What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in SOD1, Cell biology, Mutant, Amyotrophic lateral sclerosis and Superoxide dismutase.
Zuoshang Xu has researched SOD1 in several fields, including Motor neuron and Mitochondrion.
His biological study spans a wide range of topics, including Intermediate filament, Protein filament, Transfection and Anatomy.
His studies examine the connections between Amyotrophic lateral sclerosis and genetics, as well as such issues in Mutation, with regards to Actin and Filamentous actin.
As part of the same scientific family, Zuoshang Xu usually focuses on Superoxide dismutase, concentrating on Molecular biology and intersecting with Neurodegeneration and Superoxide.
His research investigates the connection with Small interfering RNA and areas like RNA interference which intersect with concerns in Enzyme complex.
His most cited work include:
- Asymmetry in the assembly of the RNAi enzyme complex. (2528 citations)
- Massive Mitochondrial Degeneration in Motor Neurons Triggers the Onset of Amyotrophic Lateral Sclerosis in Mice Expressing a Mutant SOD1 (629 citations)
- Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity (540 citations)
What are the main themes of his work throughout his whole career to date?
Zuoshang Xu spends much of his time researching Amyotrophic lateral sclerosis, SOD1, RNA interference, Cell biology and Genetically modified mouse.
His Amyotrophic lateral sclerosis research incorporates elements of Motor neuron, Neuroscience, Spinal cord and Neurodegeneration.
As a part of the same scientific study, Zuoshang Xu usually deals with the SOD1, concentrating on Mitochondrion and frequently concerns with Mitochondrial permeability transition pore.
Zuoshang Xu's looking at RNA interference as part of his Genetics and RNA and RNA interference study.
His research in Genetically modified mouse intersects with topics in Axoplasmic transport, Axon, Anatomy, Protein subunit and Wild type.
The Mutant study combines topics in areas such as Molecular biology, Gene mutation and Enzyme.
He most often published in these fields:
- Amyotrophic lateral sclerosis (35.63%)
- SOD1 (34.48%)
- RNA interference (26.44%)
What were the highlights of his more recent work (between 2012-2019)?
- Amyotrophic lateral sclerosis (35.63%)
- Central nervous system (12.64%)
- Neuroscience (22.99%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Amyotrophic lateral sclerosis, Central nervous system, Neuroscience, Spinal cord and Genetic enhancement.
The study of Amyotrophic lateral sclerosis is intertwined with the study of Cell biology in a number of ways.
Zuoshang Xu combines subjects such as Motor neuron and Denervation with his study of Cell biology.
His Neuroscience research is multidisciplinary, relying on both Mutation, Mutant, Neurodegeneration and Gene knockdown.
The Spinal cord study combines topics in areas such as Endocrinology, Lysosomal storage disease, Astrogliosis, Pathology and SOD1.
In his research on the topic of SOD1, Homologous chromosome and Function is strongly related with RNA interference.
Between 2012 and 2019, his most popular works were:
- Reactive astrocytes secrete lcn2 to promote neuron death (174 citations)
- Global CNS transduction of adult mice by intravenously delivered rAAVrh.8 and rAAVrh.10 and nonhuman primates by rAAVrh.10. (131 citations)
- Partial loss of TDP-43 function causes phenotypes of amyotrophic lateral sclerosis (97 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Central nervous system, Transgene, Genetic enhancement, Immunology and Amyotrophic lateral sclerosis.
His studies deal with areas such as Gene delivery, Neuroscience, Gene knockdown and Pathogenesis as well as Transgene.
His studies in Genetic enhancement integrate themes in fields like Cancer research and Transduction.
His Immunology research incorporates elements of RNA interference, RNAi Therapeutics and SOD1.
His work on Spinal cord expands to the thematically related Amyotrophic lateral sclerosis.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
