What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Genetics
His primary scientific interests are in Internal medicine, Endocrinology, Hypogonadotropic hypogonadism, Gonadotropin and Gonadotropin-releasing hormone.
In his research, he undertakes multidisciplinary study on Internal medicine and Kallmann syndrome.
William F. Crowley combines subjects such as Agonist and Proband with his study of Endocrinology.
His work on Hypothalamic disease as part of general Hypogonadotropic hypogonadism research is frequently linked to Congenital Hypogonadotropic Hypogonadism, thereby connecting diverse disciplines of science.
The Gonadotropin study combines topics in areas such as Blood sampling, Pulsatile flow, Menstrual cycle and Estrogen.
His Gonadotropin-releasing hormone research includes themes of Secretion and Spermatogenesis.
His most cited work include:
- The GPR54 gene as a regulator of puberty (1883 citations)
- Central challenges facing the national clinical research enterprise. (992 citations)
- A Role for Kisspeptins in the Regulation of Gonadotropin Secretion in the Mouse (908 citations)
What are the main themes of his work throughout his whole career to date?
William F. Crowley mainly focuses on Internal medicine, Endocrinology, Gonadotropin-releasing hormone, Gonadotropin and Hypogonadotropic hypogonadism.
All of his Internal medicine and Testosterone, Follicle-stimulating hormone, Sex steroid, Precocious puberty and Peptide hormone investigations are sub-components of the entire Internal medicine study.
In his works, he performs multidisciplinary study on Endocrinology and Kallmann syndrome.
His Gonadotropin-releasing hormone research includes elements of Ovulation, Endogeny, Pulsatile flow and Stimulation.
William F. Crowley works mostly in the field of Gonadotropin, limiting it down to concerns involving Menstrual cycle and, occasionally, Luteal phase.
His study on Hypothalamic disease is often connected to Congenital Hypogonadotropic Hypogonadism, Anosmia and Isolated hypogonadotropic hypogonadism as part of broader study in Hypogonadotropic hypogonadism.
He most often published in these fields:
- Internal medicine (83.97%)
- Endocrinology (83.38%)
- Gonadotropin-releasing hormone (29.45%)
What were the highlights of his more recent work (between 2012-2021)?
- Internal medicine (83.97%)
- Endocrinology (83.38%)
- Genetics (10.50%)
In recent papers he was focusing on the following fields of study:
Internal medicine, Endocrinology, Genetics, Hypogonadotropic hypogonadism and Kallmann syndrome are his primary areas of study.
His study in the fields of Gonadotropin-releasing hormone, Hormone and GnRH Neuron under the domain of Internal medicine overlaps with other disciplines such as Context and Congenital Hypogonadotropic Hypogonadism.
He has included themes like Young adult and Vaginal delivery in his Endocrinology study.
His Gene, Phenotype and Allele study, which is part of a larger body of work in Genetics, is frequently linked to Immunoglobulin D and RMST, bridging the gap between disciplines.
His studies deal with areas such as Exome sequencing, Sex steroid, Weight loss and Gonadotropin as well as Hypogonadotropic hypogonadism.
The concepts of his Gonadotropin study are interwoven with issues in Human chorionic gonadotropin and Infertility.
Between 2012 and 2021, his most popular works were:
- Mutations in FGF17, IL17RD, DUSP6, SPRY4, and FLRT3 are identified in individuals with congenital hypogonadotropic hypogonadism. (148 citations)
- Prioritizing Genetic Testing in Patients With Kallmann Syndrome Using Clinical Phenotypes (98 citations)
- SMCHD1 mutations associated with a rare muscular dystrophy can also cause isolated arhinia and Bosma arhinia microphthalmia syndrome (77 citations)
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