D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 76 Citations 17,301 203 World Ranking 11412 National Ranking 6077

Overview

What is he best known for?

The fields of study he is best known for:

  • Enzyme
  • Gene
  • Internal medicine

His primary areas of investigation include Internal medicine, Endocrinology, Oxidative stress, Mitochondrion and Alzheimer's disease. He interconnects Biochemistry and Programmed cell death in the investigation of issues within Internal medicine. The various areas that Gary E. Gibson examines in his Endocrinology study include Senescence, Oxidative phosphorylation and Pyruvate dehydrogenase complex.

His Oxidative stress study combines topics in areas such as Peroxynitrite, Neurodegeneration, Disease and Nitric oxide. His Mitochondrion study incorporates themes from Reactive oxygen species, Neuroscience, Optic nerve diseases, Glaucoma and Citric acid cycle. His study in Alzheimer's disease is interdisciplinary in nature, drawing from both Medial cortex, Cysteine metabolism and Degenerative disease.

His most cited work include:

  • Consensus report of the working group on: 'Molecular and biochemical markers of Alzheimer's disease' (522 citations)
  • Mitochondrial abnormalities in Alzheimer brain: mechanistic implications. (442 citations)
  • Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease. (354 citations)

What are the main themes of his work throughout his whole career to date?

Gary E. Gibson mainly focuses on Internal medicine, Endocrinology, Biochemistry, Mitochondrion and Oxidative stress. His Internal medicine and Thiamine, Hypoxia, Neurotransmitter, Carbohydrate metabolism and Stimulation investigations all form part of his Internal medicine research activities. His Endocrinology research integrates issues from Alzheimer's disease, Calcium and Pyruvate dehydrogenase complex.

His studies deal with areas such as Dementia and Degenerative disease as well as Alzheimer's disease. His studies in Mitochondrion integrate themes in fields like Molecular biology, Citric acid cycle and Cytosol. His Oxidative stress research is multidisciplinary, incorporating elements of Reactive oxygen species, Oxidative phosphorylation, Neurodegeneration, Disease and Programmed cell death.

He most often published in these fields:

  • Internal medicine (49.82%)
  • Endocrinology (48.75%)
  • Biochemistry (24.56%)

What were the highlights of his more recent work (between 2006-2021)?

  • Mitochondrion (20.64%)
  • Internal medicine (49.82%)
  • Endocrinology (48.75%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Mitochondrion, Internal medicine, Endocrinology, Oxidative stress and Biochemistry. His Mitochondrion research is multidisciplinary, relying on both Alzheimer's disease, Calcium, Citric acid cycle and Cytosol. His Alzheimer's disease study integrates concerns from other disciplines, such as Neuroscience, Depolarization and Voltage-dependent calcium channel.

His Internal medicine research includes themes of Degeneration and NADH dehydrogenase. The study incorporates disciplines such as Dementia, Neurodegeneration, Amyloid precursor protein and Thiamine in addition to Endocrinology. He has researched Oxidative stress in several fields, including Peroxynitrite, Reactive oxygen species, Pathology, Cognitive decline and Programmed cell death.

Between 2006 and 2021, his most popular works were:

  • Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease. (354 citations)
  • Presenilins are enriched in endoplasmic reticulum membranes associated with mitochondria. (251 citations)
  • Cause and consequence: mitochondrial dysfunction initiates and propagates neuronal dysfunction, neuronal death and behavioral abnormalities in age-associated neurodegenerative diseases. (167 citations)

In his most recent research, the most cited papers focused on:

  • Enzyme
  • Gene
  • Internal medicine

Oxidative stress, Mitochondrion, Internal medicine, Endocrinology and Biochemistry are his primary areas of study. The concepts of his Oxidative stress study are interwoven with issues in Alzheimer's disease, Coactivator and Pathology. His study looks at the intersection of Alzheimer's disease and topics like Neuroscience with Calcium metabolism, Disease, Mitochondrial biogenesis and Regulation of gene expression.

His biological study spans a wide range of topics, including Ophthalmology, Optic nerve diseases, Glaucoma, Immune system and Nerve degeneration. His research investigates the connection with Biochemistry and areas like Cell biology which intersect with concerns in Dihydrolipoamide dehydrogenase. His work carried out in the field of Citric acid cycle brings together such families of science as Molecular biology, Pyruvate dehydrogenase complex and Cytosol.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Consensus report of the working group on: 'Molecular and biochemical markers of Alzheimer's disease'

Peter Davies;Judith Resnick;Burton Resnick;Sid Gilman.
Neurobiology of Aging (1998)

809 Citations

Mitochondrial abnormalities in Alzheimer brain: mechanistic implications.

Parvesh Bubber;Vahram Haroutunian;Gene Fisch;John P. Blass.
Annals of Neurology (2005)

612 Citations

Abnormality of a Thiamine-Requiring Enzyme in Patients with Wernicke-Korsakoff Syndrome

John P. Blass;Gary E. Gibson.
The New England Journal of Medicine (1977)

529 Citations

Dietary supplementation with resveratrol reduces plaque pathology in a transgenic model of Alzheimer's disease.

Saravanan S. Karuppagounder;John T. Pinto;Hui Xu;Huan-Lian Chen.
Neurochemistry International (2009)

474 Citations

Calcium and the aging nervous system.

Gary E. Gibson;Christine Peterson.
Neurobiology of Aging (1987)

450 Citations

Reduced Activities of Thiamine-Dependent Enzymes in the Brains and Peripheral Tissues of Patients With Alzheimer's Disease

Gary E. Gibson;Kwan-Fu R. Sheu;John P. Blass;Andrea Baker.
JAMA Neurology (1988)

432 Citations

Protein-bound acrolein: a novel marker of oxidative stress in Alzheimer's disease.

Noel Y. Calingasan;Koji Uchida;Gary E. Gibson.
Journal of Neurochemistry (1999)

420 Citations

Abnormalities of mitochondrial enzymes in Alzheimer disease

G. E. Gibson;K.-F. R. Sheu;J. P. Blass.
Journal of Neural Transmission (1998)

396 Citations

Internal Ca2+ mobilization is altered in fibroblasts from patients with Alzheimer disease.

Etsuro Ito;Kotaro Oka;René Etcheberrigaray;Thomas J. Nelson.
Proceedings of the National Academy of Sciences of the United States of America (1994)

388 Citations

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