The scientist’s investigation covers issues in SOD1, Superoxide dismutase, Amyotrophic lateral sclerosis, Mitochondrion and Cell biology. Her SOD1 research includes themes of Immunology, Neurogenesis and Transgene. Her Superoxide dismutase study combines topics in areas such as Molecular biology, Reactive oxygen species and Neurodegeneration.
Maria Teresa Carrì interconnects Genetics, Gene, Motor neuron and Neuroscience in the investigation of issues within Amyotrophic lateral sclerosis. While the research belongs to areas of Mitochondrion, she spends her time largely on the problem of Cytosol, intersecting her research to questions surrounding Glutaredoxin 2, Protein aggregation and Glutaredoxin. Her biological study spans a wide range of topics, including Mutant, Microglia, Receptor, Proinflammatory cytokine and Programmed cell death.
Maria Teresa Carrì mainly investigates Superoxide dismutase, SOD1, Amyotrophic lateral sclerosis, Cell biology and Biochemistry. Maria Teresa Carrì has included themes like Molecular biology, Reactive oxygen species and Xenopus in her Superoxide dismutase study. Her research integrates issues of Immunology, Programmed cell death and Intracellular in her study of SOD1.
Her Amyotrophic lateral sclerosis research incorporates themes from Genetics, Transgene, Pathogenesis, Motor neuron and Neuroscience. Maria Teresa Carrì has researched Cell biology in several fields, including Proinflammatory cytokine, Neurodegeneration and Microglia. Her Mitochondrion course of study focuses on Protein aggregation and Cysteine.
Her scientific interests lie mostly in Cell biology, Amyotrophic lateral sclerosis, SOD1, Mitochondrion and Neurodegeneration. Her Cell biology study combines topics from a wide range of disciplines, such as Dopamine receptor, Regulation of gene expression, Cellular differentiation and Internalization. Her work deals with themes such as Genetics, Superoxide dismutase, Gene expression, Pathogenesis and Motor neuron, which intersect with Amyotrophic lateral sclerosis.
SOD1 is a subfield of Mutant that Maria Teresa Carrì explores. Her Mitochondrion research includes elements of Oxidative stress and Protein aggregation. The Neurodegeneration study combines topics in areas such as Neuroinflammation, Biochemistry, Cysteine and Microglia.
Maria Teresa Carrì mostly deals with Amyotrophic lateral sclerosis, Mitochondrion, Pathogenesis, Cell biology and Genetics. Her Mitochondrion research is multidisciplinary, incorporating perspectives in Oxidative stress and Superoxide dismutase. The concepts of her Oxidative stress study are interwoven with issues in SIRT3, Sirtuin and Neurodegeneration.
Her Pathogenesis research integrates issues from Disease and Neuroscience. Her work carried out in the field of Disease brings together such families of science as Excitotoxicity, Unfolded protein response and Microglia. Her research in Cell biology intersects with topics in Receptor, Internalization, Dopamine and LRRK2.
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Expression of a Cu,Zn superoxide dismutase typical of familial amyotrophic lateral sclerosis induces mitochondrial alteration and increase of cytosolic Ca2+ concentration in transfected neuroblastoma SH-SY5Y cells
Maria Teresa Carrì;Alberto Ferri;Andrea Battistoni;Laila Famhy.
FEBS Letters (1997)
Familial ALS-superoxide dismutases associate with mitochondria and shift their redox potentials
Alberto Ferri;Mauro Cozzolino;Claudia Crosio;Monica Nencini.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Neurodegeneration in amyotrophic lateral sclerosis: the role of oxidative stress and altered homeostasis of metals.
Maria Teresa Carrı̀;Alberto Ferri;Mauro Cozzolino;Lilia Calabrese.
Brain Research Bulletin (2003)
Apoptotic mechanisms in mutant LRRK2-mediated cell death
Ciro Iaccarino;Claudia Crosio;Carmine Vitale;Giovanna Sanna.
Human Molecular Genetics (2007)
Mitochondrial dysfunction in ALS
Mauro Cozzolino;Maria Teresa Carrì.
Progress in Neurobiology (2012)
Lessons from models of SOD1-linked familial ALS
Caterina Bendotti;Maria Teresa Carrì.
Trends in Molecular Medicine (2004)
Amyotrophic lateral sclerosis: from current developments in the laboratory to clinical implications.
Mauro Cozzolino;Alberto Ferri;Maria Teresa Carrì.
Antioxidants & Redox Signaling (2008)
Treatment with lithium carbonate does not improve disease progression in two different strains of SOD1 mutant mice.
Chiara Pizzasegola;Ilaria Caron;Cristina Daleno;Anna Ronchi.
Amyotrophic Lateral Sclerosis (2009)
Cu,Zn-superoxide dismutase-dependent apoptosis induced by nitric oxide in neuronal cells
Maria Rosa Ciriolo;Angelo De Martino;Emanuela Lafavia;Luisa Rossi.
Journal of Biological Chemistry (2000)
Cysteine 111 Affects Aggregation and Cytotoxicity of Mutant Cu,Zn-superoxide Dismutase Associated with Familial Amyotrophic Lateral Sclerosis
Mauro Cozzolino;Ilaria Amori;Maria Grazia Pesaresi;Alberto Ferri.
Journal of Biological Chemistry (2008)
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