His main research concerns Cell biology, Apoptosis, Programmed cell death, Caspase and Genetics. In the subject of general Cell biology, his work in Embryo is often linked to Pole plasm, thereby combining diverse domains of study. His work carried out in the field of Apoptosis brings together such families of science as Regulation of gene expression, microRNA, Cytochrome c oxidase and Head involution.
His Programmed cell death study integrates concerns from other disciplines, such as Cancer research, Cell growth, Survivin, Cyclin-dependent kinase and Polo-like kinase. In Caspase, Bruce A. Hay works on issues like Ectopic expression, which are connected to Cell, Mitogen-activated protein kinase and Tissue homeostasis. His studies in Drosophila melanogaster integrate themes in fields like Genome, Gene family, Comparative genomics and Fungal protein.
His primary scientific interests are in Genetics, Cell biology, Gene drive, Gene and Programmed cell death. His study on Drosophila melanogaster, Germline, RNA interference and Genome is often connected to Mechanism as part of broader study in Genetics. His Cell biology research incorporates themes from Inhibitor of apoptosis, Caspase, Apoptosis and Molecular biology.
His Apoptosis research includes themes of Cell, Function and Ectopic expression. His Gene drive study combines topics from a wide range of disciplines, such as Cas9, Essential gene and Transgene. Bruce A. Hay works mostly in the field of Programmed cell death, limiting it down to topics relating to Regulation of gene expression and, in certain cases, Phenotype.
His primary areas of study are Gene drive, Genetics, Computational biology, Gene and Cas9. His Gene drive study also includes
The concepts of his Computational biology study are interwoven with issues in Autophagy, Multicellular organism, Immunology and Gene delivery. Cell biology is closely connected to Messenger RNA in his research, which is encompassed under the umbrella topic of Coding region. His Cell biology study incorporates themes from Translational Activation, Untranslated region, Missense mutation, Disease and Regulation of gene expression.
Bruce A. Hay mainly focuses on Gene drive, Genetics, CRISPR, Gene and Ploidy. The study incorporates disciplines such as Melanogaster, Drosophila virilis, Drosophila melanogaster, Gene conversion and Essential gene in addition to Gene drive. His Genetics research is multidisciplinary, relying on both Degenerative disease and Cell biology.
His biological study spans a wide range of topics, including Ecology, Malaria vector and Insect Gene. His work in Homing endonuclease, Locus, Cas9, Marker gene and Transgene is related to Gene. His research in Ploidy intersects with topics in Threshold population and Chromosomal translocation.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin.
Ira E. Clark;Mark W. Dodson;Changan Jiang;Joseph H. Cao.
Comparative Genomics of the Eukaryotes
Gerald M. Rubin;Mark D. Yandell;Jennifer R. Wortman;George L. Gabor.
The Drosophila MicroRNA Mir-14 Suppresses Cell Death and Is Required for Normal Fat Metabolism
Peizhang Xu;Stephanie Y. Vernooy;Ming Guo;Bruce A. Hay.
Current Biology (2003)
Expression of baculovirus P35 prevents cell death in Drosophila
Bruce A. Hay;Tanya Wolff;Gerald M. Rubin.
Drosophila homologs of baculovirus inhibitor of apoptosis proteins function to block cell death
Bruce A. Hay;David A. Wassarman;Gerald M. Rubin.
A protein component of Drosophila polar granules is encoded by vasa and has extensive sequence similarity to ATP-dependent helicases
Bruce Hay;Lily Yeh Jan;Yuh Nung Jan.
The Drosophila caspase inhibitor DIAP1 is essential for cell survival and is negatively regulated by HID.
Susan L Wang;Christine J Hawkins;Soon Ji Yoo;H.-Arno J Müller.
MicroRNAs and the regulation of cell death.
Peizhang Xu;Ming Guo;Bruce A. Hay.
Trends in Genetics (2004)
Hid, Rpr and Grim negatively regulate DIAP1 levels through distinct mechanisms
Soon Ji Yoo;Jun R. Huh;Israel R. Muro;Hong Yu.
Nature Cell Biology (2002)
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