What is she best known for?
The fields of study she is best known for:
Her primary areas of investigation include Endoplasmic reticulum, Unfolded protein response, Neurodegeneration, Cell biology and Amyotrophic lateral sclerosis.
Her Endoplasmic reticulum study focuses on Protein disulfide-isomerase in particular.
Her study looks at the relationship between Unfolded protein response and topics such as Pathology, which overlap with Golgi apparatus, Eukaryotic initiation factor, Cytoplasm, Spinal cord and Receptor.
The concepts of her Neurodegeneration study are interwoven with issues in Cancer research, Kinase and Pathogenesis.
Her studies deal with areas such as Autophagy and Programmed cell death as well as Cell biology.
In general Amyotrophic lateral sclerosis study, her work on SOD1 often relates to the realm of Genome-wide association study, thereby connecting several areas of interest.
Her most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- C9ORF72, implicated in amytrophic lateral sclerosis and frontotemporal dementia, regulates endosomal trafficking (314 citations)
- Endoplasmic reticulum stress and induction of the unfolded protein response in human sporadic amyotrophic lateral sclerosis (307 citations)
What are the main themes of her work throughout her whole career to date?
Julie D. Atkin focuses on Amyotrophic lateral sclerosis, Cell biology, Endoplasmic reticulum, Unfolded protein response and Neurodegeneration.
Her work deals with themes such as Neuroscience, Pathogenesis and Frontotemporal dementia, which intersect with Amyotrophic lateral sclerosis.
Julie D. Atkin combines subjects such as Autophagy, Apoptosis, Programmed cell death and Mutant with her study of Cell biology.
Her Endoplasmic reticulum research includes themes of Downregulation and upregulation and Chaperone.
Her Unfolded protein response study which covers Golgi apparatus that intersects with Transport protein and Optineurin.
Her studies deal with areas such as C9orf72, Neuroinflammation and DNA damage as well as Neurodegeneration.
She most often published in these fields:
- Amyotrophic lateral sclerosis (96.45%)
- Cell biology (69.50%)
- Endoplasmic reticulum (72.34%)
What were the highlights of her more recent work (between 2018-2021)?
- Amyotrophic lateral sclerosis (96.45%)
- Cell biology (69.50%)
- SOD1 (46.81%)
In recent papers she was focusing on the following fields of study:
Julie D. Atkin focuses on Amyotrophic lateral sclerosis, Cell biology, SOD1, Endoplasmic reticulum and Unfolded protein response.
Julie D. Atkin interconnects Frontotemporal dementia, Neuroscience and Pathogenesis in the investigation of issues within Amyotrophic lateral sclerosis.
Her research in Cell biology intersects with topics in Cellular homeostasis, Mutant and Neurodegeneration.
Julie D. Atkin has included themes like Secretion, Neuroinflammation, Downregulation and upregulation and Microglia in her Neurodegeneration study.
A large part of her Endoplasmic reticulum studies is devoted to Golgi apparatus.
In her study, Canine degenerative myelopathy and Molecular biology is strongly linked to Protein disulfide-isomerase, which falls under the umbrella field of Unfolded protein response.
Between 2018 and 2021, her most popular works were:
- The microglial NLRP3 inflammasome is activated by amyotrophic lateral sclerosis proteins. (42 citations)
- Motor Neuron Susceptibility in ALS/FTD. (42 citations)
- Motor Neuron Susceptibility in ALS/FTD. (42 citations)
In her most recent research, the most cited papers focused on:
Her main research concerns Neurodegeneration, Amyotrophic lateral sclerosis, Frontotemporal dementia, Cell biology and Pathogenesis.
Her work carried out in the field of Neurodegeneration brings together such families of science as Inflammasome, Protein aggregation, Secretion and DNA, DNA repair.
Her Amyotrophic lateral sclerosis study contributes to a more complete understanding of Disease.
Her Frontotemporal dementia study frequently links to related topics such as Degeneration.
Julie D. Atkin has researched Cell biology in several fields, including Non-homologous end joining, DNA damage, Mutation, RNA and Mutant.
She studied Pathogenesis and HEK 293 cells that intersect with Autophagy.
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