2019 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of investigation include Apoptosis, Cell biology, Ischemia, Programmed cell death and Endocrinology. His Apoptosis research is multidisciplinary, incorporating perspectives in Immunology, Dentate gyrus, Neuroscience and Mitochondrion. His study looks at the relationship between Cell biology and fields such as Neuroprotection, as well as how they intersect with chemical problems.
His Ischemia study incorporates themes from Glutamate receptor, Molecular biology, Cerebral infarction and Pathology. His research in Programmed cell death tackles topics such as Oxidative stress which are related to areas like In vitro and Central nervous system disease. The various areas that Steven H. Graham examines in his Endocrinology study include Inflammation, Internal medicine, Glutathione and Cortex.
His primary scientific interests are in Ischemia, Internal medicine, Endocrinology, Programmed cell death and Anesthesia. Steven H. Graham has researched Ischemia in several fields, including Glutamate receptor, Neuroprotection and Pathology. His Endocrinology study combines topics in areas such as Cyclooxygenase and Excitatory postsynaptic potential.
Steven H. Graham combines subjects such as Molecular biology and Cell biology with his study of Programmed cell death. In Anesthesia, he works on issues like Central nervous system disease, which are connected to Cerebrospinal fluid. He interconnects Neuroscience and Mitochondrion in the investigation of issues within Apoptosis.
His main research concerns Programmed cell death, Brain ischemia, Neuroprotection, Cyclopentenone prostaglandins and Ischemia. His study in Programmed cell death is interdisciplinary in nature, drawing from both Autophagy, Endocrinology, Cell biology, Proteasome and Internal medicine. His research combines Molecular biology and Cell biology.
His Brain ischemia research incorporates themes from Mitochondrion, Surgery, Pharmacology and Pathology. His work deals with themes such as Cytochrome, Neuroscience and Apoptosis, which intersect with Mitochondrion. The concepts of his Ischemia study are interwoven with issues in Inflammation and Neurosurgery.
Cell biology, Ischemia, Molecular biology, Signal transduction and Stroke are his primary areas of study. His Cell biology research incorporates elements of Prostaglandin and Programmed cell death. His study on Ischemic injury is often connected to Time windows as part of broader study in Ischemia.
Steven H. Graham has included themes like Protein kinase C, Kinase, Phosphorylation and ASK1 in his Molecular biology study. His Signal transduction study integrates concerns from other disciplines, such as Basic science, Neuroscience, Neuroprotection, Pathology and Clinical success. His research integrates issues of Cyclooxygenase, Inflammation, Mitochondrion and Bioinformatics in his study of Stroke.
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Adverse Cerebral Outcomes after Coronary Bypass Surgery
G W Roach;M Kanchuger;C M Mangano;M Newman.
The New England Journal of Medicine (1996)
Induction of caspase-3-like protease may mediate delayed neuronal death in the hippocampus after transient cerebral ischemia.
Jun Chen;Tetsuya Nagayama;Kunlin Jin;R. Anne Stetler;R. Anne Stetler.
The Journal of Neuroscience (1998)
Programmed Cell Death in Cerebral Ischemia
Steven H. Graham;Jun Chen.
Journal of Cerebral Blood Flow and Metabolism (2001)
Cannabinoids and Neuroprotection in Global and Focal Cerebral Ischemia and in Neuronal Cultures
Tetsuya Nagayama;Amy D. Sinor;Roger P. Simon;Jun Chen.
The Journal of Neuroscience (1999)
In Vivo Delivery of a Bcl-xL Fusion Protein Containing the TAT Protein Transduction Domain Protects against Ischemic Brain Injury and Neuronal Apoptosis
Guodong Cao;Wei Pei;Hailiang Ge;Qinhua Liang.
The Journal of Neuroscience (2002)
Caspase-3 mediated neuronal death after traumatic brain injury in rats.
R. S. B. Clark;P. M. Kochanek;S. C. Watkins;Minzhi Chen.
Journal of Neurochemistry (2001)
Stress proteins and tolerance to focal cerebral ischemia
Jun Chen;Steven H. Graham;Raymond L. Zhu;Roger P. Simon.
Journal of Cerebral Blood Flow and Metabolism (1996)
Cyclooxygenase-2 inhibition prevents delayed death of CA1 hippocampal neurons following global ischemia.
Masaki Nakayama;Koichi Uchimura;Raymond Li Zhu;Tetsuya Nagayama.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Cerebral Injury After Cardiac Surgery: Identification of a Group at Extraordinary Risk
Richard L. Wolman;Nancy A. Nussmeier;Anil Aggarwal;Marc S. Kanchuger.
Innate gender-based proclivity in response to cytotoxicity and programmed cell death pathway.
Lina Du;Hülya Bayir;Yichen Lai;Xiaopeng Zhang.
Journal of Biological Chemistry (2004)
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