Anthony N. Moore focuses on Neuroscience, Hippocampus, Pathology, Morris water navigation task and Phosphorylation. His research in Neuroscience intersects with topics in Progenitor cell and Gene. The various areas that he examines in his Hippocampus study include Neurochemical, Fear conditioning and Pharmacology.
His Pathology research incorporates themes from Intracranial pressure and Sulforaphane. His Morris water navigation task research is multidisciplinary, incorporating elements of Ribosomal s6 kinase, P70-S6 Kinase 1, Growth factor and Activator. His work in the fields of Phosphorylation, such as Protein kinase A, intersects with other areas such as Response element.
His primary areas of study are Neuroscience, Hippocampus, Internal medicine, Pathology and Endocrinology. In his study, CREB is inextricably linked to Protein kinase A, which falls within the broad field of Neuroscience. His Hippocampus research focuses on subjects like Fear conditioning, which are linked to Classical conditioning.
The concepts of his Internal medicine study are interwoven with issues in Glasgow Coma Scale and Intracranial pressure. His research integrates issues of Inflammation, Blood–brain barrier and Pharmacology in his study of Pathology. The Morris water navigation task study combines topics in areas such as Long-term memory, Central nervous system, Phosphorylation and Wortmannin.
Anthony N. Moore mostly deals with Neuroscience, Hippocampus, Hippocampal formation, Endocrinology and Internal medicine. His is doing research in Freezing behavior, Dendritic spine, Nucleus, Lateral hypothalamus and Amygdala, both of which are found in Neuroscience. He has researched Hippocampus in several fields, including Neurogenesis, CHOP, Unfolded protein response and Neuroplasticity.
His Hippocampal formation research is multidisciplinary, relying on both Biopsy, Corpus callosum, Engram and Cellular respiration. His Dopaminergic and Dopamine receptor D2 study in the realm of Endocrinology connects with subjects such as Metabolic syndrome and Autism spectrum disorder. In general Internal medicine study, his work on Tyrosine hydroxylase, Dopamine receptor D1 and Dopamine often relates to the realm of Tryptophan hydroxylase, thereby connecting several areas of interest.
His primary areas of investigation include Hippocampus, Neuroscience, Transgene, Doublecortin and Neuroplasticity. Anthony N. Moore has included themes like Acetylcholine, Nicotinic agonist, Acetylcholine receptor and Pathology in his Hippocampus study. Anthony N. Moore integrates many fields in his works, including Neuroscience, Vascular dementia and Galantamine.
His Transgene study combines topics in areas such as Water maze, Phosphorylation, Cell biology and Morris water navigation task. His studies deal with areas such as Long-term memory and Activator as well as Morris water navigation task. His work carried out in the field of Doublecortin brings together such families of science as Neurogenesis, Unfolded protein response and CHOP.
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A Mitogen-Activated Protein Kinase Cascade in the CA1/CA2 Subfield of the Dorsal Hippocampus Is Essential for Long-Term Spatial Memory
Sonja Blum;Anthony N. Moore;Frank Adams;Pramod K. Dash.
The Journal of Neuroscience (1999)
Enhanced neurogenesis in the rodent hippocampus following traumatic brain injury.
P.K. Dash;S.A. Mach;A.N. Moore.
Journal of Neuroscience Research (2001)
Enhancing Expression of Nrf2-Driven Genes Protects the Blood–Brain Barrier after Brain Injury
Jing Zhao;Anthony N. Moore;John B. Redell;Pramod K. Dash.
The Journal of Neuroscience (2007)
A role for prefrontal cortex in memory storage for trace fear conditioning.
Jason D. Runyan;Anthony N. Moore;Pramod K. Dash.
The Journal of Neuroscience (2004)
Human traumatic brain injury alters plasma microRNA levels.
John B. Redell;Anthony N. Moore;Norman H. Ward;Georgene W. Hergenroeder.
Journal of Neurotrauma (2010)
Biomarkers for the diagnosis and prognosis of mild traumatic brain injury/concussion.
Cameron B. Jeter;Georgene W. Hergenroeder;Michael J. Hylin;John B. Redell.
Journal of Neurotrauma (2013)
Biomarkers for the diagnosis, prognosis, and evaluation of treatment efficacy for traumatic brain injury
Pramod K. Dash;Jing Zhao;Georgene Hergenroeder;Anthony N. Moore.
Sulforaphane enhances aquaporin-4 expression and decreases cerebral edema following traumatic brain injury.
Jing Zhao;Anthony N. Moore;Guy L. Clifton;Pramod K. Dash.
Journal of Neuroscience Research (2005)
Spatial Memory Formation and Memory-Enhancing Effect of Glucose Involves Activation of the Tuberous Sclerosis Complex–Mammalian Target of Rapamycin Pathway
Pramod K. Dash;Sara A. Orsi;Anthony N. Moore.
The Journal of Neuroscience (2006)
Valproate administered after traumatic brain injury provides neuroprotection and improves cognitive function in rats.
Pramod K. Dash;Sara A. Orsi;Min Zhang;Raymond J. Grill.
PLOS ONE (2010)
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