What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- DNA
Michael J. Strong focuses on Amyotrophic lateral sclerosis, Genetics, Neurofilament, Pathology and Neuroscience.
His Amyotrophic lateral sclerosis study combines topics from a wide range of disciplines, such as Molecular biology, Messenger RNA and Dementia, Frontotemporal dementia.
In Genetics, Michael J. Strong works on issues like Neurodegeneration, which are connected to Protein biosynthesis, RNA splicing, VAPB and Molecular pathology.
His research on Neurofilament often connects related topics like Peripherin.
His biological study focuses on Motor neuron.
His work carried out in the field of Neuroscience brings together such families of science as Tau protein, Disease and Retrograde signaling.
His most cited work include:
- Toward the structural genomics of complexes: Crystal structure of a PE/PPE protein complex from Mycobacterium tuberculosis (611 citations)
- Practice parameter update: the care of the patient with amyotrophic lateral sclerosis: drug, nutritional, and respiratory therapies (an evidence-based review): report of the Quality Standards Subcommittee of the American Academy of Neurology. (500 citations)
- Tuberculosis drug resistance mutation database. (436 citations)
What are the main themes of his work throughout his whole career to date?
Michael J. Strong mainly focuses on Amyotrophic lateral sclerosis, Pathology, Neuroscience, Neurofilament and Cell biology.
His Amyotrophic lateral sclerosis study integrates concerns from other disciplines, such as Frontotemporal dementia, Neurodegeneration, Molecular biology and Motor neuron, Spinal cord.
His Frontotemporal dementia research is multidisciplinary, incorporating elements of Genetics and Cognition.
His studies in Neuroscience integrate themes in fields like Tau protein and Dementia, Disease.
The various areas that Michael J. Strong examines in his Neurofilament study include Protein subunit, Biochemistry, Anatomy, SOD1 and Messenger RNA.
His study in Cell biology is interdisciplinary in nature, drawing from both RNA, RNA-binding protein and Stress granule.
He most often published in these fields:
- Amyotrophic lateral sclerosis (45.95%)
- Pathology (20.39%)
- Neuroscience (18.43%)
What were the highlights of his more recent work (between 2017-2021)?
- Amyotrophic lateral sclerosis (45.95%)
- Cell biology (15.97%)
- Nontuberculous mycobacteria (4.18%)
In recent papers he was focusing on the following fields of study:
Michael J. Strong mostly deals with Amyotrophic lateral sclerosis, Cell biology, Nontuberculous mycobacteria, Surgery and Disease.
Pathology covers he research in Amyotrophic lateral sclerosis.
Within one scientific family, Michael J. Strong focuses on topics pertaining to RNA under Cell biology, and may sometimes address concerns connected to Transcriptome.
His study on Nontuberculous mycobacteria also encompasses disciplines like
- Virulence which intersects with area such as Infectivity and Genome,
- Microbiology which intersects with area such as Mycobacterium abscessus.
Michael J. Strong combines subjects such as Surgical oncology, Antibiotics and Tuberculosis, Mycobacterium tuberculosis with his study of Disease.
His biological study spans a wide range of topics, including Myelin, Luxol fast blue stain, Staining, Stain and Neuropathology.
Between 2017 and 2021, his most popular works were:
- TDP-43 regulates the alternative splicing of hnRNP A1 to yield an aggregation-prone variant in amyotrophic lateral sclerosis (51 citations)
- TDP-43 regulates the alternative splicing of hnRNP A1 to yield an aggregation-prone variant in amyotrophic lateral sclerosis (51 citations)
- Pathologic Thr175 tau phosphorylation in CTE and CTE with ALS (24 citations)
In his most recent research, the most cited papers focused on:
- Gene
- DNA
- Internal medicine
His scientific interests lie mostly in Amyotrophic lateral sclerosis, Cell biology, Pathology, Nontuberculous mycobacteria and Chronic traumatic encephalopathy.
His Amyotrophic lateral sclerosis research is multidisciplinary, incorporating perspectives in Neurofilament, Protein aggregation, C9orf72, Trinucleotide repeat expansion and Spinal cord.
Michael J. Strong interconnects Three prime untranslated region, Intermediate filament, Regulation of gene expression and Motor neuron in the investigation of issues within Neurofilament.
His studies deal with areas such as TARDBP, Alternative splicing, Exon, RNA splicing and RNA-binding protein as well as Cell biology.
His Pathology research includes themes of Germ cell tumors, Pineal gland, Germinoma and Germ cell.
His Phosphorylation research integrates issues from Neuropathology, Tau protein, Neuroscience and Traumatic brain injury.
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