John W. Griffin spends much of his time researching Pathology, Anatomy, Neuroscience, Immunology and Axon. His Pathology research includes themes of Antibody, Acute motor axonal neuropathy and Sensory nerve. His research integrates issues of Epidermis, Atrophy and RUNX1 in his study of Neuroscience.
His Immunology research includes elements of Prednisone and Ganglioside. His Axon research is multidisciplinary, incorporating perspectives in Axolemma and Neurofilament. His research in Neurofilament intersects with topics in Spinal cord, Axoplasmic transport, Cell biology and Cytoskeleton.
John W. Griffin mainly focuses on Pathology, Neuroscience, Axon, Anatomy and Neurofilament. His work investigates the relationship between Pathology and topics such as Peripheral neuropathy that intersect with problems in Internal medicine. His Neuroscience research is multidisciplinary, relying on both Atrophy, Disease and Denervation.
John W. Griffin interconnects Axolemma, Schwann cell, Sciatic nerve and Nervous system in the investigation of issues within Axon. In his study, Slow axonal transport is strongly linked to Axoplasmic transport, which falls under the umbrella field of Neurofilament. His research investigates the connection with Cell biology and areas like Myelin which intersect with concerns in Peripheral nervous system.
John W. Griffin mostly deals with Neuroscience, Biological hazard, Axon, Cell biology and Environmental health. His Neuroscience research incorporates themes from Dynactin, Unmyelinated nerve fiber and Regeneration. His study in Axon is interdisciplinary in nature, drawing from both Luxol fast blue stain, Schwann cell, Immunology, Wallerian degeneration and Experimental autoimmune encephalomyelitis.
His biological study deals with issues like Neurofilament, which deal with fields such as Spinal cord. The various areas that John W. Griffin examines in his Cell biology study include Molecular biology, Myelin-associated glycoprotein, Sciatic nerve and Dorsal root ganglion. Pathology and Nerve fiber are commonly linked in his work.
John W. Griffin mainly investigates Neuroscience, Axon, Myelin, Wallerian degeneration and Internal medicine. John W. Griffin has researched Neuroscience in several fields, including Amyotrophic lateral sclerosis and DCTN1. His study in Axon is interdisciplinary in nature, drawing from both Axonal loss, Multiple sclerosis, Schwann cell and Luxol fast blue stain.
His Wallerian degeneration research incorporates elements of Neurofilament, Sciatic nerve and Cell biology. His study explores the link between Motor nerve and topics such as Sympathetic Fibers that cross with problems in Pathology. He studies Anatomy, focusing on Axoplasmic transport in particular.
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Neuropathic pain Redefinition and a grading system for clinical and research purposes
R. D. Treede;T. S. Jensen;J. N. Campbell;G. Cruccu.
Neurofilament gene expression: a major determinant of axonal caliber.
Paul N. Hoffman;Don W. Cleveland;John W. Griffin;Phillip W. Landes.
Proceedings of the National Academy of Sciences of the United States of America (1987)
Guillain-Barré syndrome in northern China Relationship to Campylobacter jejuni infection and anti-glycolipid antibodies
T. W. Ho;B. Mishu;C. Y. Li;C. Y. Gao.
The spectrum of neuropathy in diabetes and impaired glucose tolerance
C.J. Sumner;S. Sheth;J.W. Griffin;D.R. Cornblath.
DNA/RNA Helicase Gene Mutations in a Form of Juvenile Amyotrophic Lateral Sclerosis (ALS4)
Ying Zhang Chen;Craig L. Bennett;Huy M. Huynh;Ian P. Blair.
American Journal of Human Genetics (2004)
Proposed diagnostic criteria and nosology of acute transverse myelitis
G. Barnes;S. Benjamin;J. D. Bowen;N. Cutter.
Acute motor axonal neuropathy : a frequent cause of acute flaccid paralysis in China
G. M. McKhann;G. M. McKhann;D. R. Cornblath;J. W. Griffin;T. W. Ho;T. W. Ho.
Annals of Neurology (1993)
A treatable multifocal motor neuropathy with antibodies to GM1 ganglioside.
A. Pestronk;D. R. Cornblath;A. A. Ilyas;H. Baba.
Annals of Neurology (1988)
Wallerian degeneration in the peripheral nervous system: participation of both Schwann cells and macrophages in myelin degradation.
G. Stoll;J. W. Griffin;C. Y. Li;B. D. Trapp.
Journal of Neurocytology (1989)
Cytokine expression in the brain during the acquired immunodeficiency syndrome.
William R. Tyor;William R. Tyor;Jonathan D. Glass;John W. Griffin;P. Scott Becker.
Annals of Neurology (1992)
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