What is he best known for?
The fields of study he is best known for:
J. David Sweatt mainly focuses on Neuroscience, Synaptic plasticity, Epigenetics in learning and memory, DNA methylation and Long-term potentiation.
His research links Epigenetics with Neuroscience.
His biological study spans a wide range of topics, including Endocrinology, MAPK/ERK pathway and Cell biology.
His Epigenetics in learning and memory study combines topics from a wide range of disciplines, such as Molecular biology and Histone methylation.
His study in DNA methylation is interdisciplinary in nature, drawing from both Neurotrophic factors and Methylation.
J. David Sweatt has included themes like NMDA receptor, Reelin and Neurotransmission in his Long-term potentiation study.
His most cited work include:
- The neuronal MAP kinase cascade : A biochemical signal integration system subserving synaptic plasticity and memory (1035 citations)
- Lasting epigenetic influence of early-life adversity on the BDNF gene. (963 citations)
- Covalent Modification of DNA Regulates Memory Formation (942 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Neuroscience, Synaptic plasticity, Epigenetics, Long-term potentiation and Cell biology.
Neuroscience is often connected to Epigenetics in learning and memory in his work.
His Synaptic plasticity research is multidisciplinary, incorporating elements of Endocrinology and Excitatory postsynaptic potential.
His Epigenetics study combines topics in areas such as Chromatin, Regulation of gene expression, Histone and DNA methylation.
He has researched Long-term potentiation in several fields, including NMDA receptor, Protein kinase C and Neurotransmission.
His Cell biology study frequently links to other fields, such as Biochemistry.
He most often published in these fields:
- Neuroscience (65.52%)
- Synaptic plasticity (28.02%)
- Epigenetics (24.14%)
What were the highlights of his more recent work (between 2014-2020)?
- Neuroscience (65.52%)
- Epigenetics (24.14%)
- DNA methylation (15.09%)
In recent papers he was focusing on the following fields of study:
J. David Sweatt focuses on Neuroscience, Epigenetics, DNA methylation, Synaptic plasticity and Genetics.
J. David Sweatt combines subjects such as Epigenetics in learning and memory and Metaplasticity with his study of Neuroscience.
His research integrates issues of Chromatin, Histone and Methylation, Methyltransferase in his study of Epigenetics.
His work carried out in the field of Synaptic plasticity brings together such families of science as Hippocampal formation, Long-term potentiation, Regulator and Excitatory postsynaptic potential.
His studies deal with areas such as Pathophysiology and Autism spectrum disorder as well as Genetics.
As a part of the same scientific study, he usually deals with the Biochemistry, concentrating on Cell biology and frequently concerns with Gene.
Between 2014 and 2020, his most popular works were:
- Neural plasticity and behavior - sixty years of conceptual advances. (172 citations)
- DNA methylation regulates neuronal glutamatergic synaptic scaling (77 citations)
- Tcf4 Regulates Synaptic Plasticity, DNA Methylation, and Memory Function (71 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Neuroscience, Synaptic plasticity, DNA methylation, Synaptic scaling and Epigenetics.
His Neuroscience research includes themes of Long-term potentiation, Histone and Regulation of gene expression.
In his research, Gene knockdown is intimately related to Epigenetics in learning and memory, which falls under the overarching field of Long-term potentiation.
As part of the same scientific family, he usually focuses on Synaptic plasticity, concentrating on Regulator and intersecting with Computational biology, DNA demethylation and Memory formation.
His Synaptic scaling research integrates issues from Synaptic fatigue, Homeostatic plasticity and Cell biology.
The concepts of his Epigenetics study are interwoven with issues in Epigenomics and Gene expression.
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