His primary areas of study are Periodontitis, Immunology, Internal medicine, Inflammation and Disease. His Periodontitis research includes themes of Pathology and Risk factor. Immunology is frequently linked to Animal studies in his study.
Thomas E. Van Dyke has included themes like Eicosapentaenoic acid, Endocrinology, Periodontology and Cardiology in his Internal medicine study. Thomas E. Van Dyke mostly deals with Lipid signaling in his studies of Inflammation. Thomas E. Van Dyke has researched Disease in several fields, including Absolute risk reduction, Epidemiology, Systemic inflammation and Inflammatory response.
Immunology, Periodontitis, Inflammation, Internal medicine and Dentistry are his primary areas of study. The Immunology study combines topics in areas such as Porphyromonas gingivalis and Aggressive periodontitis. In his research, Periodontal disease and Periodontium is intimately related to Disease, which falls under the overarching field of Periodontitis.
Thomas E. Van Dyke combines subjects such as Diabetes mellitus and Receptor with his study of Inflammation. His Internal medicine research integrates issues from Gastroenterology and Endocrinology. His biological study spans a wide range of topics, including Osseointegration and Implant.
Thomas E. Van Dyke mainly investigates Inflammation, Periodontitis, Immunology, Internal medicine and Disease. His Inflammation study integrates concerns from other disciplines, such as Receptor and Regeneration, Cell biology. As a member of one scientific family, he mostly works in the field of Periodontitis, focusing on Gingivitis and, on occasion, Periodontal pathogen.
He interconnects Resolvin, Periodontal disease and Dysbiosis in the investigation of issues within Immunology. His work in Internal medicine addresses subjects such as Gastroenterology, which are connected to disciplines such as Diabetes mellitus, Meta-analysis, Total cholesterol and Triglycerides blood. The study incorporates disciplines such as Periodontium, Proportional hazards model, Apolipoprotein B and Inflammatory biomarkers in addition to Disease.
Thomas E. Van Dyke mainly investigates Periodontitis, Inflammation, Disease, Immunology and Dysbiosis. His Periodontitis research incorporates elements of Gingivitis and Gingival recession. His research in Inflammation intersects with topics in Tumor necrosis factor alpha, Hypoxia, Angiogenesis, Fusobacterium nucleatum and Andrology.
His research integrates issues of Triglycerides blood, Total cholesterol, Pathogenesis and Inflammatory biomarkers in his study of Disease. His study on Immunology is mostly dedicated to connecting different topics, such as Porphyromonas gingivalis. His Porphyromonas gingivalis study combines topics from a wide range of disciplines, such as Resolvin, Macrophage and Dental alveolus.
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Resolving inflammation : dual anti-inflammatory and pro-resolution lipid mediators
Charles N. Serhan;Nan Chiang;Thomas E. Van Dyke.
Nature Reviews Immunology (2008)
Cutting Edge: Different Toll-Like Receptor Agonists Instruct Dendritic Cells to Induce Distinct Th Responses via Differential Modulation of Extracellular Signal-Regulated Kinase-Mitogen-Activated Protein Kinase and c-Fos
Sudhanshu Agrawal;Anshu Agrawal;Barbara Doughty;Andrew Gerwitz.
Journal of Immunology (2003)
Inflammatory and immune pathways in the pathogenesis of periodontal disease.
Ali Cekici;Alpdogan Kantarci;Hatice Hasturk;Thomas E. Van Dyke.
Periodontology 2000 (2014)
Differentiation of Monocytes to Macrophages Primes Cells for Lipopolysaccharide Stimulation via Accumulation of Cytoplasmic Nuclear Factor κB
Shogo Takashiba;Thomas E. Van Dyke;Salomon Amar;Yoji Murayama.
Infection and Immunity (1999)
Lipopolysaccharides from Distinct Pathogens Induce Different Classes of Immune Responses In Vivo
Bali Pulendran;Padmasini Kumar;Christopher W. Cutler;Mansour Mohamadzadeh.
Journal of Immunology (2001)
Periodontitis and atherosclerotic cardiovascular disease: consensus report of the Joint EFP/AAP Workshop on Periodontitis and Systemic Diseases.
Maurizio S. Tonetti;Thomas E. Van Dyke.
Journal of Periodontology (2013)
Yeast zymosan, a stimulus for TLR2 and dectin-1, induces regulatory antigen-presenting cells and immunological tolerance
Stephanie Dillon;Sudhanshu Agrawal;Kaustuv Banerjee;John Letterio.
Journal of Clinical Investigation (2006)
A Toll-like receptor 2 ligand stimulates Th2 responses in vivo, via induction of extracellular signal-regulated kinase mitogen-activated protein kinase and c-Fos in dendritic cells.
Stephanie Dillon;Anshu Agrawal;Thomas Van Dyke;Gary Landreth.
Journal of Immunology (2004)
Platelet-Rich Plasma: Growth Factors and Pro- and Anti-Inflammatory Properties
Hesham El-Sharkawy;Hesham El-Sharkawy;Alpdogan Kantarci;Jennifer Deady;Hatice Hasturk.
Journal of Periodontology (2007)
Consensus Report: Aggressive Periodontitis
Niklaus Lang;P. Mark Bartold;Mary Cullinan;Marjorie Jeffcoat.
Annals of Periodontology (1999)
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