His primary scientific interests are in Neuroscience, Protein kinase C, Hippocampus, Cell biology and Hippocampal formation. His research investigates the connection with Neuroscience and areas like Signal transduction which intersect with concerns in Gene expression. He interconnects Endocrinology, Neurodegeneration, Internal medicine and Amyloid in the investigation of issues within Protein kinase C.
Daniel L. Alkon combines subjects such as Gene, Postsynaptic potential and Bioinformatics with his study of Hippocampus. His studies deal with areas such as Regulation of gene expression and Messenger RNA, RNA-binding protein, Untranslated region as well as Cell biology. Many of his research projects under Hippocampal formation are closely connected to Eyelid Conditioning with Eyelid Conditioning, tying the diverse disciplines of science together.
His scientific interests lie mostly in Neuroscience, Protein kinase C, Cell biology, Associative learning and Hermissenda. His Hippocampal formation and Hippocampus study, which is part of a larger body of work in Neuroscience, is frequently linked to Classical conditioning, bridging the gap between disciplines. His research integrates issues of Activator, Protein kinase A and Pharmacology in his study of Protein kinase C.
He has included themes like Downregulation and upregulation and Neuron in his Cell biology study. The various areas that Daniel L. Alkon examines in his Associative learning study include Artificial neural network, Content-addressable memory and Artificial intelligence. While the research belongs to areas of Biophysics, Daniel L. Alkon spends his time largely on the problem of Calcium, intersecting his research to questions surrounding Endocrinology.
Daniel L. Alkon mainly investigates Protein kinase C, Neuroscience, Disease, Bryostatin and Activator. His Protein kinase C study is related to the wider topic of Kinase. His study in Neuroscience focuses on Synaptogenesis in particular.
His work deals with themes such as Enzyme activator, Internal medicine, Inhibitory postsynaptic potential and Protein Kinase C-epsilon, which intersect with Bryostatin. His Neurotrophic factors study incorporates themes from Hippocampal formation and Endocrinology. In the field of Cell biology, his study on Protein kinase A overlaps with subjects such as LRP1.
His primary areas of study are Protein kinase C, Neuroscience, Bryostatin, Neurotrophic factors and Bryostatin 1. His work carried out in the field of Protein kinase C brings together such families of science as Activator, Long-term memory, Neurodegeneration and Pharmacology. His research investigates the link between Activator and topics such as Endocrinology that cross with problems in Internal medicine and Synapse.
His work on Synaptogenesis as part of general Neuroscience research is frequently linked to Synaptophysin, bridging the gap between disciplines. His Neurotrophic factors study integrates concerns from other disciplines, such as Hippocampal formation and Vascular disease. His Bryostatin 1 research is multidisciplinary, relying on both Expanded access, Molecular biology, Dementia and Neuroprotection.
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Accelerating the convergence of the back-propagation method
T. P. Vogl;J. K. Mangis;A. K. Rigler;W. T. Zink.
Biological Cybernetics (1988)
Brain insulin receptors and spatial memory. Correlated changes in gene expression, tyrosine phosphorylation, and signaling molecules in the hippocampus of water maze trained rats.
Weiqin Zhao;Hui Chen;Hui Xu;Elizabeth Moore.
Journal of Biological Chemistry (1999)
Insulin and the insulin receptor in experimental models of learning and memory
Wei-Qin Zhao;Hui Chen;Michael J. Quon;Daniel L. Alkon.
European Journal of Pharmacology (2004)
Calcium-mediated reduction of ionic currents: a biophysical memory trace.
Daniel L. Alkon.
A spatial-temporal model of cell activation.
Daniel L. Alkon;Howard Rasmussen.
Therapeutic effects of PKC activators in Alzheimer's disease transgenic mice.
René Etcheberrigaray;Mathew Tan;Ilse Dewachter;Cuno Kuipéri.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Internal Ca2+ mobilization is altered in fibroblasts from patients with Alzheimer disease.
Etsuro Ito;Kotaro Oka;René Etcheberrigaray;Thomas J. Nelson.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Conditioning-specific membrane changes of rabbit hippocampal neurons measured in vitro
John F. Disterhoft;Douglas A. Coulter;Daniel L. Alkon.
Proceedings of the National Academy of Sciences of the United States of America (1986)
Imaging of memory-specific changes in the distribution of protein kinase C in the hippocampus
James L. Olds;Matthew L. Anderson;Donna L. McPhie;Latonia D. Staten.
PKC signaling deficits: a mechanistic hypothesis for the origins of Alzheimer's disease.
Daniel L. Alkon;Miao-Kun Sun;Thomas J. Nelson.
Trends in Pharmacological Sciences (2007)
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