the United States and focuses primarily on research in medicine and biochemistry, genetics, and molecular biology. Their work spans multiple interconnected fields of study with a notable emphasis on neurology and molecular biology, reflecting a multidisciplinary approach to scientific investigation.

The scientist's main fields of study include:

  • Medicine
  • Biochemistry, Genetics and Molecular Biology

Within these broader areas, their research delves into several subfields such as:

  • Neurology
  • Molecular Biology
  • Epidemiology
  • Cellular and Molecular Neuroscience
  • Emergency Medicine

The core topics investigated include traumatic brain injury and its associated neurovascular disturbances, research directly focusing on traumatic brain injury, mitochondrial function and pathology, cardiac arrest and resuscitation, S100 proteins and annexins, ubiquitin and proteasome pathways, and cerebrospinal fluid and hydrocephalus.

  • Traumatic Brain Injury and Neurovascular Disturbances
  • Traumatic Brain Injury Research
  • Mitochondrial Function and Pathology
  • Cardiac Arrest and Resuscitation
  • S100 Proteins and Annexins
  • Ubiquitin and proteasome pathways
  • Cerebrospinal fluid and hydrocephalus

Several frequent publication venues for Dixon's work underline their focus on neurological trauma and neurobiology:

  • Journal of Neurotrauma
  • Experimental Neurology
  • Neurotrauma Reports
  • Molecular Neurobiology
  • Neuroscience

Dixon has collaborated regularly with a series of coauthors, suggesting ongoing research partnerships and collaborative projects. These frequent coauthors include:

  • Jeremy Henchir
  • Shaun W. Carlson
  • Audrey D. Lafrenaye
  • Patrick M. Kochanek
  • John T. Povlishock

Some of their recent papers, illustrating the scope and focus of their work, are:

  • "Pre-Clinical Common Data Elements for Traumatic Brain Injury Research: Progress and Use Cases," 2020, Journal of Neurotrauma
  • "Glibenclamide Treatment in Traumatic Brain Injury: Operation Brain Trauma Therapy," 2020, Journal of Neurotrauma
  • "Abolishing UCHL1's hydrolase activity exacerbates TBI-induced axonal injury and neuronal death in mice," 2020, Experimental Neurology
  • "Reductions in Synaptic Vesicle Glycoprotein 2 Isoforms in the Cortex and Hippocampus in a Rat Model of Traumatic Brain Injury," 2021, Molecular Neurobiology
  • "Spatial Distribution of Neuropathology and Neuroinflammation Elucidate the Biomechanics of Fluid Percussion Injury," 2021, Neurotrauma Reports

Best Publications

  • A controlled cortical impact model of traumatic brain injury in the rat.

    C E Dixon;G L Clifton;J W Lighthall;A A Yaghmai

  • A model of parasagittal controlled cortical impact in the mouse: cognitive and histopathologic effects.

    D H Smith;H D Soares;J S Pierce;K G Perlman

  • Caspase-3 mediated neuronal death after traumatic brain injury in rats.

    R. S. B. Clark;P. M. Kochanek;S. C. Watkins;Minzhi Chen

  • Prolonged memory impairment in the absence of hippocampal cell death following traumatic brain injury in the rat.

    Bruce G Lyeth;L. W. Jenkins;R. J. Hamm;C. E. Dixon

  • Effects of Matrix Metalloproteinase-9 Gene Knock-Out on Morphological and Motor Outcomes after Traumatic Brain Injury

    Xiaoying Wang;JaeChang Jung;Minoru Asahi;Wilson Chwang

  • Increased vulnerability of the midly traumatized rat brain to cerebral ischemia: the use of controlled secondary ischemia as a research tool to identify common or different mechanisms contributing to mechanical and ischemic brain injury

    L. W. Jenkins;K. Moszynski;Bruce G Lyeth;W. Lewelt

  • Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis

    James W. Bales;Amy K. Wagner;Anthony E. Kline;C. Edward Dixon

  • Inducible nitric oxide synthase is an endogenous neuroprotectant after traumatic brain injury in rats and mice.

    Elizabeth H. Sinz;Patrick M. Kochanek;C. Edward Dixon;Robert S.B. Clark

  • Ferroptosis Contributes to Neuronal Death and Functional Outcome After Traumatic Brain Injury

    Elizabeth M Kenny;Emin Fidan;Qin Yang;Tamil S Anthonymuthu

  • Blast exposure in rats with body shielding is characterized primarily by diffuse axonal injury.

    Robert H. Garman;Larry W. Jenkins;Robert C. Switzer;Richard A. Bauman

  • Early neuropathologic effects of mild or moderate hypoxemia after controlled cortical impact injury in rats.

    Robert S. B. Clark;Patrick M. Kochanek;C. Edward Dixon;Minzhi Chen

  • Adenosine A1 receptor knockout mice develop lethal status epilepticus after experimental traumatic brain injury.

    Patrick M Kochanek;Vincent A Vagni;Keri L Janesko;Christopher B Washington

  • Mitogen-Activated Protein Kinase Inhibition in Traumatic Brain Injury: In Vitro and In Vivo Effects:

    Tatsuro Mori;Xiaoying Wang;Jae-Chang Jung;Toshihisa Sumii

  • Autophagy is increased after traumatic brain injury in mice and is partially inhibited by the antioxidant gamma-glutamylcysteinyl ethyl ester.

    Lai Y;Hickey Rw;Chen Y;Bayir H

  • Neurofilament 68 and Neurofilament 200 Protein Levels Decrease After Traumatic Brain Injury

    R Posmantur;R L Hayes;C E Dixon;W C Taft

  • Reduction of cognitive and motor deficits after traumatic brain injury in mice deficient in poly(ADP-ribose) polymerase.

    Michael J. Whalen;Robert S B Clark;C. Edward Dixon;Paul Robichaud

  • Comparison of seven anesthetic agents on outcome after experimental traumatic brain injury in adult, male rats.

    Kimberly D. Statler;Henry Alexander;Vincent Vagni;C. Edward Dixon

  • Attenuation of working memory and spatial acquisition deficits after a delayed and chronic bromocriptine treatment regimen in rats subjected to traumatic brain injury by controlled cortical impact

    Anthony E. Kline;Jaime L. Massucci;Donald W. Marion;C. Edward Dixon

  • Effect of hyperventilation on extracellular concentrations of glutamate, lactate, pyruvate, and local cerebral blood flow in patients with severe traumatic brain injury.

    Donald W. Marion;Ava Puccio;Stephen R. Wisniewski;Patrick Kochanek

  • Microtubule-associated protein 2 levels decrease in hippocampus following traumatic brain injury.

    William C. Taft;Keyi Yang;C. Edward Dixon;Ronald L. Hayes

  • Role of cyclooxygenase 2 in acute spinal cord injury

    D K Resnick;S H Graham;C E Dixon;D W Marion

Frequent Co-Authors

Anthony E. Kline
Anthony E. Kline University of Pittsburgh
Amy K. Wagner
Amy K. Wagner University of Pittsburgh
W. Dalton Dietrich
W. Dalton Dietrich University of Miami
Milos D. Ikonomovic
Milos D. Ikonomovic University of Pittsburgh
Steven T. DeKosky
Steven T. DeKosky University of Florida
Xiaoming Hu
Xiaoming Hu University of Pittsburgh
Jonathan Lifshitz
Jonathan Lifshitz University of Arizona
Dandan Sun
Dandan Sun University of Pittsburgh
Eng H. Lo
Eng H. Lo Harvard University
Michael V. L. Bennett
Michael V. L. Bennett Albert Einstein College of Medicine

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